1991 - Fellow of Alfred P. Sloan Foundation
His scientific interests lie mostly in Genetics, Genome-wide association study, Single-nucleotide polymorphism, Alzheimer's disease and Locus. His Genome-wide association study study combines topics in areas such as PICALM, Case-control study, Genetic association and Macular degeneration. Lindsay A. Farrer has included themes like Apolipoprotein E, Substance dependence and Candidate gene in his Single-nucleotide polymorphism study.
He works mostly in the field of Apolipoprotein E, limiting it down to topics relating to Odds ratio and, in certain cases, Logistic regression. His study looks at the relationship between Alzheimer's disease and fields such as SORL1, as well as how they intersect with chemical problems. Lindsay A. Farrer has researched Locus in several fields, including Genetic marker, Genetic linkage and Gene mapping.
His primary scientific interests are in Genetics, Genome-wide association study, Disease, Single-nucleotide polymorphism and Alzheimer's disease. His study in Gene, Locus, Allele, Genetic linkage and Genetic association are all subfields of Genetics. Lindsay A. Farrer combines subjects such as Alcohol dependence, Oncology and Bioinformatics with his study of Genome-wide association study.
His research investigates the connection between Disease and topics such as Gerontology that intersect with issues in Epidemiology. The concepts of his Single-nucleotide polymorphism study are interwoven with issues in Sickle cell anemia, Haplotype and Candidate gene. His research in Alzheimer's disease intersects with topics in Apolipoprotein E, SORL1 and Endocrinology.
Lindsay A. Farrer mainly investigates Genome-wide association study, Genetics, Disease, Gene and Alzheimer's disease. His research integrates issues of SNP, Alcohol dependence, Bioinformatics, Genetic association and Locus in his study of Genome-wide association study. His Genetics study frequently draws connections between related disciplines such as Apolipoprotein E.
His work carried out in the field of Apolipoprotein E brings together such families of science as Oncology and Genotype. The Disease study combines topics in areas such as Genome and Whole genome sequencing. His studies in Alzheimer's disease integrate themes in fields like Computational biology and Gerontology.
Lindsay A. Farrer mainly focuses on Genome-wide association study, Genetics, Allele, Single-nucleotide polymorphism and Disease. His Genome-wide association study research includes themes of Alzheimer's disease, Genetic association and Heritability. Lindsay A. Farrer interconnects TREM2 and Apolipoprotein E in the investigation of issues within Genetics.
His Allele research is multidisciplinary, incorporating perspectives in Cannabis, Neuroscience, Immunology and Locus. His studies deal with areas such as Internal medicine and Genetic architecture as well as Single-nucleotide polymorphism. His study in Disease is interdisciplinary in nature, drawing from both Neurology, Age related and Macular degeneration.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis
Lindsay A. Farrer;L. Adrienne Cupples;Jonathan L. Haines;Bradley T Hyman.
JAMA (1997)
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium.
L A Farrer;L A Cupples;J L Haines;B Hyman.
JAMA (1997)
Complement Factor H Polymorphism and Age-Related Macular Degeneration
Albert O. Edwards;Robert Ritter;Kenneth J. Abel;Alisa Manning.
Science (2005)
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
Genome-wide analysis of genetic loci associated with Alzheimer disease.
Sudha Seshadri;Annette L. Fitzpatrick;M Arfan Ikram;Anita L. DeStefano.
JAMA (2010)
The neuronal sortilin-related receptor SORL1 is genetically associated with Alzheimer disease
Ekaterina Rogaeva;Ekaterina Rogaeva;Yan Meng;Joseph H. Lee;Yongjun Gu;Yongjun Gu.
Nature Genetics (2007)
Nicastrin modulates presenilin-mediated notch/glp-1 signal transduction and βAPP processing
Gang Yu;Masaki Nishimura;Shigeki Arawaka;Shigeki Arawaka;Diane Levitan.
Nature (2000)
Analysis of shared heritability in common disorders of the brain
Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Science (2018)
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