2009 - Fellow of the American Association for the Advancement of Science (AAAS)
Jonathan L. Haines spends much of his time researching Genetics, Genome-wide association study, Single-nucleotide polymorphism, Alzheimer's disease and Autism. His Genetics study focuses mostly on Locus, Haplotype, Genetic association, Allele and Genetic linkage. His biological study spans a wide range of topics, including Odds ratio, Polymorphism, Genomics, PICALM and Disease.
He combines subjects such as Cancer, Apolipoprotein E, Human genome and Bioinformatics with his study of Single-nucleotide polymorphism. His Alzheimer's disease study integrates concerns from other disciplines, such as Progressive supranuclear palsy, SORL1, Case-control study, Dementia and Age of onset. His research in Autism intersects with topics in Human genetics and Copy-number variation.
His scientific interests lie mostly in Genetics, Disease, Genome-wide association study, Single-nucleotide polymorphism and Alzheimer's disease. His study in Genetics focuses on Locus, Gene, Genetic association, Genetic linkage and Allele. His study in Disease focuses on Late onset in particular.
His Genome-wide association study study combines topics in areas such as Odds ratio, Glaucoma, Bioinformatics and Genomics. His Single-nucleotide polymorphism research focuses on subjects like Internal medicine, which are linked to Macular degeneration. His research on Alzheimer's disease often connects related topics like Apolipoprotein E.
Jonathan L. Haines focuses on Genetics, Disease, Alzheimer's disease, Genome-wide association study and Gene. His works in Genome, Allele, Single-nucleotide polymorphism, Genetic association and Whole genome sequencing are all subjects of inquiry into Genetics. In his study, which falls under the umbrella issue of Genetic association, Genotype is strongly linked to Odds ratio.
His Disease research is multidisciplinary, incorporating perspectives in Meta-analysis, Genetic variants and Risk factor. Within one scientific family, Jonathan L. Haines focuses on topics pertaining to Epidemiology under Alzheimer's disease, and may sometimes address concerns connected to Public health. His Genome-wide association study research incorporates themes from Missense mutation, Intraocular pressure, Linkage disequilibrium, Pedigree chart and Glaucoma.
His primary scientific interests are in Genetics, Genome-wide association study, Disease, Gene and Allele. His work often combines Genetics and TREM2 studies. His Genome-wide association study research incorporates elements of Missense mutation, Intraocular pressure, Open angle glaucoma, Linkage disequilibrium and Alzheimer's disease.
Jonathan L. Haines works mostly in the field of Disease, limiting it down to topics relating to Risk factor and, in certain cases, Cohort study and Haplotype. The Allele study combines topics in areas such as Exome sequencing, Apolipoprotein E and Genetic association. In his research, Genetic architecture and Genomics is intimately related to Locus, which falls under the overarching field of Single-nucleotide polymorphism.
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Finding the missing heritability of complex diseases.
Teri A. Manolio;Francis S. Collins;Nancy J. Cox;David B. Goldstein.
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis.
T. J. Kwiatkowski;D. A. Bosco;D. A. Bosco;A. L. LeClerc;A. L. LeClerc;E. Tamrazian.
Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs
S. Hong Lee;Stephan Ripke;Stephan Ripke;Benjamin M. Neale;Benjamin M. Neale;Stephen V. Faraone.
Nature Genetics (2013)
Common variants at MS4A4/MS4A6E , CD2AP , CD33 and EPHA1 are associated with late-onset Alzheimer's disease
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Mapping autism risk loci using genetic linkage and chromosomal rearrangements
Peter Szatmari;Andrew D. Paterson;Lonnie Zwaigenbaum;Wendy Roberts.
Nature Genetics (2007)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
Mutations in UBQLN2 cause dominant X-linked juvenile and adult-onset ALS and ALS/dementia
Han-Xiang Deng;Wenjie Chen;Seong‐Tshool Hong;Seong‐Tshool Hong;Kym M Boycott.
Interleukin 7 receptor α chain (IL7R) shows allelic and functional association with multiple sclerosis
Simon G. Gregory;Silke Schmidt;Puneet Seth;Jorge R. Oksenberg.
Nature Genetics (2007)
Convergence of Genes and Cellular Pathways Dysregulated in Autism Spectrum Disorders
Dalila Pinto;Elsa Delaby;Elsa Delaby;Elsa Delaby;Daniele Merico;Mafalda Barbosa.
American Journal of Human Genetics (2014)
Profile was last updated on December 6th, 2021.
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