His primary scientific interests are in Genetics, Allele, Single-nucleotide polymorphism, Genome-wide association study and Genotype. His Genetics and Genetic linkage, Haplotype, Linkage disequilibrium, Genetic association and SNP investigations all form part of his Genetics research activities. His Allele research integrates issues from Apolipoprotein E, Genetic variation and Risk factor.
His Single-nucleotide polymorphism research is multidisciplinary, relying on both Odds ratio, Haplogroup H and Haplogroup. The concepts of his Genome-wide association study study are interwoven with issues in Meta-analysis, Human leukocyte antigen and Parkinson's disease. His work in Genotype covers topics such as Macular degeneration which are related to areas like Case-control study.
William K. Scott mostly deals with Genetics, Macular degeneration, Disease, Single-nucleotide polymorphism and Age related. His Genome-wide association study, Genetic linkage, Locus, Haplotype and Allele investigations are all subjects of Genetics research. His research integrates issues of Genetic association and Candidate gene in his study of Genome-wide association study.
His Allele research incorporates themes from Apolipoprotein E, Genotype and Risk factor. The study incorporates disciplines such as Internal medicine, Factor H, Gene and Bioinformatics in addition to Macular degeneration. His Single-nucleotide polymorphism study integrates concerns from other disciplines, such as Genetic variation, Genetic predisposition and Case-control study.
William K. Scott mainly investigates Genetics, Genome-wide association study, Disease, Internal medicine and Macular degeneration. Much of his study explores Genetics relationship to Glaucoma. As a part of the same scientific study, William K. Scott usually deals with the Genome-wide association study, concentrating on Open angle glaucoma and frequently concerns with Odds ratio.
His Disease research includes themes of Mutation and Gene. His Internal medicine study combines topics in areas such as RNA, Endocrinology and Oncology. His study in Macular degeneration is interdisciplinary in nature, drawing from both Age related and Bioinformatics.
His scientific interests lie mostly in Genetics, Genome-wide association study, Glaucoma, Single-nucleotide polymorphism and Disease. Genetic variation, Imputation, Polymorphism, Allele frequency and Gene are the subjects of his Genetics studies. As part of his research on Allele frequency, studies on Genotype and Allele are part of the effort.
His Genome-wide association study research also works with subjects such as
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Complement factor H variant increases the risk of age-related macular degeneration.
Jonathan L. Haines;Michael A. Hauser;Silke Schmidt;William K. Scott.
Science (2005)
Common genetic variation in the HLA region is associated with late-onset sporadic Parkinson's disease
Taye H. Hamza;Cyrus P. Zabetian;Cyrus P. Zabetian;Albert Tenesa;Alain Laederach.
Nature Genetics (2010)
Seven new loci associated with age-related macular degeneration
Lars G. Fritsche;Lars G. Fritsche;Wei Chen;Wei Chen;Matthew Schu;Brian L. Yaspan.
Nature Genetics (2013)
A large genome-wide association study of age-related macular degeneration highlights contributions of rare and common variants
Lars G. Fritsche;Wilmar Igl;Jessica N.Cooke Bailey;Felix Grassmann.
Nature Genetics (2016)
Large-scale meta-analysis of genome-wide association data identifies six new risk loci for Parkinson’s disease
Mike A. Nalls;Nathan Pankratz;Christina M. Lill;Chuong B. Do.
PMC (2014)
Mitochondrial Polymorphisms Significantly Reduce the Risk of Parkinson Disease
Joelle M. van der Walt;Kristin K. Nicodemus;Eden R. Martin;William K. Scott.
American Journal of Human Genetics (2003)
Comprehensive Research Synopsis and Systematic Meta- Analyses in Parkinson's Disease Genetics: The PDGene Database
Christina M. Lill;Johannes T. Roehr;Johannes T. Roehr;Matthew B. McQueen;Fotini K. Kavvoura;Fotini K. Kavvoura;Fotini K. Kavvoura.
PLOS Genetics (2012)
Variation in the miRNA-433 Binding Site of FGF20 Confers Risk for Parkinson Disease by Overexpression of α-Synuclein
Gaofeng Wang;Joelle M. van der Walt;Gregory Mayhew;Yi Ju Li.
American Journal of Human Genetics (2008)
Genetic variants near TIMP3 and high-density lipoprotein–associated loci influence susceptibility to age-related macular degeneration
Wei Chen;Dwight Stambolian;Albert O. Edwards;Kari E. Branham.
Proceedings of the National Academy of Sciences of the United States of America (2010)
Genome-wide association study confirms SNPs in SNCA and the MAPT region as common risk factors for Parkinson disease.
Todd L. Edwards;Todd L. Edwards;William K. Scott;Cherylyn Almonte;Amber Burt.
Annals of Human Genetics (2010)
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