His scientific interests lie mostly in Genetics, Genome-wide association study, Single-nucleotide polymorphism, Disease and Internal medicine. Gene, Locus, LRRK2, Genetic variation and Linkage disequilibrium are the subjects of his Genetics studies. The study incorporates disciplines such as Meta-analysis, Quantitative trait locus, Genetic association, Alzheimer's disease and Candidate gene in addition to Genome-wide association study.
His Single-nucleotide polymorphism research is multidisciplinary, relying on both Apolipoprotein E and Allele frequency. His Disease research includes themes of Mendelian inheritance and Bioinformatics. He has researched Parkinson's disease in several fields, including Genotyping and Parkinsonism.
Andrew B. Singleton mainly investigates Genetics, Disease, Genome-wide association study, Parkinson's disease and Internal medicine. His studies in Gene, Single-nucleotide polymorphism, Locus, Mutation and Genetic variation are all subfields of Genetics research. His Disease research includes elements of Exome sequencing, Computational biology, Cohort and Bioinformatics.
His Genome-wide association study course of study focuses on Expression quantitative trait loci and Quantitative trait locus. His Parkinson's disease research is multidisciplinary, incorporating perspectives in Glucocerebrosidase, Mutation, Parkinsonism and Degenerative disease. The various areas that Andrew B. Singleton examines in his Internal medicine study include Gastroenterology, Endocrinology and Oncology.
Disease, Parkinson's disease, Genetics, Genome-wide association study and Internal medicine are his primary areas of study. The concepts of his Disease study are interwoven with issues in Bioinformatics, Gene, Single-nucleotide polymorphism, Computational biology and Genetic architecture. His work carried out in the field of Parkinson's disease brings together such families of science as Biomarker, Movement disorders and Parkinsonism.
His study looks at the relationship between Genome-wide association study and topics such as Mendelian randomization, which overlap with Observational study. Andrew B. Singleton works mostly in the field of Internal medicine, limiting it down to topics relating to Oncology and, in certain cases, Apolipoprotein E and Allele, as a part of the same area of interest. His LRRK2 study combines topics from a wide range of disciplines, such as Odds ratio, Penetrance, Lewy body and Mutation.
His primary scientific interests are in Disease, Parkinson's disease, Internal medicine, Genome-wide association study and Genetics. His studies in Disease integrate themes in fields like Single-nucleotide polymorphism, Computational biology and Genetic architecture. His Parkinson's disease research incorporates elements of Heterozygote advantage and Genetic risk.
Andrew B. Singleton combines subjects such as Oncology and DNA methylation with his study of Internal medicine. His Genome-wide association study research is multidisciplinary, incorporating elements of Dementia and Locus. Genetics and Context are two areas of study in which Andrew B. Singleton engages in interdisciplinary work.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
α-Synuclein Locus Triplication Causes Parkinson's Disease
A. B. Singleton;M. Farrer;J. Johnson;A. Singleton.
A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD
Alan E. Renton;Elisa Majounie;Adrian James Waite;Javier Simón-Sánchez;Javier Simón-Sánchez.
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease.
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Cloning of the Gene Containing Mutations that Cause PARK8-Linked Parkinson's Disease
Coro Paisán-Ruı́z;Shushant Jain;E.Whitney Evans;William P. Gilks.
New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk
Josée Dupuis;Josée Dupuis;Claudia Langenberg;Inga Prokopenko;Richa Saxena;Richa Saxena.
Nature Genetics (2010)
Rare Structural Variants Disrupt Multiple Genes in Neurodevelopmental Pathways in Schizophrenia
Tom Walsh;Jon M. McClellan;Shane E. McCarthy;Anjené M. Addington.
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)
Genetic variants in novel pathways influence blood pressure and cardiovascular disease risk
Georg B. Ehret;Georg B. Ehret;Georg B. Ehret;Patricia B. Munroe;Kenneth M. Rice;Murielle Bochud.
Genome-wide association study reveals genetic risk underlying Parkinson's disease
Javier Simón-Sánchez;Claudia Schulte;Jose M Bras;Jose M Bras;Manu Sharma.
Nature Genetics (2009)
A genome-wide association search for type 2 diabetes genes in African Americans.
N D Palmer;C W McDonough;P J Hicks;B H Roh.
PLOS ONE (2012)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: