His scientific interests lie mostly in Genetics, Disease, Parkinsonism, Parkinson's disease and Pathology. His Genetics research focuses on Locus, Allele, Genome-wide association study, Haplotype and Mutation. His study in Disease is interdisciplinary in nature, drawing from both Bioinformatics and Medical genetics.
His studies deal with areas such as Missense mutation, Proband, LRRK2, Gene duplication and Penetrance as well as Parkinsonism. His Parkinson's disease research incorporates elements of Neurodegeneration and Kinase activity. His Pathology study deals with Neurology intersecting with Orthostatic vital signs, Anxiety and Severity of illness.
Owen A. Ross mainly focuses on Genetics, Disease, Parkinson's disease, Parkinsonism and Pathology. His studies in LRRK2, Gene, Haplotype, Mutation and Genome-wide association study are all subfields of Genetics research. His Disease study contributes to a more complete understanding of Internal medicine.
His Internal medicine study combines topics in areas such as Gastroenterology and Oncology. His Parkinson's disease research includes themes of Genetic variation, Neuroscience, Allele frequency and Degenerative disease. His Parkinsonism research is multidisciplinary, incorporating elements of Missense mutation, Proband, Frontotemporal dementia, Family history and Pediatrics.
Owen A. Ross mostly deals with Disease, Internal medicine, Genetics, Parkinson's disease and Progressive supranuclear palsy. His study looks at the intersection of Disease and topics like Gene with Computational biology. His work in Internal medicine covers topics such as Oncology which are related to areas like Genetic association, Cognitive decline and Tau protein.
His research in Parkinson's disease focuses on subjects like Parkinsonism, which are connected to Apathy, Pediatrics and Mitochondrion. The study incorporates disciplines such as Neuropathology, Tau pathology, Frontotemporal dementia and Tauopathy in addition to Progressive supranuclear palsy. As a part of the same scientific family, Owen A. Ross mostly works in the field of LRRK2, focusing on Family history and, on occasion, Psychiatry.
Owen A. Ross focuses on Disease, Dementia with Lewy bodies, Progressive supranuclear palsy, Genome-wide association study and Internal medicine. His work deals with themes such as Inflammation, Inflammasome and Missense mutation, which intersect with Disease. His biological study spans a wide range of topics, including Parkinson's disease and Medical genetics.
Owen A. Ross interconnects Frontotemporal dementia and Tauopathy in the investigation of issues within Progressive supranuclear palsy. His study with Genome-wide association study involves better knowledge in Genetics. Owen A. Ross does research in Genetics, focusing on Genetic variation specifically.
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Diagnosis and management of dementia with Lewy bodies Fourth consensus report of the DLB Consortium
Ian G. McKeith;Bradley F. Boeve;Dennis W. DIckson;Glenda Halliday.
VPS35 Mutations in Parkinson Disease
Carles Vilariño-Güell;Christian Wider;Owen A. Ross;Justus C. Dachsel.
American Journal of Human Genetics (2011)
Neuropathologically defined subtypes of Alzheimer's disease with distinct clinical characteristics: a retrospective study.
Melissa E Murray;Neill R Graff-Radford;Owen A Ross;Ronald C Petersen.
Lancet Neurology (2011)
Association between alcohol and cardiovascular disease:Mendelian randomisation analysis based on individual participant data
Michael V Holmes;Michael V Holmes;Caroline E Dale;Luisa Zuccolo;Richard J Silverwood.
Identification of a Novel LRRK2 Mutation Linked to Autosomal Dominant Parkinsonism: Evidence of a Common Founder across European Populations
Jennifer Kachergus;Ignacio F. Mata;Mary Hulihan;Julie P. Taylor.
American Journal of Human Genetics (2005)
Identification of common variants influencing risk of the tauopathy progressive supranuclear palsy
Günter U. Höglinger;Nadine M. Melhem;Dennis W. Dickson;Patrick M A Sleiman.
Nature Genetics (2011)
Pharmacological Rescue of Mitochondrial Deficits in iPSC-Derived Neural Cells from Patients with Familial Parkinson’s Disease
Oliver Cooper;Hyemyung Seo;Shaida Andrabi;Cristina Guardia-Laguarta.
Science Translational Medicine (2012)
Phenotypic variation in a large Swedish pedigree due to SNCA duplication and triplication.
J Fuchs;Christer Nilsson;J Kachergus;M Munz.
Mutations in the colony stimulating factor 1 receptor ( CSF1R ) gene cause hereditary diffuse leukoencephalopathy with spheroids
Rosa Rademakers;Matt Baker;Alexandra M Nicholson;Nicola J Rutherford.
Nature Genetics (2012)
Genomic investigation of alpha-synuclein multiplication and parkinsonism
Owen A. Ross;Adam T. Braithwaite;Lisa M. Skipper;Jennifer Kachergus.
Annals of Neurology (2008)
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