What is he best known for?
The fields of study he is best known for:
Nilufer Ertekin-Taner spends much of his time researching Genetics, Genome-wide association study, Alzheimer's disease, Disease and Single-nucleotide polymorphism.
His studies in Genetics integrate themes in fields like Apolipoprotein E and Age of onset.
His Genome-wide association study research integrates issues from Temporal cortex, Case-control study, Genetic association and Allele frequency.
His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Genetic marker, Gene, Epigenomics and CpG site.
His studies deal with areas such as Pharmacogenomics and Cerebrospinal fluid, Neuroscience as well as Disease.
His study looks at the relationship between Single-nucleotide polymorphism and topics such as Odds ratio, which overlap with Logistic regression, PCDH11X and Gastroenterology.
His most cited work include:
- Common variants at MS4A4/MS4A6E , CD2AP , CD33 and EPHA1 are associated with late-onset Alzheimer's disease (1385 citations)
- Alzheimer's disease: early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci (534 citations)
- Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)
What are the main themes of his work throughout his whole career to date?
Nilufer Ertekin-Taner spends much of his time researching Genetics, Disease, Genome-wide association study, Gene and Alzheimer's disease.
His research on Genetics frequently links to adjacent areas such as Apolipoprotein E.
His research integrates issues of TREM2, Computational biology, Immunology and Bioinformatics in his study of Disease.
His Genome-wide association study course of study focuses on Oncology and Cohort.
His Alzheimer's disease research is multidisciplinary, incorporating perspectives in Age of onset, Linkage disequilibrium, Frontotemporal dementia and Cognitive decline.
His Single-nucleotide polymorphism study incorporates themes from Odds ratio, Internal medicine and Allele.
He most often published in these fields:
- Genetics (42.49%)
- Disease (42.06%)
- Genome-wide association study (24.03%)
What were the highlights of his more recent work (between 2019-2021)?
- Disease (42.06%)
- Gene (21.89%)
- Transcriptome (10.30%)
In recent papers he was focusing on the following fields of study:
Disease, Gene, Transcriptome, Neuroscience and Gene expression are his primary areas of study.
The various areas that Nilufer Ertekin-Taner examines in his Disease study include Neurology, Microglia and Bioinformatics.
His study on Gene is covered under Genetics.
In his research, Parkinson's disease is intimately related to Phenotype, which falls under the overarching field of Neuroscience.
In his study, Cell and Organoid is strongly linked to Neurodegeneration, which falls under the umbrella field of Gene expression.
His work carried out in the field of Genome-wide association study brings together such families of science as Haplotype and Locus.
Between 2019 and 2021, his most popular works were:
- Large-scale proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation (113 citations)
- Meta-Analysis of the Alzheimer's Disease Human Brain Transcriptome and Functional Dissection in Mouse Models. (32 citations)
- Alzheimer's Risk Factors Age, APOE Genotype, and Sex Drive Distinct Molecular Pathways. (19 citations)
In his most recent research, the most cited papers focused on:
Nilufer Ertekin-Taner focuses on Apolipoprotein E, Gene, Neuroscience, Transcriptome and Disease.
His Apolipoprotein E research incorporates elements of Dementia and Pathogenesis.
Gene is a subfield of Genetics that Nilufer Ertekin-Taner investigates.
His research investigates the connection between Genetics and topics such as TREM2 that intersect with issues in Genetic association and Downregulation and upregulation.
His Disease research focuses on Genotype and how it connects with Standardized uptake value, Frontotemporal lobar degeneration and Pittsburgh compound B.
Nilufer Ertekin-Taner studied Genome-wide association study and Case-control study that intersect with Temporal cortex.
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