What is she best known for?
The fields of study she is best known for:
- Gene
- Genetics
- Internal medicine
The scientist’s investigation covers issues in Genetics, Genome-wide association study, Single-nucleotide polymorphism, Allele and Alzheimer's disease.
Her research investigates the connection with Genetics and areas like Case-control study which intersect with concerns in Variation.
Her Genome-wide association study research includes elements of Apolipoprotein E, Minor allele frequency, Allele frequency and Age of onset.
Her Single-nucleotide polymorphism research is multidisciplinary, incorporating perspectives in Odds ratio, Disease, Haplotype and Candidate gene.
Her Allele research integrates issues from Proband and Genotype.
Her study in Alzheimer's disease is interdisciplinary in nature, drawing from both Common disease-common variant, Sibling, Gene expression, TREM2 and Dementia.
Her most cited work include:
- Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)
- Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (2556 citations)
- TREM2 Variants in Alzheimer's Disease (1646 citations)
What are the main themes of her work throughout her whole career to date?
Julie Williams spends much of her time researching Genetics, Disease, Genome-wide association study, Single-nucleotide polymorphism and Allele.
Genetics is closely attributed to Alzheimer's disease in her study.
She has researched Disease in several fields, including Immunology and Bioinformatics.
The study of Genome-wide association study is intertwined with the study of PICALM in a number of ways.
Her studies in Single-nucleotide polymorphism integrate themes in fields like Haplotype and Candidate gene.
Her research in Allele tackles topics such as Apolipoprotein E which are related to areas like Age of onset.
She most often published in these fields:
- Genetics (42.50%)
- Disease (33.25%)
- Genome-wide association study (16.75%)
What were the highlights of her more recent work (between 2015-2021)?
- Disease (33.25%)
- Genetics (42.50%)
- Gene (14.00%)
In recent papers she was focusing on the following fields of study:
Disease, Genetics, Gene, Genome-wide association study and Genetic association are her primary areas of study.
Her Disease study incorporates themes from Oncology and Bioinformatics.
Her Genetics research incorporates elements of TREM2 and Late onset.
Her Gene study combines topics from a wide range of disciplines, such as Lipid metabolism, Computational biology and Immunology.
Her Genome-wide association study research entails a greater understanding of Single-nucleotide polymorphism.
The various areas that Julie Williams examines in her Genetic association study include Alzheimer's disease, Dementia, Microglia and Heritability.
Between 2015 and 2021, her most popular works were:
- Analysis of shared heritability in common disorders of the brain (726 citations)
- Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)
- A common haplotype lowers PU.1 expression in myeloid cells and delays onset of Alzheimer's disease. (196 citations)
In her most recent research, the most cited papers focused on:
Her primary areas of investigation include Disease, Genome-wide association study, Genetics, Genetic association and Dementia.
Her work in the fields of Disease, such as Apolipoprotein E, overlaps with other areas such as Complement component 9.
Her Genome-wide association study study results in a more complete grasp of Single-nucleotide polymorphism.
Gene, Genetic risk and Heritability are the primary areas of interest in her Genetics study.
Julie Williams works mostly in the field of Genetic association, limiting it down to concerns involving Meta-analysis and, occasionally, Lipid metabolism and Logistic regression.
Her Dementia research includes themes of Referral, Alzheimer's disease, Penetrance, Family history and Amyloid.
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