What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Genetics, Huntington's disease, Alzheimer's disease, Genome-wide association study and Locus.
His work in Allele, Linkage disequilibrium and Gene are all subfields of Genetics research.
His work in the fields of Huntington's disease, such as Huntingtin and Huntingtin Protein, overlaps with other areas such as Law and economics.
Lesley Jones usually deals with Alzheimer's disease and limits it to topics linked to Apolipoprotein E and Genetic association.
His Genome-wide association study study is related to the wider topic of Single-nucleotide polymorphism.
His Single-nucleotide polymorphism research includes themes of Disease and Age of onset.
His most cited work include:
- Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)
- Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease (1387 citations)
- Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease (1242 citations)
What are the main themes of his work throughout his whole career to date?
Genetics, Huntington's disease, Disease, Gene and Huntingtin are his primary areas of study.
His Single-nucleotide polymorphism, Genome-wide association study, Allele, Locus and Genetic association study are his primary interests in Genetics.
Apolipoprotein E and Case-control study is closely connected to Alzheimer's disease in his research, which is encompassed under the umbrella topic of Genome-wide association study.
His Huntington's disease research is multidisciplinary, relying on both Phenotype, Neuroscience, Neurodegeneration and Trinucleotide repeat expansion.
Lesley Jones studied Disease and Psychiatry that intersect with Cognitive decline.
His Huntingtin study combines topics in areas such as Molecular biology and Cell biology.
He most often published in these fields:
- Genetics (46.98%)
- Huntington's disease (37.21%)
- Disease (26.05%)
What were the highlights of his more recent work (between 2017-2021)?
- Huntington's disease (37.21%)
- Genetics (46.98%)
- Trinucleotide repeat expansion (14.88%)
In recent papers he was focusing on the following fields of study:
Lesley Jones mainly focuses on Huntington's disease, Genetics, Trinucleotide repeat expansion, Disease and Gene.
His Huntington's disease study incorporates themes from Irritability, Depression and Locus.
His study in Genome-wide association study, MSH3, Allele, Genetic association and Single-nucleotide polymorphism falls within the category of Genetics.
His Genome-wide association study research is multidisciplinary, incorporating elements of Dementia and Pathogenesis.
The study incorporates disciplines such as Meta-analysis, Lipid metabolism and Alzheimer's disease in addition to Genetic association.
His work deals with themes such as Psychiatry, Psychosis and Oncology, which intersect with Disease.
Between 2017 and 2021, his most popular works were:
- CAG Repeat Not Polyglutamine Length Determines Timing of Huntington’s Disease Onset (105 citations)
- A genetic association study of glutamine-encoding DNA sequence structures, somatic CAG expansion, and DNA repair gene variants, with Huntington disease clinical outcomes (49 citations)
- FAN1 modifies Huntington's disease progression by stabilizing the expanded HTT CAG repeat. (44 citations)
In his most recent research, the most cited papers focused on:
Lesley Jones mostly deals with Genetics, Huntington's disease, Trinucleotide repeat expansion, Gene and Locus.
In his study, which falls under the umbrella issue of Genetics, Pathogenesis is strongly linked to Disease.
His research in Huntington's disease intersects with topics in Allele and Exon.
His research investigates the link between Trinucleotide repeat expansion and topics such as MSH3 that cross with problems in Somatic cell, MLH3, Polyglutamine tract, Genotype and Candidate gene.
His study looks at the relationship between Locus and topics such as Human genetics, which overlap with Cognitive decline, Medical genetics, Gene mapping, Haplotype and Human genome.
His Genome-wide association study research entails a greater understanding of Single-nucleotide polymorphism.
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