Fellow of The Academy of Medical Sciences, United Kingdom
Simon Lovestone mainly investigates Alzheimer's disease, Disease, Neuroscience, Genetics and Dementia. To a larger extent, Simon Lovestone studies Pathology with the aim of understanding Alzheimer's disease. His Disease study is associated with Internal medicine.
His Neuroscience study combines topics in areas such as Phenotype, Schizophrenia, Neurodegeneration and GSK-3. His Dementia research incorporates elements of Psychiatry, Proteomics and Bioinformatics. Simon Lovestone interconnects Odds ratio and Apolipoprotein E in the investigation of issues within Single-nucleotide polymorphism.
Disease, Alzheimer's disease, Internal medicine, Dementia and Genetics are his primary areas of study. Simon Lovestone has researched Disease in several fields, including Biomarker, Neuroscience and Bioinformatics. His Alzheimer's disease research is multidisciplinary, relying on both Immunology, Magnetic resonance imaging, Apolipoprotein E, Atrophy and Degenerative disease.
His Internal medicine research incorporates themes from Endocrinology and Oncology. The concepts of his Dementia study are interwoven with issues in Clinical trial, Cohort study, Gerontology and Psychiatry, Cognition. His Genetics research focuses on Single-nucleotide polymorphism, Allele, Genome-wide association study, Locus and Genetic association.
Simon Lovestone mostly deals with Disease, Internal medicine, Dementia, Oncology and Neuroscience. His work in Disease addresses issues such as Genome-wide association study, which are connected to fields such as Genetic architecture and Genetic association. As a part of the same scientific family, Simon Lovestone mostly works in the field of Internal medicine, focusing on Endocrinology and, on occasion, Inflammation.
His Dementia research integrates issues from Apolipoprotein E, Cognition, Cohort and Biomarker discovery. As a part of the same scientific study, he usually deals with the Neuroscience, concentrating on Candidate gene and frequently concerns with Thalamus. His Alzheimer's disease research includes themes of Cerebral cortex and Atrophy.
His primary areas of study are Alzheimer's disease, Dementia, Internal medicine, Disease and Neuroscience. His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Hippocampal formation, Genome-wide association study, Atrophy and Biomarker discovery. His biological study spans a wide range of topics, including Psychiatry, Brain Structure and Function, Cognition and Neurology.
The study incorporates disciplines such as Endocrinology and Oncology in addition to Internal medicine. His research integrates issues of Analysis of variance, Statistics, Clinical trial, Epidemiology and Intensive care medicine in his study of Disease. The concepts of his Neuroscience study are interwoven with issues in Schizophrenia and Cognitive decline.
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Consensus guidelines for the clinical and pathologic diagnosis of dementia with Lewy bodies (DLB) Report of the consortium on DLB international workshop
I. G. Mckeith;D. Galasko;K. Kosaka;E. K. Perry.
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))
D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
The GSK3 hypothesis of Alzheimer's disease.
Claudie Hooper;Richard Killick;Simon Lovestone.
Journal of Neurochemistry (2008)
Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease.
Lesley Jones;Peter A. Holmans;Marian L. Hamshere;Denise Harold.
PLOS ONE (2010)
Head injury as a risk factor for Alzheimer’s disease: the evidence 10 years on; a partial replication
S Fleminger;D L Oliver;S Lovestone;S Rabe-Hesketh.
Journal of Neurology, Neurosurgery, and Psychiatry (2003)
Common genetic variants influence human subcortical brain structures.
Derrek P. Hibar;Jason L. Stein;Jason L. Stein;Miguel E. Renteria;Alejandro Arias-Vasquez.
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