John Powell spends much of his time researching Genetics, Genome-wide association study, Alzheimer's disease, Single-nucleotide polymorphism and Disease. His studies examine the connections between Genetics and genetics, as well as such issues in Amyotrophic lateral sclerosis, with regards to SNP and Age of onset. His Genome-wide association study research includes elements of Odds ratio, Gene expression, Linkage disequilibrium, Neurodegeneration and Genetic association.
His Alzheimer's disease research integrates issues from Common disease-common variant, Oncology, Case-control study, Dementia and Entorhinal cortex. John Powell focuses mostly in the field of Single-nucleotide polymorphism, narrowing it down to matters related to Apolipoprotein E and, in some cases, Bioinformatics and Temporal cortex. His Allele research incorporates elements of Psychiatric genetics and Degenerative disease.
John Powell mostly deals with Genetics, Genome-wide association study, Disease, Internal medicine and Gene. Much of his study explores Genetics relationship to Amyotrophic lateral sclerosis. His study focuses on the intersection of Genome-wide association study and fields such as Bipolar disorder with connections in the field of Psychosis.
His Internal medicine research includes themes of Endocrinology and Oncology. His work in Gene tackles topics such as Molecular biology which are related to areas like Complementary DNA. John Powell works mostly in the field of Single-nucleotide polymorphism, limiting it down to topics relating to Allele and, in certain cases, Genotype, as a part of the same area of interest.
His primary areas of investigation include Genetics, Genome-wide association study, Alzheimer's disease, Disease and Schizophrenia. His Genetics study frequently intersects with other fields, such as Amyotrophic lateral sclerosis. His study on Genome-wide association study is covered under Single-nucleotide polymorphism.
His work carried out in the field of Alzheimer's disease brings together such families of science as Common disease-common variant, Case-control study, Dementia, Apolipoprotein E and Neuroscience. His biological study spans a wide range of topics, including Plasma samples and Bioinformatics. John Powell has included themes like SORL1 and Exome in his Genetic association study.
Genome-wide association study, Genetics, Alzheimer's disease, Case-control study and Genetic association are his primary areas of study. His Genome-wide association study study combines topics in areas such as Odds ratio, Schizophrenia, Disease and Bipolar disorder. His Schizophrenia study incorporates themes from Young adult and Allele.
His Genetics study combines topics from a wide range of disciplines, such as Amyotrophic lateral sclerosis and Age of onset. John Powell interconnects Common disease-common variant, SORL1, Neurodegeneration, Dementia and Entorhinal cortex in the investigation of issues within Alzheimer's disease. His Case-control study research incorporates themes from Linkage disequilibrium and Genotype.
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Biological insights from 108 schizophrenia-associated genetic loci
Stephan Ripke;Stephan Ripke;Benjamin M. Neale;Benjamin M. Neale;Aiden Corvin;James T. R. Walters.
Nature (2014)
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))
D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
Genome-wide association analysis identifies 13 new risk loci for schizophrenia
Stephan Ripke;Stephan Ripke;Colm T. O'Dushlaine;Kimberly D. Chambert;Jennifer L. Moran.
Nature Genetics (2013)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
Tyrosine hydroxylase-immunoreactive boutons in synaptic contact with identified striatonigral neurons, with particular reference to dendritic spines
T.F. Freund;J.F. Powell;A.D. Smith.
Neuroscience (1984)
Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease.
Lesley Jones;Peter A. Holmans;Marian L. Hamshere;Denise Harold.
PLOS ONE (2010)
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