D-Index & Metrics Best Publications
Genetics
UK
2023

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 107 Citations 60,804 788 World Ranking 3598 National Ranking 364
Genetics D-index 105 Citations 54,440 706 World Ranking 383 National Ranking 65

Research.com Recognitions

Awards & Achievements

2023 - Research.com Genetics in United Kingdom Leader Award

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

Amyotrophic lateral sclerosis, Genetics, Cell biology, Mutation and Pathology are his primary areas of study. His biological study focuses on TARDBP. His research integrates issues of Neurofilament and RNA-binding protein in his study of Cell biology.

His study looks at the relationship between Mutation and topics such as Mutant, which overlap with Nuclear localization sequence. His Pathology research includes themes of Motor cortex and Internal medicine. His studies in Neurodegeneration integrate themes in fields like Molecular biology and RNA splicing.

His most cited work include:

  • Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (2556 citations)
  • The ATLAS Experiment at the CERN Large Hadron Collider (2415 citations)
  • TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis (1926 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Amyotrophic lateral sclerosis, Biochemistry, Internal medicine, Peptide and Endocrinology. His Amyotrophic lateral sclerosis study integrates concerns from other disciplines, such as Mutation, Genetics, C9orf72 and Neuroscience. His Genetics research focuses on Gene, Genome-wide association study, Single-nucleotide polymorphism and Locus.

His Biochemistry study deals with Amphibian intersecting with Cloning. Christopher Shaw has included themes like cDNA library, Molecular biology, Protein primary structure and Secretion in his Peptide study. Christopher Shaw focuses mostly in the field of Endocrinology, narrowing it down to matters related to Neuropeptide and, in some cases, Immunocytochemistry.

He most often published in these fields:

  • Amyotrophic lateral sclerosis (24.67%)
  • Biochemistry (19.93%)
  • Internal medicine (19.71%)

What were the highlights of his more recent work (between 2013-2021)?

  • Amyotrophic lateral sclerosis (24.67%)
  • Peptide (18.94%)
  • Genetics (12.44%)

In recent papers he was focusing on the following fields of study:

Christopher Shaw focuses on Amyotrophic lateral sclerosis, Peptide, Genetics, Biochemistry and Cell biology. Christopher Shaw interconnects Mutation, Gene, C9orf72 and Neuroscience in the investigation of issues within Amyotrophic lateral sclerosis. His work carried out in the field of Peptide brings together such families of science as Molecular cloning, Peptide sequence, Secretion and Antimicrobial.

Many of his studies on Genetics apply to Case-control study as well. His biological study deals with issues like Amphibian, which deal with fields such as Endocrinology. His Cell biology study also includes fields such as

  • Autophagy, which have a strong connection to Lysosome,
  • RNA that intertwine with fields like Neurodegeneration and Messenger RNA.

Between 2013 and 2021, his most popular works were:

  • The ATLAS Experiment at the CERN Large Hadron Collider (2415 citations)
  • Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways (597 citations)
  • Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

Christopher Shaw mostly deals with Amyotrophic lateral sclerosis, Genetics, Cell biology, C9orf72 and Gene. His Amyotrophic lateral sclerosis research incorporates elements of Genome-wide association study, Frontotemporal dementia, Mutation, Neurodegeneration and Neuroscience. In his research, Allele and Linkage disequilibrium is intimately related to Case-control study, which falls under the overarching field of Genetics.

His work deals with themes such as Autophagy, AMPA receptor, Messenger RNA and RNA, which intersect with Cell biology. His study in C9orf72 is interdisciplinary in nature, drawing from both C9orf72 Protein and Motor neuron. His work in Gene covers topics such as Bioinformatics which are related to areas like Pathophysiology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The ATLAS Experiment at the CERN Large Hadron Collider

G. Aad;E. Abat;J. Abdallah;J. Abdallah;A. A. Abdelalim.
(2020)

17964 Citations

Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease

Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)

2992 Citations

Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))

D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)

2857 Citations

TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis

Jemeen Sreedharan;Ian P. Blair;Vineeta B. Tripathi;Xun Hu.
Science (2008)

2652 Citations

Expected performance of the ATLAS experiment - detector, trigger and physics

G. Aad;E. Abat;B. Abbott;J. Abdallah.
Jan 2009. 1852pp. arXiv:0901.0512 (2009)

2634 Citations

Mutations in FUS, an RNA Processing Protein, Cause Familial Amyotrophic Lateral Sclerosis Type 6

Caroline Vance;Boris Rogelj;Tibor Hortobágyi;Kurt J. De Vos.
Science (2009)

2623 Citations

Expected Performance of the ATLAS Experiment - Detector, Trigger and Physics

G. Aad;E. Abat;B. Abbott.
arXiv: High Energy Physics - Experiment (2008)

2425 Citations

Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.

Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)

1904 Citations

Charged-particle multiplicities in pp interactions measured with the ATLAS detector at the LHC

G. Aad;B. Abbott;J. Abdallah;A. A. Abdelalim.
New Journal of Physics (2011)

1369 Citations

Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS

Hong Joo Kim;Nam Chul Kim;Yong Dong Wang;Emily A. Scarborough.
Nature (2013)

1262 Citations

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