2023 - Research.com Genetics in United Kingdom Leader Award
Amyotrophic lateral sclerosis, Genetics, Cell biology, Mutation and Pathology are his primary areas of study. His biological study focuses on TARDBP. His research integrates issues of Neurofilament and RNA-binding protein in his study of Cell biology.
His study looks at the relationship between Mutation and topics such as Mutant, which overlap with Nuclear localization sequence. His Pathology research includes themes of Motor cortex and Internal medicine. His studies in Neurodegeneration integrate themes in fields like Molecular biology and RNA splicing.
His scientific interests lie mostly in Amyotrophic lateral sclerosis, Biochemistry, Internal medicine, Peptide and Endocrinology. His Amyotrophic lateral sclerosis study integrates concerns from other disciplines, such as Mutation, Genetics, C9orf72 and Neuroscience. His Genetics research focuses on Gene, Genome-wide association study, Single-nucleotide polymorphism and Locus.
His Biochemistry study deals with Amphibian intersecting with Cloning. Christopher Shaw has included themes like cDNA library, Molecular biology, Protein primary structure and Secretion in his Peptide study. Christopher Shaw focuses mostly in the field of Endocrinology, narrowing it down to matters related to Neuropeptide and, in some cases, Immunocytochemistry.
Christopher Shaw focuses on Amyotrophic lateral sclerosis, Peptide, Genetics, Biochemistry and Cell biology. Christopher Shaw interconnects Mutation, Gene, C9orf72 and Neuroscience in the investigation of issues within Amyotrophic lateral sclerosis. His work carried out in the field of Peptide brings together such families of science as Molecular cloning, Peptide sequence, Secretion and Antimicrobial.
Many of his studies on Genetics apply to Case-control study as well. His biological study deals with issues like Amphibian, which deal with fields such as Endocrinology. His Cell biology study also includes fields such as
Christopher Shaw mostly deals with Amyotrophic lateral sclerosis, Genetics, Cell biology, C9orf72 and Gene. His Amyotrophic lateral sclerosis research incorporates elements of Genome-wide association study, Frontotemporal dementia, Mutation, Neurodegeneration and Neuroscience. In his research, Allele and Linkage disequilibrium is intimately related to Case-control study, which falls under the overarching field of Genetics.
His work deals with themes such as Autophagy, AMPA receptor, Messenger RNA and RNA, which intersect with Cell biology. His study in C9orf72 is interdisciplinary in nature, drawing from both C9orf72 Protein and Motor neuron. His work in Gene covers topics such as Bioinformatics which are related to areas like Pathophysiology.
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The ATLAS Experiment at the CERN Large Hadron Collider
G. Aad;E. Abat;J. Abdallah;J. Abdallah;A. A. Abdelalim.
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))
D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)
TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis
Jemeen Sreedharan;Ian P. Blair;Vineeta B. Tripathi;Xun Hu.
Expected performance of the ATLAS experiment - detector, trigger and physics
G. Aad;E. Abat;B. Abbott;J. Abdallah.
Jan 2009. 1852pp. arXiv:0901.0512 (2009)
Mutations in FUS, an RNA Processing Protein, Cause Familial Amyotrophic Lateral Sclerosis Type 6
Caroline Vance;Boris Rogelj;Tibor Hortobágyi;Kurt J. De Vos.
Expected Performance of the ATLAS Experiment - Detector, Trigger and Physics
G. Aad;E. Abat;B. Abbott.
arXiv: High Energy Physics - Experiment (2008)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
Charged-particle multiplicities in pp interactions measured with the ATLAS detector at the LHC
G. Aad;B. Abbott;J. Abdallah;A. A. Abdelalim.
New Journal of Physics (2011)
Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS
Hong Joo Kim;Nam Chul Kim;Yong Dong Wang;Emily A. Scarborough.
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