What is he best known for?
The fields of study he is best known for:
His primary areas of study are Alzheimer's disease, Amyloid precursor protein, Genetics, Genetically modified mouse and Internal medicine.
His Alzheimer's disease research incorporates themes from Mutation, Immunology, Epigenomics, Differentially methylated regions and Degenerative disease.
The study incorporates disciplines such as Molecular biology, Amyloid beta and Neuroscience in addition to Amyloid precursor protein.
His Genetics research focuses on subjects like Late onset, which are linked to Human genetics and Molecular genetics.
In his study, Mutant and Senile plaques is inextricably linked to Presenilin, which falls within the broad field of Genetically modified mouse.
In his work, Amyloid is strongly intertwined with Endocrinology, which is a subfield of Internal medicine.
His most cited work include:
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice (3809 citations)
- Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease (2614 citations)
- Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (2556 citations)
What are the main themes of his work throughout his whole career to date?
Steven G. Younkin focuses on Genetics, Disease, Internal medicine, Alzheimer's disease and Genome-wide association study.
His study brings together the fields of Apolipoprotein E and Genetics.
His biological study deals with issues like Endocrinology, which deal with fields such as Amyloid precursor protein.
His Amyloid precursor protein study incorporates themes from Molecular biology and Presenilin.
As a part of the same scientific family, he mostly works in the field of Alzheimer's disease, focusing on Amyloid and, on occasion, Biochemistry, Genetically modified mouse and Amyloid beta.
His Genome-wide association study research includes elements of Temporal cortex, Case-control study, PICALM, Genetic association and Candidate gene.
He most often published in these fields:
- Genetics (31.16%)
- Disease (26.91%)
- Internal medicine (22.95%)
What were the highlights of his more recent work (between 2013-2021)?
- Genetics (31.16%)
- Disease (26.91%)
- Genome-wide association study (17.28%)
In recent papers he was focusing on the following fields of study:
Steven G. Younkin spends much of his time researching Genetics, Disease, Genome-wide association study, Alzheimer's disease and Single-nucleotide polymorphism.
His Genetics study frequently involves adjacent topics like Case-control study.
The subject of his Disease research is within the realm of Internal medicine.
His Internal medicine research is multidisciplinary, incorporating perspectives in Endocrinology and Pathology.
Steven G. Younkin combines subjects such as Progressive supranuclear palsy, Temporal cortex, Endophenotype, Cerebral amyloid angiopathy and Apolipoprotein E with his study of Genome-wide association study.
His Alzheimer's disease research incorporates themes from Biological pathway, Transcriptome, Dementia and Oncology.
Between 2013 and 2021, his most popular works were:
- Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease (1242 citations)
- Alzheimer's disease: early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci (534 citations)
- Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease (429 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Genetics, Alzheimer's disease, Genome-wide association study, Single-nucleotide polymorphism and Disease.
Steven G. Younkin undertakes multidisciplinary studies into Genetics and TREM2 in his work.
His research investigates the connection between Alzheimer's disease and topics such as Dementia that intersect with issues in Lower risk.
His work in Genome-wide association study addresses issues such as Genetic association, which are connected to fields such as Haplotype.
His research investigates the link between Single-nucleotide polymorphism and topics such as Apolipoprotein E that cross with problems in Cell biology and Allele.
The Internal medicine study combines topics in areas such as PICALM and Endophenotype.
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