D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 39 Citations 7,213 118 World Ranking 3406 National Ranking 1539

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Alzheimer's disease
  • Disease

His scientific interests lie mostly in Pathology, Dementia, Frontotemporal lobar degeneration, Amyotrophic lateral sclerosis and Disease. His Pathology study frequently links to related topics such as White matter. His work in Dementia covers topics such as Alzheimer's disease which are related to areas like Dementia with Lewy bodies and Biomarker.

David J. Irwin has included themes like Progressive supranuclear palsy and Frontal lobe in his Frontotemporal lobar degeneration study. The concepts of his Amyotrophic lateral sclerosis study are interwoven with issues in C9orf72 Protein, C9orf72, Anatomy and Atrophy. His Disease study integrates concerns from other disciplines, such as Neuroscience and Oncology.

His most cited work include:

  • Clinical diagnosis of progressive supranuclear palsy: The movement disorder society criteria (612 citations)
  • Stages of pTDP‐43 pathology in amyotrophic lateral sclerosis (534 citations)
  • Parkinson's disease dementia: convergence of α-synuclein, tau and amyloid-β pathologies (426 citations)

What are the main themes of his work throughout his whole career to date?

David J. Irwin focuses on Pathology, Frontotemporal lobar degeneration, Frontotemporal dementia, Disease and Dementia. His work on White matter expands to the thematically related Pathology. His study in Frontotemporal lobar degeneration is interdisciplinary in nature, drawing from both Primary progressive aphasia, C9orf72, Progressive supranuclear palsy and Oncology.

His Frontotemporal dementia research is multidisciplinary, relying on both Tau protein, Neuropsychology, Psychiatry, Asymptomatic and Neuroscience. David J. Irwin interconnects Differential diagnosis and Autopsy in the investigation of issues within Disease. His Dementia research is multidisciplinary, incorporating perspectives in Alzheimer's disease, Cognition and Parkinson's disease.

He most often published in these fields:

  • Pathology (39.11%)
  • Frontotemporal lobar degeneration (31.00%)
  • Frontotemporal dementia (27.31%)

What were the highlights of his more recent work (between 2019-2021)?

  • Pathology (39.11%)
  • Frontotemporal dementia (27.31%)
  • Frontotemporal lobar degeneration (31.00%)

In recent papers he was focusing on the following fields of study:

David J. Irwin spends much of his time researching Pathology, Frontotemporal dementia, Frontotemporal lobar degeneration, Disease and Atrophy. The concepts of his Pathology study are interwoven with issues in Temporal lobe and In vivo. In general Frontotemporal dementia, his work in C9orf72 is often linked to Grossman linking many areas of study.

His Frontotemporal lobar degeneration research is multidisciplinary, relying on both Primary progressive aphasia, Clinical Dementia Rating, Corticobasal degeneration and Cerebrospinal fluid. David J. Irwin works in the field of Disease, namely Progressive supranuclear palsy. His research investigates the link between Alzheimer's disease and topics such as Dementia that cross with problems in Positron emission tomography and Oncology.

Between 2019 and 2021, his most popular works were:

  • Positron Emission Tomography Imaging With [18F]flortaucipir and Postmortem Assessment of Alzheimer Disease Neuropathologic Changes (55 citations)
  • Age at symptom onset and death and disease duration in genetic frontotemporal dementia: an international retrospective cohort study. (43 citations)
  • Distribution patterns of tau pathology in progressive supranuclear palsy (29 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Alzheimer's disease
  • Disease

Frontotemporal dementia, Frontotemporal lobar degeneration, Atrophy, Disease and Neuropathology are his primary areas of study. In the subject of general Frontotemporal dementia, his work in C9orf72 is often linked to Grossman, thereby combining diverse domains of study. His research in Frontotemporal lobar degeneration intersects with topics in TAR DNA-BINDING PROTEIN, Unknown Significance, Audiology, Clinical Dementia Rating and Primary progressive aphasia.

His Disease research is within the category of Pathology. His Pathology research incorporates themes from Biochemical biomarkers and Appropriate Use Criteria. His Neuropathology study incorporates themes from Dementia with Lewy bodies, Dementia, Cohort and Autopsy.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Clinical diagnosis of progressive supranuclear palsy: The movement disorder society criteria

Günter U Höglinger;Gesine Respondek;Maria Stamelou;Carolin Kurz.
Movement Disorders (2017)

729 Citations

Stages of pTDP‐43 pathology in amyotrophic lateral sclerosis

Johannes Brettschneider;Kelly Del Tredici;Jon B. Toledo;John L. Robinson.
Annals of Neurology (2013)

645 Citations

Parkinson's disease dementia: convergence of α-synuclein, tau and amyloid-β pathologies

David J. Irwin;Virginia M.-Y. Lee;John Q. Trojanowski.
Nature Reviews Neuroscience (2013)

629 Citations

Neuropathologic substrates of Parkinson disease dementia.

David J. Irwin;Matthew T. White;Jon B. Toledo;Sharon X. Xie.
Annals of Neurology (2012)

401 Citations

Association of Cerebrospinal Fluid β-Amyloid 1-42, T-tau, P-tau181, and α-Synuclein Levels With Clinical Features of Drug-Naive Patients With Early Parkinson Disease

Ju-Hee Kang;Ju-Hee Kang;David J. Irwin;Alice S. Chen-Plotkin;Andrew Siderowf.
JAMA Neurology (2013)

299 Citations

APOE ε4 increases risk for dementia in pure synucleinopathies.

Debby Tsuang;James B. Leverenz;Oscar L. Lopez;Ronald L. Hamilton.
JAMA Neurology (2013)

267 Citations

Neuropathological and Genetic Correlates of Survival and Dementia Onset in Synucleinopathies: A Retrospective Analysis

David J Irwin;Murray Grossman;Daniel Weintraub;Daniel Weintraub;Howard I Hurtig.
Lancet Neurology (2017)

254 Citations

Sequential distribution of pTDP-43 pathology in behavioral variant frontotemporal dementia (bvFTD)

Johannes Brettschneider;Johannes Brettschneider;Kelly Del Tredici;David J. Irwin;Murray Grossman.
Acta Neuropathologica (2014)

213 Citations

Neurodegenerative disease concomitant proteinopathies are prevalent, age-related and APOE4-associated

John L Robinson;Edward B Lee;Sharon X Xie;Lior Rennert.
Brain (2018)

201 Citations

Pattern of ubiquilin pathology in ALS and FTLD indicates presence of C9ORF72 hexanucleotide expansion

Johannes Brettschneider;Johannes Brettschneider;Vivianna M. Van Deerlin;John L. Robinson;Linda Kwong.
Acta Neuropathologica (2012)

199 Citations

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