His scientific interests lie mostly in Pathology, Alzheimer's disease, Amyloid, Cerebral amyloid angiopathy and Genetics. His Pathology study combines topics from a wide range of disciplines, such as Neuropil, Transgene and Ischemia. The various areas that Matthew P. Frosch examines in his Alzheimer's disease study include Frontotemporal lobar degeneration, Dementia and Neuroscience.
His research integrates issues of Clinical trial, Amyloid precursor protein, Insulin-degrading enzyme, Cognitive decline and Positron emission tomography in his study of Amyloid. His Cerebral amyloid angiopathy study combines topics in areas such as Magnetic resonance imaging, Hyperintensity and Neuroimaging. His Senile plaques research incorporates elements of Hippocampal sclerosis and Neurodegeneration.
His primary areas of study are Pathology, Alzheimer's disease, Cerebral amyloid angiopathy, Internal medicine and Neuroscience. His Pathology study frequently links to related topics such as Genetically modified mouse. He combines subjects such as Apolipoprotein E and Cell biology with his study of Alzheimer's disease.
Matthew P. Frosch has researched Cerebral amyloid angiopathy in several fields, including Intracerebral hemorrhage and Magnetic resonance imaging, Hyperintensity. He has included themes like Gastroenterology, Endocrinology and Oncology in his Internal medicine study. His Neuroscience study frequently links to adjacent areas such as Disease.
Matthew P. Frosch mostly deals with Pathology, Cerebral amyloid angiopathy, Disease, Internal medicine and Neuroscience. His research on Pathology often connects related areas such as White matter. The concepts of his Cerebral amyloid angiopathy study are interwoven with issues in Magnetic resonance imaging, In vivo and Fibrin.
His Internal medicine research is multidisciplinary, relying on both Gastroenterology and Oncology. In his study, Alzheimer's disease is inextricably linked to Positron emission tomography, which falls within the broad field of Dementia. His research in Amyloid intersects with topics in Amyloid β and Biochemistry.
Matthew P. Frosch spends much of his time researching Pathology, Cerebral amyloid angiopathy, Disease, Long-term potentiation and Magnetic resonance imaging. Many of his research projects under Pathology are closely connected to Monoamine oxidase A with Monoamine oxidase A, tying the diverse disciplines of science together. His Cerebral amyloid angiopathy research incorporates themes from Inferior longitudinal fasciculus, Diffusion MRI, Myelin, Gliosis and Histopathology.
His Disease study incorporates themes from Cancer, Pathological, In vitro, Protein aggregation and Neuroscience. His work deals with themes such as Fibrin, Angiography, Cortex, Neuropathology and Hematoma, which intersect with Magnetic resonance imaging. His biological study deals with issues like Amyloid β, which deal with fields such as Amyloid.
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Neuropathological Alterations in Alzheimer Disease
Alberto Serrano-Pozo;Matthew P. Frosch;Eliezer Masliah;Bradley T. Hyman.
Cold Spring Harbor Perspectives in Medicine (2011)
National Institute on Aging-Alzheimer's Association guidelines for the neuropathologic assessment of Alzheimer's disease
Bradley T. Hyman;Creighton H. Phelps;Thomas G. Beach;Eileen H. Bigio.
Alzheimers & Dementia (2012)
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Insulin-degrading enzyme regulates the levels of insulin, amyloid β-protein, and the β-amyloid precursor protein intracellular domain in vivo
Wesley Farris;Stefan Mansourian;Yang Chang;Loren Lindsley.
Proceedings of the National Academy of Sciences of the United States of America (2003)
National Institute on Aging–Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease: a practical approach
Thomas J. Montine;Creighton H. Phelps;Thomas G. Beach;Eileen H. Bigio.
Acta Neuropathologica (2012)
Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature
Peter T Nelson;Irina Alafuzoff;Eileen H Bigio;Constantin Bouras.
Journal of Neuropathology and Experimental Neurology (2012)
Ubiquitination of a New Form of α-Synuclein by Parkin from Human Brain: Implications for Parkinson's Disease
Hideki Shimura;Michael G. Schlossmacher;Nobutaka Hattori;Matthew P. Frosch.
An Integrative Model of Cellular States, Plasticity, and Genetics for Glioblastoma
Cyril Neftel;Julie Laffy;Mariella G. Filbin;Toshiro Hara.
Enhanced Proteolysis of β-Amyloid in APP Transgenic Mice Prevents Plaque Formation, Secondary Pathology, and Premature Death
Malcolm A Leissring;Wesley Farris;Alice Y Chang;Dominic M Walsh.
A computational atlas of the hippocampal formation using ex vivo, ultra-high resolution MRI: Application to adaptive segmentation of in vivo MRI
Juan Eugenio Iglesias;Jean C. Augustinack;Khoa Nguyen;Christopher M. Player.
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