Avindra Nath mainly focuses on Immunology, Neurotoxicity, Cell biology, Virology and Pathology. His Immunology research is multidisciplinary, incorporating perspectives in Cerebrospinal fluid and Dementia. His work carried out in the field of Neurotoxicity brings together such families of science as NMDA receptor, Viral replication, Gliosis and Pharmacology.
His Cell biology study incorporates themes from Blood–brain barrier, Apoptosis, Programmed cell death, Receptor and Endothelium. Avindra Nath interconnects Regulation of gene expression, Microglia and Neuron in the investigation of issues within Virology. His studies in Pathology integrate themes in fields like JC virus, Progressive multifocal leukoencephalopathy and Antigen.
His primary areas of study are Immunology, Virology, Neurotoxicity, Pathology and Cell biology. His Immunology study combines topics in areas such as Immune reconstitution inflammatory syndrome, Dementia, Acquired immunodeficiency syndrome and Central nervous system. The study of Virology is intertwined with the study of Microglia in a number of ways.
His study looks at the relationship between Neurotoxicity and fields such as Pharmacology, as well as how they intersect with chemical problems. His Pathology research incorporates elements of Magnetic resonance imaging and Progressive multifocal leukoencephalopathy. His study explores the link between Progressive multifocal leukoencephalopathy and topics such as Leukoencephalopathy that cross with problems in JC virus.
Avindra Nath focuses on Virology, Immunology, Disease, Severe acute respiratory syndrome coronavirus 2 and Coronavirus disease 2019. The Virology study combines topics in areas such as Hiv 1 tat, Protease and Coronavirus Infections. Immunology is closely attributed to Neurocognitive in his research.
He has researched Disease in several fields, including Pathophysiology and Pediatrics. His Severe acute respiratory syndrome coronavirus 2 research includes elements of Pandemic and Intensive care medicine. His study looks at the intersection of Coronavirus disease 2019 and topics like 2019-20 coronavirus outbreak with Anosmia, Ageusia and Disease severity.
His primary areas of investigation include Immunology, Virology, Severe acute respiratory syndrome coronavirus 2, Multiple sclerosis and Disease. His work deals with themes such as Neuropathology and Pathological, which intersect with Immunology. His Virology research incorporates themes from Drug development, Guillain-Barre syndrome and Myelitis.
His Severe acute respiratory syndrome coronavirus 2 research also works with subjects such as
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Chemotherapy-Refractory Diffuse Large B-Cell Lymphoma and Indolent B-Cell Malignancies Can Be Effectively Treated With Autologous T Cells Expressing an Anti-CD19 Chimeric Antigen Receptor
James N. Kochenderfer;Mark E. Dudley;Sadik H. Kassim;Robert P.T. Somerville.
Journal of Clinical Oncology (2015)
Cancer regression and neurological toxicity following anti-MAGE-A3 TCR gene therapy.
Richard A. Morgan;Nachimuthu Chinnasamy;Daniel Abate-Daga;Alena Gros.
Journal of Immunotherapy (2013)
A full genome search in multiple sclerosis
G C Ebers;K Kukay;D E Bulman;A D Sadovnick.
Nature Genetics (1996)
TNF neutralization in MS - Results of a randomized, placebo-controlled multicenter study
Arnason Bgw.;G Jacobs;M Hanlon;B H Clay.
Natalizumab-associated progressive multifocal leukoencephalopathy in patients with multiple sclerosis: lessons from 28 cases
David B Clifford;Andrea DeLuca;David M Simpson;Gabriele Arendt.
Lancet Neurology (2010)
Induction of monocyte chemoattractant protein-1 in HIV-1 Tat-stimulated astrocytes and elevation in AIDS dementia
Katherine Conant;Alfredo Garzino-Demo;Avindra Nath;Justin C. McArthur.
Proceedings of the National Academy of Sciences of the United States of America (1998)
PML diagnostic criteria: Consensus statement from the AAN neuroinfectious disease section
Joseph R. Berger;Allen J. Aksamit;David B. Clifford;Larry Davis.
Disruption of neurogenesis by amyloid β-peptide, and perturbed neural progenitor cell homeostasis, in models of Alzheimer's disease
Norman J. Haughey;Avi Nath;Sic L. Chan;A. C. Borchard.
Journal of Neurochemistry (2002)
HIV-1 Protein Tat Induces Apoptosis of Hippocampal Neurons by a Mechanism Involving Caspase Activation, Calcium Overload, and Oxidative Stress
Inna I. Kruman;Avindra Nath;Mark P. Mattson.
Experimental Neurology (1998)
Uptake of HIV-1 tat protein mediated by low-density lipoprotein receptor-related protein disrupts the neuronal metabolic balance of the receptor ligands.
Ying Liu;Melina Jones;Cynthia M. Hingtgen;Guojun Bu.
Nature Medicine (2000)
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