Eliseo A. Eugenin mostly deals with Immunology, Gap junction, Blood–brain barrier, Cell biology and Chemokine. His Immunology research includes themes of Astrocyte and Virology. His Virology research is multidisciplinary, incorporating elements of Neuroinflammation and Immune system.
His Gap junction study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha and Molecular biology. His studies in Blood–brain barrier integrate themes in fields like Acquired immunodeficiency syndrome, Microglia and Monocyte. The concepts of his Cell biology study are interwoven with issues in NMDA receptor and Adenosine triphosphate.
His scientific interests lie mostly in Immunology, Cell biology, Virology, Gap junction and Inflammation. Much of his study explores Immunology relationship to Blood–brain barrier. His Cell biology research incorporates elements of Apoptosis, Downregulation and upregulation and Transactivation.
Eliseo A. Eugenin combines subjects such as Receptor, Acquired immunodeficiency syndrome and Immune system with his study of Virology. In general Gap junction, his work in Connexin and Lucifer yellow is often linked to Microvesicles linking many areas of study. Eliseo A. Eugenin has researched Inflammation in several fields, including Tumor necrosis factor alpha, Purinergic receptor and Macrophage.
Eliseo A. Eugenin spends much of his time researching Virology, Virus, Immunology, Cell biology and Astrocyte. The Virology study combines topics in areas such as Acquired immunodeficiency syndrome, HIV Proteins, Pathogenesis and Transactivation. His work on Viral replication as part of general Virus study is frequently connected to Alternative complement pathway, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His Immunology research is mostly focused on the topic Inflammation. He has included themes like Bystander effect and Glycogenolysis in his Cell biology study. His study in Astrocyte is interdisciplinary in nature, drawing from both Microglia and Pannexin.
His primary areas of study are Virology, Immunology, HIV Proteins, Acquired immunodeficiency syndrome and Transmission. The study incorporates disciplines such as T cell, Immune system, In vitro and Platelet in addition to Virology. His Immunology research is multidisciplinary, relying on both Intracellular, Second messenger system, Peripheral blood mononuclear cell, Cognitive decline and Biomarker.
His Peripheral blood mononuclear cell research integrates issues from Secretion, Spleen, Disease, Human brain and Astrocytoma. His study connects Virus and Spleen. His Virus research includes elements of Hemostasis and Central nervous system, Astrocyte, Blood–brain barrier.
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Metabolic inhibition induces opening of unapposed connexin 43 gap junction hemichannels and reduces gap junctional communication in cortical astrocytes in culture
Jorge E. Contreras;Helmut A. Sánchez;Eliseo A. Eugenín;Dina Speidel.
Proceedings of the National Academy of Sciences of the United States of America (2002)
CCL2/monocyte chemoattractant protein-1 mediates enhanced transmigration of human immunodeficiency virus (HIV)-infected leukocytes across the blood-brain barrier : A potential mechanism of HIV-CNS invasion and NeuroAIDS
Eliseo A. Eugenin;Kristin Osiecki;Lillie Lopez;Harris Goldstein.
The Journal of Neuroscience (2006)
MCP-1 (CCL2) protects human neurons and astrocytes from NMDA or HIV-tat-induced apoptosis.
Eliseo Eugenin;T. G. D'Aversa;L. Lopez;T. M. Calderon.
Journal of Neurochemistry (2003)
Microglial stimulation of glioblastoma invasion involves epidermal growth factor receptor (EGFR) and colony stimulating factor 1 receptor (CSF-1R) signaling.
Salvatore J Coniglio;Eliseo Eugenin;Kostantin Dobrenis;E Richard Stanley.
Molecular Medicine (2012)
Human immunodeficiency virus infection of human astrocytes disrupts blood-brain barrier integrity by a gap junction-dependent mechanism.
Eliseo A. Eugenin;Janice E. Clements;M. Christine Zink;Joan W. Berman.
The Journal of Neuroscience (2011)
Microglia at brain stab wounds express connexin 43 and in vitro form functional gap junctions after treatment with interferon-γ and tumor necrosis factor-α
Eliseo A. Eugenín;Dominik Eckardt;Martin Theis;Klaus Willecke.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Tunneling nanotubes (TNT) are induced by HIV-infection of macrophages: a potential mechanism for intercellular HIV trafficking.
Eliseo Eugenin;P. J. Gaskill;J. W. Berman.
Cellular Immunology (2009)
TNF-α Plus IFN-γ Induce Connexin43 Expression and Formation of Gap Junctions Between Human Monocytes/Macrophages That Enhance Physiological Responses
Eliseo A. Eugenín;Eliseo A. Eugenín;María C. Brañes;Joan W. Berman;Juan C. Sáez;Juan C. Sáez.
Journal of Immunology (2003)
HIV tat and neurotoxicity
J. E. King;Eliseo Eugenin;C. M. Buckner;J. W. Berman.
Microbes and Infection (2006)
HIV-tat induces formation of an LRP–PSD-95– NMDAR–nNOS complex that promotes apoptosis in neurons and astrocytes
Eliseo A. Eugenin;Jessie E. King;Avindra Nath;Tina M. Calderon.
Proceedings of the National Academy of Sciences of the United States of America (2007)
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