What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Biochemistry
His primary areas of investigation include Cell biology, Connexin, Gap junction, Connexon and Intracellular.
His Cell biology study combines topics in areas such as Apoptosis and Programmed cell death.
His Connexin research includes elements of Purinergic receptor, Endocrinology, Calcium in biology, Gliotransmitter and Ion channel.
His work carried out in the field of Gap junction brings together such families of science as Cell signaling, Transfection, Paracrine signalling and Cytoplasm.
His biological study spans a wide range of topics, including Reperfusion injury, Cardioprotection, Ischemia and Membrane potential.
His study looks at the relationship between Extracellular and topics such as Calcium, which overlap with Monocyte, Reactive oxygen species and Andrology.
His most cited work include:
- Engaging neuroscience to advance translational research in brain barrier biology (394 citations)
- The gap junction cellular internet: connexin hemichannels enter the signalling limelight. (349 citations)
- Intracellular calcium changes trigger connexin 32 hemichannel opening (217 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Connexin, Gap junction, Intracellular and Internal medicine.
His studies in Cell biology integrate themes in fields like Apoptosis, Programmed cell death and Calcium.
His Connexin research includes themes of Inflammation, Calcium in biology and Neuroscience.
His study in Gap junction is interdisciplinary in nature, drawing from both Cell signaling, Signal transduction, Paracrine signalling, Membrane potential and Pharmacology.
His Intracellular research incorporates themes from Biophysics and Cytoplasm.
His Internal medicine research is multidisciplinary, incorporating perspectives in Endocrinology and Cardiology.
He most often published in these fields:
- Cell biology (44.10%)
- Connexin (32.64%)
- Gap junction (25.00%)
What were the highlights of his more recent work (between 2015-2021)?
- Connexin (32.64%)
- Cell biology (44.10%)
- Gap junction (25.00%)
In recent papers he was focusing on the following fields of study:
Connexin, Cell biology, Gap junction, Neuroscience and Intracellular are his primary areas of study.
His Connexin research incorporates elements of Oxidative stress, Paracrine signalling, Inflammation, Biophysics and Patch clamp.
His Cell biology research is multidisciplinary, relying on both Autophagy, Bystander effect, Calcium, Inositol trisphosphate receptor and Programmed cell death.
The study incorporates disciplines such as Extracellular, Endothelial stem cell and Blastocyst in addition to Programmed cell death.
His study explores the link between Gap junction and topics such as Pharmacology that cross with problems in In vivo and Anticonvulsant.
His Neuroscience research is multidisciplinary, incorporating elements of Neurotransmission, Pannexin and Homeostasis.
Between 2015 and 2021, his most popular works were:
- Vaccination with Necroptotic Cancer Cells Induces Efficient Anti-tumor Immunity. (155 citations)
- Connexins in Cardiovascular and Neurovascular Health and Disease: Pharmacological Implications (113 citations)
- Mitochondrial Cx43 hemichannels contribute to mitochondrial calcium entry and cell death in the heart. (70 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Cancer
Luc Leybaert mainly focuses on Connexin, Gap junction, Neuroscience, Cell biology and Inflammation.
He interconnects Synaptic plasticity, Spontaneous alternation, Paracrine signalling and Function in the investigation of issues within Connexin.
His Gap junction study improves the overall literature in Intracellular.
His Neuroscience study combines topics in areas such as Therapeutic targeting, Neurotransmission and Peptide mimetic.
Luc Leybaert studies Cell biology, focusing on Pannexin in particular.
His Inflammation research focuses on Signal transduction and how it connects with Protein kinase A and Casein kinase 1.
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