What is he best known for?
The fields of study he is best known for:
- Amino acid
- Biochemistry
- Apoptosis
His primary scientific interests are in Cell biology, Receptor, Endoplasmic reticulum, Calcium signaling and Inositol.
His Cell biology research incorporates elements of Autophagy, Apoptosis, Programmed cell death and Voltage-dependent calcium channel.
He has researched Programmed cell death in several fields, including Autophagosome, Immunoprecipitation, Sequestosome 1, Computational biology and Physiology.
The study incorporates disciplines such as Calcium, Phosphorylation and COS cells in addition to Receptor.
His research integrates issues of Subcellular localization and Activator in his study of Endoplasmic reticulum.
The concepts of his Inositol study are interwoven with issues in Amino acid, Transmembrane domain, Molecular biology and Binding site.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Endoplasmic-Reticulum Calcium Depletion and Disease (266 citations)
What are the main themes of his work throughout his whole career to date?
Jan B. Parys focuses on Cell biology, Receptor, Inositol, Endoplasmic reticulum and Inositol trisphosphate receptor.
His work carried out in the field of Cell biology brings together such families of science as Autophagy and Programmed cell death.
His Receptor research is multidisciplinary, relying on both Endocrinology, Binding site and Voltage-dependent calcium channel.
The Inositol study combines topics in areas such as Molecular biology, Stimulation, Kinase and Adenosine triphosphate.
His Endoplasmic reticulum study integrates concerns from other disciplines, such as Apoptosis and Mitochondrion.
His study in Inositol trisphosphate receptor is interdisciplinary in nature, drawing from both Inositol trisphosphate, Signal transduction and Function.
He most often published in these fields:
- Cell biology (59.67%)
- Receptor (38.00%)
- Inositol (36.67%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (59.67%)
- Endoplasmic reticulum (31.67%)
- Programmed cell death (14.67%)
In recent papers he was focusing on the following fields of study:
Jan B. Parys spends much of his time researching Cell biology, Endoplasmic reticulum, Programmed cell death, Mitochondrion and Receptor.
His study in the fields of Intracellular under the domain of Cell biology overlaps with other disciplines such as Protein family.
Jan B. Parys combines subjects such as Autophagy, Kinase and Calcium signaling with his study of Endoplasmic reticulum.
His Autophagy research is multidisciplinary, incorporating elements of Computational biology and Protein kinase A.
His Programmed cell death research incorporates themes from Autolysosome and Peptide.
His work on Ryanodine receptor as part of general Receptor study is frequently linked to Gene isoform, bridging the gap between disciplines.
Between 2017 and 2021, his most popular works were:
- Emerging molecular mechanisms in chemotherapy: Ca 2+ signaling at the mitochondria-associated endoplasmic reticulum membranes (51 citations)
- Constitutive IP3 signaling underlies the sensitivity of B-cell cancers to the Bcl-2/IP3 receptor disruptor BIRD-2. (48 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)
In his most recent research, the most cited papers focused on:
- Amino acid
- Biochemistry
- Apoptosis
The scientist’s investigation covers issues in Cell biology, Endoplasmic reticulum, Receptor, Programmed cell death and Mitochondrion.
Jan B. Parys frequently studies issues relating to Inositol trisphosphate receptor and Cell biology.
His study looks at the intersection of Endoplasmic reticulum and topics like Calcium signaling with Kinase, Cell fate determination and Suppressor.
His Receptor research integrates issues from Amino acid, Point mutation and Bcl-2-associated X protein.
He has included themes like Autophagy, Peptide and Autolysosome in his Programmed cell death study.
His work deals with themes such as Computational biology and Multicellular organism, which intersect with Autophagy.
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