Receptor, Cell biology, Endocrinology, Internal medicine and Biophysics are his primary areas of study. His studies in Cell biology integrate themes in fields like Cell morphology and Ion transporter. The study incorporates disciplines such as Cholecystokinin, Microfluorimetry, Calcium and Fura-2 in addition to Endocrinology.
The concepts of his Internal medicine study are interwoven with issues in Chloride channel and Membrane potential. His Biophysics study combines topics in areas such as Ryanodine receptor and Patch clamp. His Inositol research incorporates themes from Carbachol and Phosphorylation.
His primary areas of study are Cell biology, Receptor, Inositol, Endocrinology and Internal medicine. His Biochemistry research extends to Cell biology, which is thematically connected. His research in Biochemistry intersects with topics in Cell culture and Biophysics.
His Receptor study combines topics in areas such as Calcium, Mitochondrion, Phosphorylation and Gene isoform. His study in Inositol is interdisciplinary in nature, drawing from both Wild type, Mutant, Transfection, Proteolysis and Adenosine triphosphate. David I. Yule focuses mostly in the field of Endocrinology, narrowing it down to matters related to Cholecystokinin and, in some cases, Acetylcholine.
His main research concerns Cell biology, Receptor, Endoplasmic reticulum, Inositol and Intracellular. His biological study spans a wide range of topics, including Agonist, Secretion and Calcium. His Receptor research is multidisciplinary, relying on both Protease, Acetylcholine, Cytosol, Mitochondrion and Gene isoform.
His work in Protease addresses subjects such as Thrombin, which are connected to disciplines such as Endocrinology. His studies deal with areas such as Stimulation, NFAT and Calcium signaling as well as Endoplasmic reticulum. His Inositol research is multidisciplinary, incorporating perspectives in Cell culture, Biophysics, Proteolysis, Phosphorylation and Effector.
His scientific interests lie mostly in Cell biology, Receptor, Inositol, Gene isoform and Endoplasmic reticulum. Cell biology is closely attributed to Inositol phosphate in his study. His Receptor research includes themes of Calcium and Mitochondrion.
When carried out as part of a general Calcium research project, his work on Calcium flux and Calcium signaling is frequently linked to work in Context, therefore connecting diverse disciplines of study. His Inositol research integrates issues from Cell culture, Biophysics and Cytosol. His Endoplasmic reticulum study integrates concerns from other disciplines, such as Inflammation, Endothelium, Intracellular and Endothelial dysfunction.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Calcium and mitochondria
Thomas E. Gunter;David I. Yule;Karlene K. Gunter;Roman A. Eliseev.
FEBS Letters (2004)
Regulation of fluid and electrolyte secretion in salivary gland acinar cells.
James E. Melvin;David Yule;Trevor Shuttleworth;Ted Begenisich.
Annual Review of Physiology (2005)
Cytoplasmic Ca2+ oscillations evoked by receptor stimulation, G-protein activation, internal application of inositol trisphosphate or Ca2+: simultaneous microfluorimetry and Ca2+ dependent Cl- current recording in single pancreatic acinar cells.
Y.V. Osipchuk;M. Wakui;D.I. Yule;D.V. Gallacher.
The EMBO Journal (1990)
U73122 inhibits Ca2+ oscillations in response to cholecystokinin and carbachol but not to JMV-180 in rat pancreatic acinar cells.
David I. Yule;John A. Williams.
Journal of Biological Chemistry (1992)
Identification of a Ryanodine Receptor in Rat Heart Mitochondria
Gisela Beutner;Virendra K. Sharma;David R. Giovannucci;David I. Yule.
Journal of Biological Chemistry (2001)
Evidence that zymogen granules are not a physiologically relevant calcium pool. Defining the distribution of inositol 1,4,5-trisphosphate receptors in pancreatic acinar cells.
David I. Yule;Stephen A. Ernst;Hirohide Ohnishi;Richard J.H. Wojcikiewicz.
Journal of Biological Chemistry (1997)
Calcium wave propagation in pancreatic acinar cells: functional interaction of inositol 1,4,5-trisphosphate receptors, ryanodine receptors, and mitochondria.
Stephen V. Straub;David R. Giovannucci;David I. Yule.
The Journal of General Physiology (2000)
Phosphorylation of inositol 1,4,5-trisphosphate receptors in parotid acinar cells. A mechanism for the synergistic effects of cAMP on Ca2+ signaling.
Jason I.E. Bruce;Trevor J. Shuttleworth;David R. Giovannucci;David I. Yule.
Journal of Biological Chemistry (2002)
Control of calcium oscillations by membrane fluxes
J Sneyd;KT Tsaneva-Atanasova;DI Yule;JL Thompson.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Crosstalk between cAMP and Ca2+ signaling in non-excitable cells.
Jason I.E Bruce;Stephen V Straub;David I Yule.
Cell Calcium (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Michigan–Ann Arbor
Thomas Jefferson University
Cardiff University
KU Leuven
KU Leuven
ShanghaiTech University
Thomas Jefferson University
Pennsylvania State University
National Institutes of Health
KU Leuven
Instituto Superior Técnico
Rakuten (Japan)
Fırat University
Czech Academy of Sciences
Simon Fraser University
University of California, San Diego
Radboud University Nijmegen
Osaka Metropolitan University
University of Verona
University of Montreal
Mayo Clinic
Instituto de Salud Carlos III
Old Dominion University
Centre for Mental Health
University of Pittsburgh
Australian National University