His primary areas of investigation include Endoplasmic reticulum, Cell biology, Biochemistry, Biophysics and Calcium signaling. His research in Endoplasmic reticulum tackles topics such as Calcium metabolism which are related to areas like Patch clamp, Calcium pump, Intracellular and Endocrinology. His study in the field of Inositol trisphosphate, Nicotinic acid adenine dinucleotide phosphate and Inositol trisphosphate receptor is also linked to topics like Cyclic ADP-ribose.
His Biophysics research focuses on subjects like Acetylcholine, which are linked to Ionomycin and Ion transporter. Calcium signaling is a subfield of Calcium that Alexei V. Tepikin tackles. His research integrates issues of Endocytic cycle and Vacuole in his study of Calcium.
His primary scientific interests are in Cell biology, Calcium, Endoplasmic reticulum, Biochemistry and Biophysics. His Cell biology research incorporates elements of Acinar cell and Pancreas. His Calcium study which covers Endocrinology that intersects with Receptor, Pancreatitis, Cholecystokinin and Calcium ATPase.
The Endoplasmic reticulum study combines topics in areas such as Cell and Organelle. His work in the fields of Biochemistry, such as Inositol trisphosphate, Membrane potential, Fatty acid and Pancreatic acinar cells, intersects with other areas such as Cyclic ADP-ribose. The various areas that Alexei V. Tepikin examines in his Biophysics study include Extracellular, Stimulation, Acetylcholine and Intracellular.
Alexei V. Tepikin focuses on Cell biology, Pharmacology, Acute pancreatitis, Bioenergetics and Acinar cell. His Cell biology research is multidisciplinary, incorporating perspectives in Endocytic cycle and Apoptosis. His Pharmacology study deals with Pancreatitis intersecting with Endocrinology.
His biological study deals with issues like Calcium, which deal with fields such as Toxicity. His work focuses on many connections between Acinar cell and other disciplines, such as Mitochondrial permeability transition pore, that overlap with his field of interest in Necrosis, Ryanodine receptor and Inositol trisphosphate. Reactive oxygen species is a subfield of Biochemistry that Alexei V. Tepikin studies.
The scientist’s investigation covers issues in Acinar cell, Internal medicine, Endocrinology, Mitochondrial permeability transition pore and Pancreatitis. His Acinar cell study integrates concerns from other disciplines, such as Thapsigargin and Cell biology. His study in Cell biology is interdisciplinary in nature, drawing from both Necrosis, Oxidative stress and Apoptosis.
His Internal medicine study frequently draws parallels with other fields, such as Mitochondrion. His Inositol research extends to Endocrinology, which is thematically connected. His Mitochondrial permeability transition pore study incorporates themes from Inositol trisphosphate, Pharmacology and Ryanodine receptor.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
ATP-DEPENDENT ACCUMULATION AND INOSITOL TRISPHOSPHATE- OR CYCLIC ADP-RIBOSE-MEDIATED RELEASE OF CA2+ FROM THE NUCLEAR ENVELOPE
Oleg V Gerasimenko;Julia V Gerasimenko;Alexei V Tepikin;Ole H Petersen.
Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate-evoked local cytosolic Ca2+ signals
Hanna Tinel;Jose M. Cancela;Hideo Mogami;Julia V. Gerasimenko.
The EMBO Journal (1999)
Perinuclear, perigranular and sub‐plasmalemmal mitochondria have distinct functions in the regulation of cellular calcium transport
Myoung Kyu Park;Myoung Kyu Park;Michael C. Ashby;Gul Erdemli;Ole Holger Petersen.
The EMBO Journal (2001)
Menadione-induced reactive oxygen species generation via redox cycling promotes apoptosis of murine pancreatic acinar cells.
Dabid N. Criddle;Stuart Gillies;Heidi K. Baumgartner-Wilson;Mohammed Jaffar;Mohammed Jaffar.
Journal of Biological Chemistry (2006)
Polarized Calcium Signaling in Exocrine Gland Cells
Ole H Petersen;Alexei V Tepikin.
Annual Review of Physiology (2008)
Calcium uptake via endocytosis with rapid release from acidifying endosomes
Julia V. Gerasimenko;Alexei V. Tepikin;Ole H. Petersen;Oleg V. Gerasimenko.
Current Biology (1998)
Ca2+ flow via tunnels in polarized cells: recharging of apical Ca2+ stores by focal Ca2+ entry through basal membrane patch.
Hideo Mogami;Kyoko Nakano;Alexei V Tepikin;Ole H Petersen.
Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis
David N. Criddle;John Murphy;Gregorio Fistetto;Stephanie Barrow.
The endoplasmic reticulum as one continuous Ca2+ pool: visualization of rapid Ca2+ movements and equilibration
Myoung Kyu Park;Ole Holger Petersen;Alexei V. Tepikin.
The EMBO Journal (2000)
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