D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 66 Citations 21,853 159 World Ranking 5452 National Ranking 413

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Biochemistry
  • Cell membrane

His primary areas of investigation include Endoplasmic reticulum, Cell biology, Biochemistry, Biophysics and Calcium signaling. His research in Endoplasmic reticulum tackles topics such as Calcium metabolism which are related to areas like Patch clamp, Calcium pump, Intracellular and Endocrinology. His study in the field of Inositol trisphosphate, Nicotinic acid adenine dinucleotide phosphate and Inositol trisphosphate receptor is also linked to topics like Cyclic ADP-ribose.

His Biophysics research focuses on subjects like Acetylcholine, which are linked to Ionomycin and Ion transporter. Calcium signaling is a subfield of Calcium that Alexei V. Tepikin tackles. His research integrates issues of Endocytic cycle and Vacuole in his study of Calcium.

His most cited work include:

  • ATP-DEPENDENT ACCUMULATION AND INOSITOL TRISPHOSPHATE- OR CYCLIC ADP-RIBOSE-MEDIATED RELEASE OF CA2+ FROM THE NUCLEAR ENVELOPE (332 citations)
  • Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate‐evoked local cytosolic Ca2+ signals (325 citations)
  • Perinuclear, perigranular and sub‐plasmalemmal mitochondria have distinct functions in the regulation of cellular calcium transport (271 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cell biology, Calcium, Endoplasmic reticulum, Biochemistry and Biophysics. His Cell biology research incorporates elements of Acinar cell and Pancreas. His Calcium study which covers Endocrinology that intersects with Receptor, Pancreatitis, Cholecystokinin and Calcium ATPase.

The Endoplasmic reticulum study combines topics in areas such as Cell and Organelle. His work in the fields of Biochemistry, such as Inositol trisphosphate, Membrane potential, Fatty acid and Pancreatic acinar cells, intersects with other areas such as Cyclic ADP-ribose. The various areas that Alexei V. Tepikin examines in his Biophysics study include Extracellular, Stimulation, Acetylcholine and Intracellular.

He most often published in these fields:

  • Cell biology (40.45%)
  • Calcium (28.09%)
  • Endoplasmic reticulum (26.97%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cell biology (40.45%)
  • Pharmacology (8.43%)
  • Acute pancreatitis (8.99%)

In recent papers he was focusing on the following fields of study:

Alexei V. Tepikin focuses on Cell biology, Pharmacology, Acute pancreatitis, Bioenergetics and Acinar cell. His Cell biology research is multidisciplinary, incorporating perspectives in Endocytic cycle and Apoptosis. His Pharmacology study deals with Pancreatitis intersecting with Endocrinology.

His biological study deals with issues like Calcium, which deal with fields such as Toxicity. His work focuses on many connections between Acinar cell and other disciplines, such as Mitochondrial permeability transition pore, that overlap with his field of interest in Necrosis, Ryanodine receptor and Inositol trisphosphate. Reactive oxygen species is a subfield of Biochemistry that Alexei V. Tepikin studies.

Between 2013 and 2021, his most popular works were:

  • Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP (106 citations)
  • Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP (106 citations)
  • Inhibitors of ORAI1 Prevent Cytosolic Calcium-Associated Injury of Human Pancreatic Acinar Cells and Acute Pancreatitis in 3 Mouse Models. (101 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cell membrane
  • Biochemistry

The scientist’s investigation covers issues in Acinar cell, Internal medicine, Endocrinology, Mitochondrial permeability transition pore and Pancreatitis. His Acinar cell study integrates concerns from other disciplines, such as Thapsigargin and Cell biology. His study in Cell biology is interdisciplinary in nature, drawing from both Necrosis, Oxidative stress and Apoptosis.

His Internal medicine study frequently draws parallels with other fields, such as Mitochondrion. His Inositol research extends to Endocrinology, which is thematically connected. His Mitochondrial permeability transition pore study incorporates themes from Inositol trisphosphate, Pharmacology and Ryanodine receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

ATP-DEPENDENT ACCUMULATION AND INOSITOL TRISPHOSPHATE- OR CYCLIC ADP-RIBOSE-MEDIATED RELEASE OF CA2+ FROM THE NUCLEAR ENVELOPE

Oleg V Gerasimenko;Julia V Gerasimenko;Alexei V Tepikin;Ole H Petersen.
Cell (1995)

450 Citations

Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate-evoked local cytosolic Ca2+ signals

Hanna Tinel;Jose M. Cancela;Hideo Mogami;Julia V. Gerasimenko.
The EMBO Journal (1999)

418 Citations

Perinuclear, perigranular and sub‐plasmalemmal mitochondria have distinct functions in the regulation of cellular calcium transport

Myoung Kyu Park;Myoung Kyu Park;Michael C. Ashby;Gul Erdemli;Ole Holger Petersen.
The EMBO Journal (2001)

355 Citations

Menadione-induced reactive oxygen species generation via redox cycling promotes apoptosis of murine pancreatic acinar cells.

Dabid N. Criddle;Stuart Gillies;Heidi K. Baumgartner-Wilson;Mohammed Jaffar;Mohammed Jaffar.
Journal of Biological Chemistry (2006)

349 Citations

Polarized Calcium Signaling in Exocrine Gland Cells

Ole H Petersen;Alexei V Tepikin.
Annual Review of Physiology (2008)

302 Citations

Calcium uptake via endocytosis with rapid release from acidifying endosomes

Julia V. Gerasimenko;Alexei V. Tepikin;Ole H. Petersen;Oleg V. Gerasimenko.
Current Biology (1998)

282 Citations

Ca2+ flow via tunnels in polarized cells: recharging of apical Ca2+ stores by focal Ca2+ entry through basal membrane patch.

Hideo Mogami;Kyoko Nakano;Alexei V Tepikin;Ole H Petersen.
Cell (1997)

281 Citations

Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis

David N. Criddle;John Murphy;Gregorio Fistetto;Stephanie Barrow.
Gastroenterology (2006)

274 Citations

The endoplasmic reticulum as one continuous Ca2+ pool: visualization of rapid Ca2+ movements and equilibration

Myoung Kyu Park;Ole Holger Petersen;Alexei V. Tepikin.
The EMBO Journal (2000)

272 Citations

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