D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 77 Citations 19,362 316 World Ranking 11061 National Ranking 5880

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

The scientist’s investigation covers issues in Internal medicine, Endocrinology, Pancreatitis, Pancreas and Pancreatic cancer. His Internal medicine research incorporates themes from Gastroenterology, Trypsinogen and Intracellular. His Endocrinology study incorporates themes from Cholecystokinin, Apoptosis, Calcium and Cell biology.

Stephen J. Pandol has researched Pancreatitis in several fields, including Cytokine, Inflammation, Acute pancreatitis, Pancreatic disease and Mitochondrion. His research in Pancreas tackles topics such as Hepatic stellate cell which are related to areas like Immunology and Fibrosis. His Pancreatic cancer study integrates concerns from other disciplines, such as Cancer cell, Cancer research, Carcinogenesis and Pathology.

His most cited work include:

  • Acute Pancreatitis: Bench to the Bedside (508 citations)
  • The pancreatic stellate cell: a star on the rise in pancreatic diseases. (468 citations)
  • PANCREATIC ACINAR CELLS PRODUCE, RELEASE, AND RESPOND TO TUMOR NECROSIS FACTOR-ALPHA : ROLE IN REGULATING CELL DEATH AND PANCREATITIS (340 citations)

What are the main themes of his work throughout his whole career to date?

Stephen J. Pandol mainly focuses on Internal medicine, Pancreatitis, Endocrinology, Cancer research and Pancreatic cancer. His Internal medicine research integrates issues from Gastroenterology, Diabetes mellitus and Oncology. As part of one scientific family, Stephen J. Pandol deals mainly with the area of Pancreatitis, narrowing it down to issues related to the Acute pancreatitis, and often Intensive care medicine.

His studies deal with areas such as Cholecystokinin, Calcium and Intracellular as well as Endocrinology. His study explores the link between Cancer research and topics such as Apoptosis that cross with problems in Mitochondrion. His Acinar cell research includes themes of Secretion and Endoplasmic reticulum, Cell biology.

He most often published in these fields:

  • Internal medicine (50.37%)
  • Pancreatitis (38.20%)
  • Endocrinology (32.77%)

What were the highlights of his more recent work (between 2018-2021)?

  • Pancreatitis (38.20%)
  • Internal medicine (50.37%)
  • Pancreatic cancer (29.03%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Pancreatitis, Internal medicine, Pancreatic cancer, Cancer research and Acute pancreatitis. The Pancreatitis study combines topics in areas such as Intracellular, PROTEIN KINASE D, Pharmacology and Pancreas. His Internal medicine research includes elements of Diabetes mellitus and Oncology.

His studies in Pancreatic cancer integrate themes in fields like Epidemiology, Incidence, Relative risk, Disease and Risk factor. Stephen J. Pandol interconnects Hepatic stellate cell, Cancer cell, Tumor microenvironment, Metastasis and GSK3B in the investigation of issues within Cancer research. The various areas that Stephen J. Pandol examines in his Acute pancreatitis study include Necrosis, Trypsinogen activation, Retrospective cohort study and Intensive care medicine.

Between 2018 and 2021, his most popular works were:

  • Acute Pancreatitis: A Multifaceted Set of Organelle and Cellular Interactions (46 citations)
  • TRPV4 channel opening mediates pressure-induced pancreatitis initiated by Piezo1 activation (32 citations)
  • Early detection of pancreatic cancer (28 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Acute Pancreatitis: Bench to the Bedside

Stephen J. Pandol;Ashok K. Saluja;Clement W. Imrie;Peter A. Banks.
Gastroenterology (2007)

732 Citations

The pancreatic stellate cell: a star on the rise in pancreatic diseases.

M. Bishr Omary;Aurelia Lugea;Anson W. Lowe;Stephen J. Pandol.
Journal of Clinical Investigation (2007)

672 Citations

Early NF-κB activation is associated with hormone-induced pancreatitis

Ilya Gukovsky;Anna S. Gukovskaya;Thane A. Blinman;Vjekoslav Zaninovic.
American Journal of Physiology-gastrointestinal and Liver Physiology (1998)

503 Citations

PANCREATIC ACINAR CELLS PRODUCE, RELEASE, AND RESPOND TO TUMOR NECROSIS FACTOR-ALPHA : ROLE IN REGULATING CELL DEATH AND PANCREATITIS

A S Gukovskaya;I Gukovsky;V Zaninovic;M Song.
Journal of Clinical Investigation (1997)

466 Citations

Reactive Oxygen Species Produced by NAD(P)H Oxidase Inhibit Apoptosis in Pancreatic Cancer Cells

Eva C. Vaquero;Mouad Edderkaoui;Stephen J. Pandol;Ilya Gukovsky.
Journal of Biological Chemistry (2004)

416 Citations

A Starring Role for Stellate Cells in the Pancreatic Cancer Microenvironment

Minoti V. Apte;Jeremy S. Wilson;Aurelia Lugea;Aurelia Lugea;Stephen J. Pandol;Stephen J. Pandol;Stephen J. Pandol.
Gastroenterology (2013)

348 Citations

Hormone-evoked calcium release from intracellular stores is a quantal process

Shmuel Muallem;S. J. Pandol;T. G. Beeker.
Journal of Biological Chemistry (1989)

343 Citations

Food‐derived polyphenols inhibit pancreatic cancer growth through mitochondrial cytochrome C release and apoptosis

Michelle Mouria;Anna S. Gukovskaya;Yoon Jung;Peter Buechler.
International Journal of Cancer (2002)

339 Citations

The role of neutrophils and platelet-activating factor in mediating experimental pancreatitis

D Sandoval;A Gukovskaya;P Reavey;S Gukovsky.
Gastroenterology (1996)

309 Citations

Mechanism of chloride secretion induced by carbachol in a colonic epithelial cell line.

K Dharmsathaphorn;S J Pandol.
Journal of Clinical Investigation (1986)

304 Citations

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