Kim E. Barrett mostly deals with Secretion, Cell biology, Endocrinology, Internal medicine and Immunology. His Secretion research is multidisciplinary, incorporating elements of Epithelium, Intestinal epithelium, Intestinal mucosa and Microbiology. Kim E. Barrett interconnects Calcium metabolism and Cytosol in the investigation of issues within Cell biology.
His study explores the link between Endocrinology and topics such as Kinase that cross with problems in Cell growth, Protein kinase B and Transforming growth factor. His research in Immunology intersects with topics in Barrier function, Intestinal physiology, Inflammatory bowel disease and Depression. His Biochemistry research includes themes of Biophysics and Calcium.
Kim E. Barrett mainly focuses on Cell biology, Internal medicine, Endocrinology, Secretion and Immunology. In his research on the topic of Cell biology, Phosphatidylinositol is strongly related with Epidermal growth factor. In most of his Internal medicine studies, his work intersects topics such as Gastroenterology.
His research integrates issues of Receptor and Calcium in his study of Endocrinology. As a member of one scientific family, Kim E. Barrett mostly works in the field of Secretion, focusing on Intestinal epithelium and, on occasion, Pathogenesis. The study incorporates disciplines such as Immunoglobulin E, Degranulation, Pharmacology and Histamine H4 receptor in addition to Mast cell.
His primary areas of study are Cell biology, Diarrhea, Immunology, Internal medicine and Cancer research. The various areas that he examines in his Cell biology study include Nod, Pathogenesis and Gut–brain axis. His Immunology research incorporates elements of Intestinal epithelium and MEDLINE.
His work deals with themes such as Gastroenterology, Endocrinology, Real-time polymerase chain reaction and Intracellular pH, which intersect with Internal medicine. Kim E. Barrett has researched Endocrinology in several fields, including FODMAP and Intensive care medicine. His Pathogen research incorporates themes from Secretion and Cholera toxin.
Kim E. Barrett focuses on Immunology, Inflammatory bowel disease, Cell biology, Internal medicine and Pathophysiology. His studies in Immunology integrate themes in fields like Murine model, Transporter, Intestinal epithelium and Depression. Kim E. Barrett focuses mostly in the field of Inflammatory bowel disease, narrowing it down to matters related to Colitis and, in some cases, Pharmacology, Catalase, Agonist, Inflammation and Reactive oxygen species.
His work carried out in the field of Cell biology brings together such families of science as Serotonin reuptake inhibitor, Serotonin and Innate immune system, Pattern recognition receptor. Kim E. Barrett has included themes like Endocrinology and Intensive care medicine in his Internal medicine study. His Endocrinology research is multidisciplinary, incorporating perspectives in Fibrosis, Intracellular pH, Oxygen and MAPK/ERK pathway.
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Live probiotics protect intestinal epithelial cells from the effects of infection with enteroinvasive Escherichia coli (EIEC)
S Resta-Lenert;K E Barrett.
Chloride secretion by the intestinal epithelium: molecular basis and regulatory aspects.
Kim E. Barrett;Stephen J. Keely.
Annual Review of Physiology (2000)
Ganong's Review of Medical Physiology
Kim E. Barrett.
Probiotics and Commensals Reverse TNF-α– and IFN-γ–Induced Dysfunction in Human Intestinal Epithelial Cells
Silvia C. Resta-Lenert;Kim E. Barrett.
Role of intestinal epithelial cells in the host secretory response to infection by invasive bacteria. Bacterial entry induces epithelial prostaglandin h synthase-2 expression and prostaglandin E2 and F2alpha production.
Lars Eckmann;William F. Stenson;Tor C. Savidge;David C. Lowe.
Journal of Clinical Investigation (1997)
Long-term uncoupling of chloride secretion from intracellular calcium levels by Ins(3,4,5,6)P4
Mana Vajanaphanich;Carsten Schultz;Marco T. Rudolf;Matthew Wasserman.
Inhibition of Ca2+-dependent Cl- secretion in T84 cells : membrane target(s) of inhibition is agonist specific
Kim E. Barrett;Jane Smitham;Alexis Traynor-Kaplan;Jorge M. Uribe.
American Journal of Physiology-cell Physiology (1998)
Carbachol-stimulated Transactivation of Epidermal Growth Factor Receptor and Mitogen-activated Protein Kinase in T84 Cells Is Mediated by Intracellular Ca2+, PYK-2, and p60 src
Stephen J. Keely;Sean O. Calandrella;Kim E. Barrett.
Journal of Biological Chemistry (2000)
Carbachol Stimulates Transactivation of Epidermal Growth Factor Receptor and Mitogen-activated Protein Kinase in T84Cells IMPLICATIONS FOR CARBACHOL-STIMULATED CHLORIDE SECRETION
Stephen J. Keely;Jorge M. Uribe;Kim E. Barrett.
Journal of Biological Chemistry (1998)
Positive and negative regulation of chloride secretion in T84 cells
K. E. Barrett.
American Journal of Physiology-cell Physiology (1993)
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