D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 91 Citations 33,101 606 World Ranking 7278 National Ranking 3934

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Disease

David C. Whitcomb focuses on Pancreatitis, Internal medicine, Pancreatic disease, Hereditary pancreatitis and Acute pancreatitis. His Pancreatitis research includes elements of Pancreatic cancer, Intensive care medicine and Disease, Etiology, Pathology. David C. Whitcomb has researched Internal medicine in several fields, including Gastroenterology, Endocrinology and Surgery.

David C. Whitcomb has included themes like Genetics, PRSS2, Procollagen peptidase, Proinflammatory cytokine and Fibrosis in his Pancreatic disease study. His work in Hereditary pancreatitis covers topics such as Genetic disorder which are related to areas like Trypsinogen activation, Trypsin and Pediatrics. He interconnects Creatinine, Blood urea nitrogen, Predictive value of tests, Complication and Severity of illness in the investigation of issues within Acute pancreatitis.

His most cited work include:

  • Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene (1252 citations)
  • Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene (1252 citations)
  • Chronic pancreatitis: Diagnosis, classification, and new genetic developments (960 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Internal medicine, Pancreatitis, Acute pancreatitis, Gastroenterology and Endocrinology. His studies deal with areas such as Diabetes mellitus, Surgery and Oncology as well as Internal medicine. His work carried out in the field of Pancreatitis brings together such families of science as Hereditary pancreatitis, Pancreatic disease, Pancreas and Disease, Pathology.

His study in Hereditary pancreatitis is interdisciplinary in nature, drawing from both Genetics and Bioinformatics. His work focuses on many connections between Acute pancreatitis and other disciplines, such as Intensive care medicine, that overlap with his field of interest in MEDLINE. His Gastroenterology study frequently draws connections to adjacent fields such as Prospective cohort study.

He most often published in these fields:

  • Internal medicine (56.19%)
  • Pancreatitis (48.53%)
  • Acute pancreatitis (31.11%)

What were the highlights of his more recent work (between 2015-2021)?

  • Internal medicine (56.19%)
  • Pancreatitis (48.53%)
  • Acute pancreatitis (31.11%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Internal medicine, Pancreatitis, Acute pancreatitis, Gastroenterology and Intensive care medicine. His work in the fields of Internal medicine, such as Prospective cohort study, Pancreatic cancer, Cohort and Etiology, overlaps with other areas such as In patient. His research in Pancreatitis intersects with topics in MEDLINE, Diabetes mellitus, Pancreas, Disease and Abdominal pain.

His Pancreas research incorporates elements of Cystic fibrosis and Gene. His Acute pancreatitis research incorporates themes from Retrospective cohort study, Systemic inflammatory response syndrome, Necrosis, Severity of illness and Intensive care unit. His research integrates issues of Surgery and Cohort study, Pathology in his study of Gastroenterology.

Between 2015 and 2021, his most popular works were:

  • Chronic pancreatitis: An international draft consensus proposal for a new mechanistic definition (203 citations)
  • Guidelines for the understanding and management of pain in chronic pancreatitis (87 citations)
  • Peripancreatic fat necrosis worsens acute pancreatitis independent of pancreatic necrosis via unsaturated fatty acids increased in human pancreatic necrosis collections (71 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Disease

His primary areas of investigation include Pancreatitis, Internal medicine, Acute pancreatitis, Intensive care medicine and Diabetes mellitus. He studies Pancreatitis, namely Pancreatitis, chronic. His Internal medicine study incorporates themes from Gastroenterology and Surgery.

The Acute pancreatitis study combines topics in areas such as Necrosis, Body mass index, Pancreatic disease and Psychological intervention. His Pancreatic disease research integrates issues from Genetically engineered and Medical diagnosis. His work in Intensive care medicine tackles topics such as Pathology which are related to areas like Genetic enhancement, Gene mutation and Hereditary pancreatitis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene.

David C. Whitcomb;Michael C. Gorry;Robert A. Preston;William Furey.
Nature Genetics (1996)

1885 Citations

Chronic pancreatitis: Diagnosis, classification, and new genetic developments

Babak Etemad;David C. Whitcomb.
Gastroenterology (2001)

1690 Citations

IAP Guidelines for the Surgical Management of Acute Pancreatitis

Waldemar Uhl;Andrew Warshaw;Clement Imrie;Claudio Bassi.
Pancreatology (2002)

1346 Citations

Hereditary Pancreatitis and the Risk of Pancreatic Cancer

Albert B. Lowenfels;Patrick Maisonneuve;Eugene P. DiMagno;Yoram Elitsur.
Journal of the National Cancer Institute (1997)

1218 Citations

Clinical practice. Acute pancreatitis.

David C Whitcomb.
The New England Journal of Medicine (2006)

901 Citations

Comparison of BISAP, Ranson's, APACHE-II, and CTSI scores in predicting organ failure, complications, and mortality in acute pancreatitis

Georgios I Papachristou;Venkata Muddana;Dhiraj Yadav;Michael O'Connell.
The American Journal of Gastroenterology (2010)

718 Citations

Clinical and genetic characteristics of hereditary pancreatitis in Europe

Nathan Howes;Markus M. Lerch;William Greenhalf;Deborah D. Stocken.
Clinical Gastroenterology and Hepatology (2004)

623 Citations

Association of Acetaminophen Hepatotoxicity With Fasting and Ethanol Use

David C. Whitcomb;Geoffrey D. Block.
JAMA (1994)

621 Citations

SPINK1/PSTI polymorphisms act as disease modifiers in familial and idiopathic chronic pancreatitis.

Roland H. Pfützer;M.Michael Barmada;Andrew P.J. Brunskill;Robert Finch.
Gastroenterology (2000)

618 Citations

Mutations in the cationic trypsinogen gene are associated with recurrent acute and chronic pancreatitis

MC Gorry;D Gabbaizedeh;W Furey;LK Gates.
Gastroenterology (1997)

546 Citations

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