David C. Whitcomb focuses on Pancreatitis, Internal medicine, Pancreatic disease, Hereditary pancreatitis and Acute pancreatitis. His Pancreatitis research includes elements of Pancreatic cancer, Intensive care medicine and Disease, Etiology, Pathology. David C. Whitcomb has researched Internal medicine in several fields, including Gastroenterology, Endocrinology and Surgery.
David C. Whitcomb has included themes like Genetics, PRSS2, Procollagen peptidase, Proinflammatory cytokine and Fibrosis in his Pancreatic disease study. His work in Hereditary pancreatitis covers topics such as Genetic disorder which are related to areas like Trypsinogen activation, Trypsin and Pediatrics. He interconnects Creatinine, Blood urea nitrogen, Predictive value of tests, Complication and Severity of illness in the investigation of issues within Acute pancreatitis.
His primary scientific interests are in Internal medicine, Pancreatitis, Acute pancreatitis, Gastroenterology and Endocrinology. His studies deal with areas such as Diabetes mellitus, Surgery and Oncology as well as Internal medicine. His work carried out in the field of Pancreatitis brings together such families of science as Hereditary pancreatitis, Pancreatic disease, Pancreas and Disease, Pathology.
His study in Hereditary pancreatitis is interdisciplinary in nature, drawing from both Genetics and Bioinformatics. His work focuses on many connections between Acute pancreatitis and other disciplines, such as Intensive care medicine, that overlap with his field of interest in MEDLINE. His Gastroenterology study frequently draws connections to adjacent fields such as Prospective cohort study.
His primary areas of study are Internal medicine, Pancreatitis, Acute pancreatitis, Gastroenterology and Intensive care medicine. His work in the fields of Internal medicine, such as Prospective cohort study, Pancreatic cancer, Cohort and Etiology, overlaps with other areas such as In patient. His research in Pancreatitis intersects with topics in MEDLINE, Diabetes mellitus, Pancreas, Disease and Abdominal pain.
His Pancreas research incorporates elements of Cystic fibrosis and Gene. His Acute pancreatitis research incorporates themes from Retrospective cohort study, Systemic inflammatory response syndrome, Necrosis, Severity of illness and Intensive care unit. His research integrates issues of Surgery and Cohort study, Pathology in his study of Gastroenterology.
His primary areas of investigation include Pancreatitis, Internal medicine, Acute pancreatitis, Intensive care medicine and Diabetes mellitus. He studies Pancreatitis, namely Pancreatitis, chronic. His Internal medicine study incorporates themes from Gastroenterology and Surgery.
The Acute pancreatitis study combines topics in areas such as Necrosis, Body mass index, Pancreatic disease and Psychological intervention. His Pancreatic disease research integrates issues from Genetically engineered and Medical diagnosis. His work in Intensive care medicine tackles topics such as Pathology which are related to areas like Genetic enhancement, Gene mutation and Hereditary pancreatitis.
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Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene.
David C. Whitcomb;Michael C. Gorry;Robert A. Preston;William Furey.
Nature Genetics (1996)
Chronic pancreatitis: Diagnosis, classification, and new genetic developments
Babak Etemad;David C. Whitcomb.
IAP Guidelines for the Surgical Management of Acute Pancreatitis
Waldemar Uhl;Andrew Warshaw;Clement Imrie;Claudio Bassi.
Hereditary Pancreatitis and the Risk of Pancreatic Cancer
Albert B. Lowenfels;Patrick Maisonneuve;Eugene P. DiMagno;Yoram Elitsur.
Journal of the National Cancer Institute (1997)
Clinical practice. Acute pancreatitis.
David C Whitcomb.
The New England Journal of Medicine (2006)
Comparison of BISAP, Ranson's, APACHE-II, and CTSI scores in predicting organ failure, complications, and mortality in acute pancreatitis
Georgios I Papachristou;Venkata Muddana;Dhiraj Yadav;Michael O'Connell.
The American Journal of Gastroenterology (2010)
Clinical and genetic characteristics of hereditary pancreatitis in Europe
Nathan Howes;Markus M. Lerch;William Greenhalf;Deborah D. Stocken.
Clinical Gastroenterology and Hepatology (2004)
Association of Acetaminophen Hepatotoxicity With Fasting and Ethanol Use
David C. Whitcomb;Geoffrey D. Block.
SPINK1/PSTI polymorphisms act as disease modifiers in familial and idiopathic chronic pancreatitis.
Roland H. Pfützer;M.Michael Barmada;Andrew P.J. Brunskill;Robert Finch.
Mutations in the cationic trypsinogen gene are associated with recurrent acute and chronic pancreatitis
MC Gorry;D Gabbaizedeh;W Furey;LK Gates.
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