Hidekazu Tsukamoto mainly focuses on Internal medicine, Endocrinology, Hepatic stellate cell, Liver cytology and Pathology. Alcoholic liver disease, Liver injury, Tumor necrosis factor alpha, Hepatic stellate cell activation and Cytokine are subfields of Internal medicine in which his conducts study. His Endocrinology research integrates issues from Ethanol, Liquid diet and Mononuclear cell infiltration.
His Hepatic stellate cell research is multidisciplinary, incorporating elements of Cancer research, Peroxisome proliferator-activated receptor, Transforming growth factor, Cell biology and Fibrosis. His biological study spans a wide range of topics, including Molecular biology and Receptor. His Molecular biology study incorporates themes from Transferrin receptor, Transferrin and Hepcidin.
Hidekazu Tsukamoto mostly deals with Internal medicine, Endocrinology, Hepatic stellate cell, Cancer research and Alcoholic liver disease. As a part of the same scientific study, Hidekazu Tsukamoto usually deals with the Endocrinology, concentrating on Cytokine and frequently concerns with Kupffer cell and Proinflammatory cytokine. He has included themes like Peroxisome proliferator-activated receptor, Wnt signaling pathway, Molecular biology and Cell biology in his Hepatic stellate cell study.
His Molecular biology research also works with subjects such as
Hidekazu Tsukamoto spends much of his time researching Cancer research, Internal medicine, Hepatic stellate cell, Alcoholic liver disease and Endocrinology. His Cancer research research is multidisciplinary, incorporating perspectives in Carcinogenesis, Cancer and Downregulation and upregulation. His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology and Hepatocyte.
His studies deal with areas such as Wnt signaling pathway, Signal transduction, Cell biology, Molecular biology and Fibrosis as well as Hepatic stellate cell. His Alcoholic liver disease research incorporates elements of Microbiology, Steatohepatitis, Fatty liver and Hepatitis. His Endocrinology research is multidisciplinary, relying on both Alcohol and Immunohistochemistry.
His scientific interests lie mostly in Hepatic stellate cell, Cancer research, Internal medicine, Alcoholic liver disease and Immunology. His studies in Hepatic stellate cell integrate themes in fields like Chromatin immunoprecipitation, Chromatin, microRNA, Cell biology and Fibrosis. Hidekazu Tsukamoto combines subjects such as LIN28 and Endocrinology with his study of Internal medicine.
Hidekazu Tsukamoto studies Transforming growth factor which is a part of Endocrinology. His research in Alcoholic liver disease intersects with topics in Hepatology, Microbiology and Fatty liver. His Immunology research includes themes of Epigenetic Repression, Steatohepatitis, Histone methylation and Alcoholic hepatitis.
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Myofibroblasts revert to an inactive phenotype during regression of liver fibrosis
Tatiana Kisseleva;Min Cong;YongHan Paik;David Scholten.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Peroxisome Proliferator-activated Receptors and Hepatic Stellate Cell Activation
Takeo Miyahara;Laura Schrum;Richard Rippe;Shigang Xiong.
Journal of Biological Chemistry (2000)
Experimental liver cirrhosis induced by alcohol and iron.
H Tsukamoto;W Horne;S Kamimura;O Niemelä.
Journal of Clinical Investigation (1995)
Current concepts in the pathogenesis of alcoholic liver injury
Hidekazu Tsukamoto;Shelly C. Lu.
The FASEB Journal (2001)
Experimental models of hepatic fibrosis: a review.
Hidekazu Tsukamoto;Masaki Matsuoka;Samuel W. French.
Seminars in Liver Disease (1990)
Stimulation of hepatic lipocyte collagen production by Kupffer cell‐derived transforming growth factor β: Implication for a pathogenetic role in alcoholic liver fibrogenesis
Masaki Matsuoka;Masaki Matsuoka;Hidekazu Tsukamoto.
StellaTUM: current consensus and discussion on pancreatic stellate cell research
Mert Erkan;Guido Adler;Minoti V. Apte;Max G. Bachem.
MeCP2 controls an epigenetic pathway that promotes myofibroblast transdifferentiation and fibrosis.
Jelena Mann;David C.K. Chu;Aidan Maxwell;Fiona Oakley.
Interaction of the hereditary hemochromatosis protein HFE with transferrin receptor 2 is required for transferrin-induced hepcidin expression.
Junwei Gao;Juxing Chen;Maxwell Kramer;Hidekazu Tsukamoto;Hidekazu Tsukamoto.
Cell Metabolism (2009)
Ethanol-induced liver fibrosis in rats fed high fat diet.
Hidekazu Tsukamoto;Hidekazu Tsukamoto;Hidekazu Tsukamoto;Sally J. Towner;Sally J. Towner;Sally J. Towner;Lefran M. Clofalo;Lefran M. Clofalo;Lefran M. Clofalo;Samuel W. French;Samuel W. French;Samuel W. French.
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