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Medicine

D-Index
105
Citations
34877
World Ranking
6841
National Ranking
3610

Overview

Ronald G. Thurman was affiliated with the University of North Carolina at Chapel Hill in the United States. Their research contributions spanned fields primarily related to Medicine, with significant focus in Epidemiology, Pathology and Forensic Medicine, Hepatology, Surgery, and Molecular Biology.

Their work addressed several key topics, including:

  • Liver Disease Diagnosis and Treatment
  • Alcohol Consumption and Health Effects
  • Liver physiology and pathology
  • Renal Transplantation Outcomes and Treatments
  • Polyomavirus and related diseases
  • Cytomegalovirus and herpesvirus research
  • Drug-Induced Hepatotoxicity and Protection

Ronald G. Thurman published extensively in UNC Libraries, which served as the main venue for their scientific articles. Some of the recent papers associated with their work included:

  • NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease, 2020, UNC Libraries
  • Viral delivery of superoxide dismutase gene reduces cyclosporine A-induced nephrotoxicity, 2021, UNC Libraries
  • Overexpression of Manganese Superoxide Dismutase Prevents Alcohol-induced Liver Injury in the Rat, 2021, UNC Libraries
  • Glycine reduces platelet aggregation, 2020, UNC Libraries
  • Green Tea Polyphenols Stimulate Mitochondrial Biogenesis and Improve Renal Function after Chronic Cyclosporin A Treatment in Rats, 2020, UNC Libraries

Throughout their career, Thurman frequently collaborated with a number of co-authors, indicating a network of scientific partnerships. Notable frequent co-authors included:

  • Michael D. Wheeler
  • Henry D. Connor
  • Ronald P. Mason
  • Zhi Zhong
  • Blair U. Bradford

The areas of study they contributed to reflected a multidisciplinary approach within medical sciences, focusing on mechanisms and treatment strategies related to liver diseases, the effects of alcohol consumption, and renal transplantation outcomes.

The profile of Ronald G. Thurman depicts a researcher dedicated to understanding complex medical conditions through experimental and clinical investigation, primarily through a focus on liver physiology, pathology, and related systemic effects.

Best Publications

  • Essential role of tumor necrosis factor α in alcohol-induced liver injury in mice

    Ming Yin;Michael D. Wheeler;Hiroshi Kono;Blair U. Bradford

  • Antibodies to tumor necrosis factor alfa attenuate hepatic necrosis and inflammation caused by chronic exposure to ethanol in the rat.

    Yuji Iimuro;Randle M. Gallucci;Michael I. Luster;Hiroshi Kono

  • NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease.

    Hiroshi Kono;Ivan I Rusyn;Ming Yin;Erwin Gäbele

  • Kupffer cell activation and endothelial cell damage after storage of rat livers: effects of reperfusion.

    Jane C. Caldwell‐Kenkel;Robert T. Currin;Yukio Tanaka;Ronald G. Thurman

  • II. Alcoholic liver injury involves activation of Kupffer cells by endotoxin.

    R. G. Thurman

  • Reperfusion injury to endothelial cells following cold ischemic storage of rat livers.

    Jane C. Caldwell-Kenkel;Robert T. Currin;Yukio Tanaka;Ronald G. Thurman

  • Interaction of Mixed-Function Oxidation with Biosynthetic Processes

    Roland Scholz;Werner Hansen;Ronald G. Thurman

  • REPERFUSION INJURY AFTER LIVER PRESERVATION FOR TRANSPLANTATION

    John J. Lemasters;Ronald G. Thurman

  • Evidence that hypoxia markers detect oxygen gradients in liver: pimonidazole and retrograde perfusion of rat liver.

    G. E. Arteel;R. G. Thurman;J. M. Yates;J. A. Raleigh

  • THE ROLE OF KUPFFER CELL OXIDANT PRODUCTION IN EARLY ETHANOL-INDUCED LIVER DISEASE

    Michael D. Wheeler;Hiroshi Kono;Ming Yin;Mikio Nakagami

  • Overexpression of Manganese Superoxide Dismutase Prevents Alcohol-induced Liver Injury in the Rat

    Michael D. Wheeler;Mikio Nakagami;Blair U. Bradford;Takehiko Uesugi

  • Alcohol Causes Both Tolerance and Sensitization of Rat Kupffer Cells via Mechanisms Dependent on Endotoxin

    Nobuyuki Enomoto;Kenichi Ikejima;Blair Bradford;Chantal Rivera

  • Cyclosporin A increases hypoxia and free radical production in rat kidneys: prevention by dietary glycine

    Zhi Zhong;Gavin E. Arteel;Henry D. Connor;Ming Yin

  • Selective loss of nonparenchymal cell viability after cold ischemic storage of rat livers.

    Caldwell-Kenkel Jc;Thurman Rg;Lemasters Jj

  • Diphenyleneiodonium sulfate, an NADPH oxidase inhibitor, prevents early alcohol-induced liver injury in the rat.

    Hiroshi Kono;Ivan Rusyn;Takehiko Uesugi;Shunhei Yamashina

  • Reperfusion after liver transplantation in rats differentially activates the mitogen-activated protein kinases.

    C A Bradham;R F Stachlewitz;W Gao;T Qian

  • Reductive metabolism of the hypoxia marker pimonidazole is regulated by oxygen tension independent of the pyridine nucleotide redox state.

    Gavin E. Arteel;Ronald G. Thurman;James A. Raleigh

  • Centrilobular injury following hypoxia in isolated, perfused rat liver

    John J. Lemasters;Sungchul Ji;Ronald G. Thurman

  • CYP2E1 is not involved in early alcohol-induced liver injury

    Hiroshi Kono;Blair U. Bradford;Ming Yin;Kathleen K. Sulik

  • Periportal and pericentral pyridine nucleotide fluorescence from the surface of the perfused liver: evaluation of the hypothesis that chronic treatment with ethanol produces pericentral hypoxia

    Sungchul Ji;John J. Lemasters;Vickie Christenson;Ronald G. Thurman

Frequent Co-Authors

John J. Lemasters
John J. Lemasters Medical University of South Carolina
Ronald P. Mason
Ronald P. Mason National Institutes of Health
Ivan Rusyn
Ivan Rusyn Texas A&M University
David A. Brenner
David A. Brenner University of California, San Diego
Nobuyuki Enomoto
Nobuyuki Enomoto University of Yamanashi
James A. Swenberg
James A. Swenberg University of North Carolina at Chapel Hill
Richard J. Samulski
Richard J. Samulski University of North Carolina at Chapel Hill
Jeffrey M. Peters
Jeffrey M. Peters Pennsylvania State University
Frank J. Gonzalez
Frank J. Gonzalez National Institutes of Health
Lee M. Graves
Lee M. Graves University of North Carolina at Chapel Hill

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