Jeffrey M. Peters mainly focuses on Internal medicine, Endocrinology, Peroxisome proliferator-activated receptor, Peroxisome and Receptor. His Internal medicine study combines topics from a wide range of disciplines, such as Nuclear receptor, Phthalate and Cell growth. His Endocrinology study incorporates themes from Liver X receptor, Peroxisome proliferator-activated receptor delta, Peroxisome proliferator-activated receptor gamma and Carbohydrate-responsive element-binding protein.
His biological study spans a wide range of topics, including Carcinogen, Fatty acid-binding protein, Glucose homeostasis, Fibrate and Cell biology. Jeffrey M. Peters has researched Peroxisome in several fields, including Carcinogenesis, Mechanism of action, Peroxisome proliferator-activated receptor alpha and Fatty acid. His Receptor research includes elements of Cancer research, Transcription factor, Immunology, Ratón and Toxicity.
His primary areas of investigation include Peroxisome proliferator-activated receptor, Internal medicine, Endocrinology, Receptor and Cell biology. The study incorporates disciplines such as Cell growth, Carcinogenesis, Peroxisome proliferator-activated receptor delta, Nuclear receptor and Peroxisome in addition to Peroxisome proliferator-activated receptor. His Peroxisome research includes themes of Lipid metabolism and Fatty acid.
His study in Internal medicine is interdisciplinary in nature, drawing from both Fetus, Gene expression and Hepatocyte. Jeffrey M. Peters has included themes like Inflammation, Toxicity, Peroxisome Proliferation and Peroxisome proliferator-activated receptor alpha in his Endocrinology study. His Receptor research is multidisciplinary, relying on both Transcription factor, Pharmacology and Downregulation and upregulation.
Jeffrey M. Peters mostly deals with Peroxisome proliferator-activated receptor, Internal medicine, Cancer research, Endocrinology and Receptor. His Peroxisome proliferator-activated receptor research is multidisciplinary, incorporating perspectives in Cellular differentiation, Peroxisome proliferator-activated receptor delta, Nuclear receptor, Cell biology and Peroxisome. In the field of Internal medicine, his study on Esophageal adenocarcinoma and Amphiregulin overlaps with subjects such as Esophagectomy and Vascular endothelial growth factor A.
Jeffrey M. Peters interconnects Cell culture, Cell, Epiregulin, Carcinogenesis and Cancer cell in the investigation of issues within Cancer research. In the subject of general Endocrinology, his work in Choline is often linked to Wet age-related macular degeneration, thereby combining diverse domains of study. His Receptor research incorporates elements of Toxicity, Mode of action, Cell growth and Aryl hydrocarbon receptor.
Jeffrey M. Peters spends much of his time researching Cancer research, Peroxisome proliferator-activated receptor delta, Nuclear receptor, Signal transduction and Carcinogenesis. His research in Cancer research intersects with topics in Internal medicine and Endocrinology. His work deals with themes such as Liver cancer, Epidermal growth factor, Epiregulin and Mode of action, which intersect with Endocrinology.
The concepts of his Peroxisome proliferator-activated receptor delta study are interwoven with issues in Molecular biology, Cell and Peroxisome proliferator-activated receptor. Jeffrey M. Peters combines subjects such as Inflammation and Cell growth with his study of Nuclear receptor. As part of one scientific family, Jeffrey M. Peters deals mainly with the area of Carcinogenesis, narrowing it down to issues related to the Pharmacology, and often Receptor, Gemfibrozil, Toxicodynamics, Toxicity and Peroxisome proliferator-activated receptor alpha.
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Peroxisome proliferator–activated receptor α mediates the adaptive response to fasting
Sander Kersten;Josiane Seydoux;Jeffrey M. Peters;Frank J. Gonzalez.
Journal of Clinical Investigation (1999)
The PPARalpha-leukotriene B4 pathway to inflammation control.
Pallavi R. Devchand;Hansjörg Keller;Jeffrey Maurice Peters;Manuel Vazquez.
Nature (1996)
Peroxisome Proliferator-activated Receptor α Negatively Regulates the Vascular Inflammatory Gene Response by Negative Cross-talk with Transcription Factors NF-κB and AP-1
Philippe Delerive;Karolien De Bosscher;Sandrine Besnard;Wim Vanden Berghe.
Journal of Biological Chemistry (1999)
Altered Constitutive Expression of Fatty Acid-metabolizing Enzymes in Mice Lacking the Peroxisome Proliferator-activated Receptor α (PPARα)
Toshifumi Aoyama;Jeffrey M. Peters;Nobuko Iritani;Tamie Nakajima.
Journal of Biological Chemistry (1998)
Growth, Adipose, Brain, and Skin Alterations Resulting from Targeted Disruption of the Mouse Peroxisome Proliferator-Activated Receptor β(δ)
Jeffrey M. Peters;Susanna S. T. Lee;Wen Li;Jerrold M. Ward.
Molecular and Cellular Biology (2000)
Expression of Putative Fatty Acid Transporter Genes Are Regulated by Peroxisome Proliferator-activated Receptor α and γ Activators in a Tissue- and Inducer-specific Manner
Kiyoto Motojima;Patricia Passilly;Jeffrey M. Peters;Frank J. Gonzalez.
Journal of Biological Chemistry (1998)
PPARα Agonist-Induced Rodent Tumors: Modes of Action and Human Relevance
James E. Klaunig;Michael A. Babich;Karl P. Baetcke;Jon C. Cook.
Critical Reviews in Toxicology (2003)
PPARδ regulates glucose metabolism and insulin sensitivity
Chih Hao Lee;Peter Olson;Peter Olson;Andrea Hevener;Isaac Mehl;Isaac Mehl.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Role of PPAR alpha in the mechanism of action of the nongenotoxic carcinogen and peroxisome proliferator Wy-14,643.
Jeffrey M. Peters;Russell C. Cattley;Frank J. Gonzalez.
Carcinogenesis (1997)
Alterations in Lipoprotein Metabolism in Peroxisome Proliferator-activated Receptor α-deficient Mice
Jeffrey M. Peters;Nathalie Hennuyer;Bart Staels;Jean Charles Fruchart.
Journal of Biological Chemistry (1997)
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