Shantu Amin mainly focuses on Biochemistry, Carcinogen, Pharmacology, Carcinogenesis and Nitrosamine. His Carcinogen study combines topics from a wide range of disciplines, such as Endocrinology, Chrysene, Pyrene and Internal medicine. His Pharmacology research incorporates elements of Chemotherapy and Tumor initiation.
His Carcinogenesis study deals with Cancer research intersecting with Cancer cell, Metastasis and Cell culture. His studies in Nitrosamine integrate themes in fields like Corn oil, Pathology, Lung and Ratón. His research in Benzopyrene focuses on subjects like DNA repair, which are connected to Stereochemistry, Oligonucleotide and Base pair.
Shantu Amin mainly investigates Carcinogen, Biochemistry, Stereochemistry, Cancer research and DNA. His Carcinogen study combines topics in areas such as Carcinogenesis, Molecular biology, Endocrinology and Internal medicine. As part of one scientific family, Shantu Amin deals mainly with the area of Biochemistry, narrowing it down to issues related to the Pharmacology, and often Microsome.
His Stereochemistry study incorporates themes from Epoxide, Chrysene, Pyrene, Adduct and Diol. He interconnects Apoptosis, Cancer, Immunology and Cell growth in the investigation of issues within Cancer research. His work carried out in the field of Nitrosamine brings together such families of science as Toxicity and Ratón.
The scientist’s investigation covers issues in Cancer research, Cancer, Apoptosis, Biochemistry and Cancer cell. His study in Cancer research is interdisciplinary in nature, drawing from both Carcinogenesis, Cell culture, STAT3 and Stem cell. His Cancer research integrates issues from Immunology, Kinase, Pharmacology and Survival rate.
His work is connected to Carcinogen, DNA adduct, DNA, Deoxyadenosine and Benzopyrene, as a part of Biochemistry. Shantu Amin combines subjects such as Molecular biology, Toxicology, Ovary and Chrysene with his study of Carcinogen. While the research belongs to areas of DNA, Shantu Amin spends his time largely on the problem of Adduct, intersecting his research to questions surrounding Guanine and Pyrene.
His scientific interests lie mostly in Biochemistry, Cancer research, Aryl hydrocarbon receptor, Signal transduction and Cell biology. Biochemistry and In vivo are commonly linked in his work. The various areas that Shantu Amin examines in his Cancer research study include Cell culture, Endocrinology, Cancer and Apoptosis.
His biological study spans a wide range of topics, including Cancer cell, Suppressor and CYP1B1. His DNA repair research includes themes of Mouth neoplasm and Carcinogen. His research links Epoxide with Carcinogen.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Activation of chemically diverse procarcinogens by human cytochrome P-450 1B1
Tsutomu Shimada;Carrie L. Hayes;Hiroshi Yamazaki;Shantu Amin.
Cancer Research (1996)
Intestinal farnesoid X receptor signaling promotes nonalcoholic fatty liver disease
Changtao Jiang;Changtao Jiang;Cen Xie;Fei Li;Limin Zhang.
Journal of Clinical Investigation (2015)
Inhibition of tobacco-specific nitrosamine-induced lung tumorigenesis in A/J mice by green tea and its major polyphenol as antioxidants.
Yong Xu;Chi-Tang Ho;Shantu G. Amin;Chi Han.
Cancer Research (1992)
Induction of Lung and Exocrine Pancreas Tumors in F344 Rats by Tobacco-specific and Areca-derived N-Nitrosamines
Abraham Rivenson;Dietrich Hoffmann;Bogdan Prokopczyk;Shantu Amin.
Cancer Research (1988)
NMR SOLUTION STRUCTURES OF STEREOISOMERIC COVALENT POLYCYCLIC AROMATIC CARCINOGEN-DNA ADDUCTS : PRINCIPLES, PATTERNS, AND DIVERSITY
Nicholas E. Geacintov;Monique Cosman;Brian E. Hingerty;Shantu Amin.
Chemical Research in Toxicology (1997)
Autophagosomal Membrane Serves as Platform for Intracellular Death-inducing Signaling Complex (iDISC)-mediated Caspase-8 Activation and Apoptosis
Megan M. Young;Yoshinori Takahashi;Osman Khan;Sungman Park.
Journal of Biological Chemistry (2012)
Intestine-selective farnesoid X receptor inhibition improves obesity-related metabolic dysfunction.
Changtao Jiang;Cen Xie;Ying Lv;Jing Li.
Nature Communications (2015)
Targeting of Lung Cancer Mutational Hotspots by Polycyclic Aromatic Hydrocarbons
Leslie E. Smith;Mikhail F. Denissenko;Mikhail F. Denissenko;William P. Bennett;Haiying Li.
Journal of the National Cancer Institute (2000)
The major lipid peroxidation product, trans- 4-hydroxy-2-nonenal, preferentially forms DNA adducts at codon 249 of human p53 gene, a unique mutational hotspot in hepatocellular carcinoma
Wenwei Hu;Zhaohui Feng;Jamie Eveleigh;Ganesh Iyer.
Carcinogenesis (2002)
Inhibition of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone-induced DNA adduct formation and tumorigenicity in the lung of F344 rats by dietary phenethyl isothiocyanate.
Mark A. Morse;Chung-Xiou Wang;Gary D. Stoner;Swapna Mandal.
Cancer Research (1989)
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