D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 86 Citations 36,993 378 World Ranking 1865 National Ranking 1051

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Cancer

The scientist’s investigation covers issues in Apoptosis, Cancer cell, Biochemistry, Diallyl trisulfide and Cancer research. His Apoptosis research integrates issues from Reactive oxygen species, LNCaP and In vivo. His research integrates issues of Cell cycle, Cell cycle checkpoint, Immunology and Withaferin A in his study of Cancer cell.

His studies deal with areas such as Prostate cancer, Cell growth and Pathology as well as Cancer research. His research in Programmed cell death intersects with topics in Autophagy, MAP1LC3B, Chaperone-mediated autophagy, Autophagosome and Sequestosome 1. His work on BECN1 and Autolysosome as part of his general Autophagy study is frequently connected to Vacuole, thereby bridging the divide between different branches of science.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Sulforaphane-induced Cell Death in Human Prostate Cancer Cells Is Initiated by Reactive Oxygen Species (307 citations)
  • Sulforaphane-induced G2/M Phase Cell Cycle Arrest Involves Checkpoint Kinase 2-mediated Phosphorylation of Cell Division Cycle 25C (302 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cancer research, Biochemistry, Cancer cell, Apoptosis and Glutathione. Shivendra V. Singh combines subjects such as Cancer, Prostate cancer, LNCaP, Cell growth and Benzyl isothiocyanate with his study of Cancer research. Biochemistry is often connected to In vivo in his work.

The Cancer cell study combines topics in areas such as Autophagy, Cell culture, Endocrinology and Withaferin A. His research in Apoptosis intersects with topics in Reactive oxygen species and Cell biology. His Glutathione research incorporates themes from Molecular biology, Isozyme and Carcinogen, Benzopyrene.

He most often published in these fields:

  • Cancer research (37.92%)
  • Biochemistry (33.56%)
  • Cancer cell (33.56%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cancer research (37.92%)
  • Cancer (24.16%)
  • Cancer cell (33.56%)

In recent papers he was focusing on the following fields of study:

Shivendra V. Singh mainly investigates Cancer research, Cancer, Cancer cell, Apoptosis and Withaferin A. The concepts of his Cancer research study are interwoven with issues in Cell growth, Downregulation and upregulation, Benzyl isothiocyanate, Breast cancer and Prostate cancer. In his research, Diallyl disulfide and Estrogen receptor is intimately related to Diallyl trisulfide, which falls under the overarching field of Cancer.

The Cancer cell study combines topics in areas such as Autophagy, Molecular biology and Endocrinology. His studies deal with areas such as Kinase and In vivo as well as Withaferin A. His Programmed cell death research includes elements of Chaperone-mediated autophagy, Computational biology and Autolysosome.

Between 2013 and 2021, his most popular works were:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Growth arrest by the antitumor steroidal lactone withaferin a in human breast cancer cells is associated with down-regulation and covalent binding at cysteine 303 of β-tubulin (79 citations)
  • Molecular Targets and Mechanisms of Cancer Prevention and Treatment by Withaferin A, A Naturally Occurring Steroidal Lactone (73 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Cancer

His primary areas of study are Withaferin A, Cancer research, Cancer cell, Apoptosis and Prostate cancer. Shivendra V. Singh interconnects Mammary tumor, Kinase, MAPK/ERK pathway and Pharmacology in the investigation of issues within Withaferin A. His studies in Cancer research integrate themes in fields like Growth inhibition, Cell growth, Downregulation and upregulation, Transcription factor and In vivo.

His Cancer cell research is multidisciplinary, incorporating elements of Molecular biology and Cell Migration Inhibition. His biological study spans a wide range of topics, including Cell culture and Cell biology. His Programmed cell death research integrates issues from Autophagosome and Multicellular organism.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Sulforaphane-induced Cell Death in Human Prostate Cancer Cells Is Initiated by Reactive Oxygen Species

Shivendra V. Singh;Sanjay K. Srivastava;Sunga Choi;Karen L. Lew.
Journal of Biological Chemistry (2005)

398 Citations

Sulforaphane-induced G2/M Phase Cell Cycle Arrest Involves Checkpoint Kinase 2-mediated Phosphorylation of Cell Division Cycle 25C

Shivendra V. Singh;Anna Herman-Antosiewicz;Ajita V. Singh;Karen L. Lew.
Journal of Biological Chemistry (2004)

397 Citations

Sulforaphane Causes Autophagy to Inhibit Release of Cytochrome c and Apoptosis in Human Prostate Cancer Cells

Anna Herman-Antosiewicz;Daniel E. Johnson;Shivendra V. Singh.
Cancer Research (2006)

364 Citations

Allyl isothiocyanate, a constituent of cruciferous vegetables, inhibits proliferation of human prostate cancer cells by causing G2/M arrest and inducing apoptosis.

Dong Xiao;Sanjay K. Srivastava;Karen L. Lew;Yan Zeng.
Carcinogenesis (2003)

352 Citations

Diallyl trisulfide-induced apoptosis in human prostate cancer cells involves c-Jun N-terminal kinase and extracellular-signal regulated kinase-mediated phosphorylation of Bcl-2.

Dong Xiao;Sunga Choi;Daniel E Johnson;Victor G Vogel.
Oncogene (2004)

307 Citations

Multitargeted prevention and therapy of cancer by diallyl trisulfide and related Allium vegetable-derived organosulfur compounds.

Anna A. Powolny;Shivendra V. Singh.
Cancer Letters (2008)

292 Citations

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

279 Citations

Cell cycle arrest, apoptosis induction and inhibition of nuclear factor kappa B activation in anti-proliferative activity of benzyl isothiocyanate against human pancreatic cancer cells.

Sanjay K. Srivastava;Shivendra V. Singh.
Carcinogenesis (2004)

242 Citations

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