The scientist’s investigation covers issues in Apoptosis, Cancer cell, Biochemistry, Diallyl trisulfide and Cancer research. His Apoptosis research integrates issues from Reactive oxygen species, LNCaP and In vivo. His research integrates issues of Cell cycle, Cell cycle checkpoint, Immunology and Withaferin A in his study of Cancer cell.
His studies deal with areas such as Prostate cancer, Cell growth and Pathology as well as Cancer research. His research in Programmed cell death intersects with topics in Autophagy, MAP1LC3B, Chaperone-mediated autophagy, Autophagosome and Sequestosome 1. His work on BECN1 and Autolysosome as part of his general Autophagy study is frequently connected to Vacuole, thereby bridging the divide between different branches of science.
His primary scientific interests are in Cancer research, Biochemistry, Cancer cell, Apoptosis and Glutathione. Shivendra V. Singh combines subjects such as Cancer, Prostate cancer, LNCaP, Cell growth and Benzyl isothiocyanate with his study of Cancer research. Biochemistry is often connected to In vivo in his work.
The Cancer cell study combines topics in areas such as Autophagy, Cell culture, Endocrinology and Withaferin A. His research in Apoptosis intersects with topics in Reactive oxygen species and Cell biology. His Glutathione research incorporates themes from Molecular biology, Isozyme and Carcinogen, Benzopyrene.
Shivendra V. Singh mainly investigates Cancer research, Cancer, Cancer cell, Apoptosis and Withaferin A. The concepts of his Cancer research study are interwoven with issues in Cell growth, Downregulation and upregulation, Benzyl isothiocyanate, Breast cancer and Prostate cancer. In his research, Diallyl disulfide and Estrogen receptor is intimately related to Diallyl trisulfide, which falls under the overarching field of Cancer.
The Cancer cell study combines topics in areas such as Autophagy, Molecular biology and Endocrinology. His studies deal with areas such as Kinase and In vivo as well as Withaferin A. His Programmed cell death research includes elements of Chaperone-mediated autophagy, Computational biology and Autolysosome.
His primary areas of study are Withaferin A, Cancer research, Cancer cell, Apoptosis and Prostate cancer. Shivendra V. Singh interconnects Mammary tumor, Kinase, MAPK/ERK pathway and Pharmacology in the investigation of issues within Withaferin A. His studies in Cancer research integrate themes in fields like Growth inhibition, Cell growth, Downregulation and upregulation, Transcription factor and In vivo.
His Cancer cell research is multidisciplinary, incorporating elements of Molecular biology and Cell Migration Inhibition. His biological study spans a wide range of topics, including Cell culture and Cell biology. His Programmed cell death research integrates issues from Autophagosome and Multicellular organism.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Sulforaphane-induced Cell Death in Human Prostate Cancer Cells Is Initiated by Reactive Oxygen Species
Shivendra V. Singh;Sanjay K. Srivastava;Sunga Choi;Karen L. Lew.
Journal of Biological Chemistry (2005)
Sulforaphane-induced G2/M Phase Cell Cycle Arrest Involves Checkpoint Kinase 2-mediated Phosphorylation of Cell Division Cycle 25C
Shivendra V. Singh;Anna Herman-Antosiewicz;Ajita V. Singh;Karen L. Lew.
Journal of Biological Chemistry (2004)
Sulforaphane Causes Autophagy to Inhibit Release of Cytochrome c and Apoptosis in Human Prostate Cancer Cells
Anna Herman-Antosiewicz;Daniel E. Johnson;Shivendra V. Singh.
Cancer Research (2006)
Allyl isothiocyanate, a constituent of cruciferous vegetables, inhibits proliferation of human prostate cancer cells by causing G2/M arrest and inducing apoptosis.
Dong Xiao;Sanjay K. Srivastava;Karen L. Lew;Yan Zeng.
Carcinogenesis (2003)
Diallyl trisulfide-induced apoptosis in human prostate cancer cells involves c-Jun N-terminal kinase and extracellular-signal regulated kinase-mediated phosphorylation of Bcl-2.
Dong Xiao;Sunga Choi;Daniel E Johnson;Victor G Vogel.
Oncogene (2004)
Multitargeted prevention and therapy of cancer by diallyl trisulfide and related Allium vegetable-derived organosulfur compounds.
Anna A. Powolny;Shivendra V. Singh.
Cancer Letters (2008)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Cell cycle arrest, apoptosis induction and inhibition of nuclear factor kappa B activation in anti-proliferative activity of benzyl isothiocyanate against human pancreatic cancer cells.
Sanjay K. Srivastava;Shivendra V. Singh.
Carcinogenesis (2004)
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