What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
His main research concerns Hepatic stellate cell, Fibrosis, Internal medicine, Molecular biology and Cell biology.
His Hepatic stellate cell research includes themes of Hepatic fibrosis, Liver injury, Messenger RNA, Liver cytology and Proinflammatory cytokine.
David A. Brenner combines subjects such as Cancer research, Inflammation, Immunology, Extracellular matrix and Transforming growth factor beta with his study of Fibrosis.
His Internal medicine study integrates concerns from other disciplines, such as Gastroenterology and Endocrinology.
As a part of the same scientific study, David A. Brenner usually deals with the Molecular biology, concentrating on Gene expression and frequently concerns with Hepatic stellate cell activation and Procollagen peptidase.
His Cell biology research is multidisciplinary, incorporating perspectives in Apoptosis, Chemokine secretion and Mitochondrial permeability transition pore.
His most cited work include:
- Erratum: Liver fibrosis (Journal of Clinical Investigation (2005) 115 (209-218) DOI:10.1172/JCI200524282) (1494 citations)
- TLR4 enhances TGF-beta signaling and hepatic fibrosis. (1317 citations)
- The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy. (1205 citations)
What are the main themes of his work throughout his whole career to date?
David A. Brenner mainly investigates Hepatic stellate cell, Internal medicine, Fibrosis, Endocrinology and Molecular biology.
His Hepatic stellate cell research includes elements of Cancer research, Liver injury, Hepatic fibrosis and Cell biology.
His Internal medicine research focuses on Gastroenterology and how it relates to Liver biopsy.
He works mostly in the field of Fibrosis, limiting it down to topics relating to Steatohepatitis and, in certain cases, Alcoholic liver disease, as a part of the same area of interest.
The study incorporates disciplines such as Proinflammatory cytokine, Kupffer cell and Hepatocyte in addition to Endocrinology.
The concepts of his Molecular biology study are interwoven with issues in Reporter gene, Gene expression, Transcription factor, Biochemistry and Messenger RNA.
He most often published in these fields:
- Hepatic stellate cell (31.19%)
- Internal medicine (29.54%)
- Fibrosis (19.47%)
What were the highlights of his more recent work (between 2015-2021)?
- Fibrosis (19.47%)
- Internal medicine (29.54%)
- Cancer research (17.00%)
In recent papers he was focusing on the following fields of study:
David A. Brenner focuses on Fibrosis, Internal medicine, Cancer research, Hepatic stellate cell and Pathology.
His research in Fibrosis tackles topics such as Proinflammatory cytokine which are related to areas like Oxidative stress.
His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology and Endocrinology.
His Cancer research research incorporates elements of Inflammation, Alcoholic liver disease, Downregulation and upregulation and Pathogenesis.
His Hepatic stellate cell study combines topics in areas such as Myofibroblast, Liver injury, Molecular biology and Extracellular matrix, Cell biology.
The concepts of his Fatty liver study are interwoven with issues in Liver disease and Cirrhosis.
Between 2015 and 2021, his most popular works were:
- Gut Microbiome-Based Metagenomic Signature for Non-invasive Detection of Advanced Fibrosis in Human Nonalcoholic Fatty Liver Disease (343 citations)
- Liver inflammation and fibrosis (338 citations)
- Magnetic Resonance Elastography vs Transient Elastography in Detection of Fibrosis and Noninvasive Measurement of Steatosis in Patients With Biopsy-Proven Nonalcoholic Fatty Liver Disease. (273 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
His primary scientific interests are in Fibrosis, Hepatic stellate cell, Internal medicine, Cancer research and Cirrhosis.
His research investigates the connection between Fibrosis and topics such as Fatty liver that intersect with issues in Liver disease.
His research integrates issues of Peroxisome proliferator-activated receptor, ETS1, Liver injury, Chromatin immunoprecipitation and Cell biology in his study of Hepatic stellate cell.
David A. Brenner combines subjects such as Gastroenterology and Endocrinology with his study of Internal medicine.
The study incorporates disciplines such as Myofibroblast, Denervation, Inflammation, Heterozygote advantage and SOD1 in addition to Cancer research.
David A. Brenner has researched Cirrhosis in several fields, including Microbiome and Immunology, Hepatitis.
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