His primary areas of investigation include Mitochondrion, Glutathione, Biochemistry, Cell biology and Programmed cell death. His study in Mitochondrion is interdisciplinary in nature, drawing from both Reactive oxygen species, Endocrinology, Oxidative phosphorylation and Internal medicine. His Glutathione research integrates issues from Organic anion transporter 1, Molecular biology, Alcoholic liver disease and Cytosol.
His Antioxidant, Transporter and Glutathione disulfide study in the realm of Biochemistry connects with subjects such as Synthetic membrane and Vesicle. His work carried out in the field of Cell biology brings together such families of science as Apoptosis, Caspase and Neurodegeneration. His Programmed cell death study incorporates themes from Carcinogenesis, Cancer cell, Digitonin, Cell fractionation and Cyclosporin a.
His primary scientific interests are in Mitochondrion, Internal medicine, Biochemistry, Endocrinology and Cell biology. His Mitochondrion research incorporates elements of Oxidative stress, Reactive oxygen species, Glutathione and Programmed cell death. His research in Programmed cell death intersects with topics in Cancer cell and Autophagy.
His Internal medicine study combines topics from a wide range of disciplines, such as Endoplasmic reticulum and Tauroursodeoxycholic acid. José C. Fernández-Checa interconnects Unfolded protein response and Steatohepatitis, Fatty liver in the investigation of issues within Endocrinology. He usually deals with Cell biology and limits it to topics linked to Ceramide and Sphingolipid.
José C. Fernández-Checa mostly deals with Cancer research, Programmed cell death, Mitochondrion, Cell biology and Endocrinology. His biological study spans a wide range of topics, including Autophagy and Cancer cell. His Mitochondrion research includes themes of Pathogenesis, Reactive oxygen species, Liver injury, Pharmacology and Cholesterol.
His Cell biology research is multidisciplinary, incorporating perspectives in Ceramide, Surgery and Hepatic stellate cell. His research integrates issues of Liver disease, Internal medicine and Steatohepatitis, Fatty liver in his study of Endocrinology. His Oxidative stress research incorporates themes from Glutathione and Ischemia.
José C. Fernández-Checa focuses on Cell biology, Programmed cell death, Mitochondrion, Apoptosis and Autophagy. José C. Fernández-Checa works mostly in the field of Cell biology, limiting it down to topics relating to Cholesterol and, in certain cases, Alzheimer's disease, Intracellular and Endoplasmic reticulum. His Mitochondrion research is included under the broader classification of Biochemistry.
José C. Fernández-Checa has researched Apoptosis in several fields, including Melatonin and Cancer cell. His studies in Autophagy integrate themes in fields like Computational biology and Acid sphingomyelinase. His Myriocin research incorporates themes from Internal medicine and Endocrinology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Parasites & Vectors (2016)
Direct Effect of Ceramide on the Mitochondrial Electron Transport Chain Leads to Generation of Reactive Oxygen Species ROLE OF MITOCHONDRIAL GLUTATHIONE
Carmen García-Ruiz;Anna Colell;Montserrat Marí;Albert Morales.
Journal of Biological Chemistry (1997)
Mitochondrial glutathione, a key survival antioxidant.
Montserrat Marí;Albert Morales;Anna Colell;Carmen García-Ruiz.
Antioxidants & Redox Signaling (2009)
Mitochondrial free cholesterol loading sensitizes to TNF- and Fas-mediated steatohepatitis
Montserrat Marí;Francisco Caballero;Anna Colell;Albert Morales.
Cell Metabolism (2006)
GSH transport in mitochondria: defense against TNF-induced oxidative stress and alcohol-induced defect
J. C. Fernandez-Checa;N. Kaplowitz;C. Garcia-Ruiz;A. Colell.
American Journal of Physiology-gastrointestinal and Liver Physiology (1997)
Selective glutathione depletion of mitochondria by ethanol sensitizes hepatocytes to tumor necrosis factor
Anna Colell;Carmen García-Ruiz;Merce Miranda;Esther Ardite.
Gastroenterology (1998)
Hepatic mitochondrial glutathione: transport and role in disease and toxicity
Jose C. Fernandez-Checa;Neil Kaplowitz.
Toxicology and Applied Pharmacology (2005)
Glutathione and mitochondria.
Vicent Ribas;Carmen García-Ruiz;Carmen García-Ruiz;José C. Fernández-Checa;José C. Fernández-Checa.
Frontiers in Pharmacology (2014)
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