What is he best known for?
The fields of study he is best known for:
Apoptosis, Programmed cell death, Cell biology, Caspase and Mitochondrion are his primary areas of study.
The study incorporates disciplines such as Necrosis, Cell and Cell growth in addition to Apoptosis.
His Programmed cell death research includes elements of Autophagy, Neuroscience and Intracellular, Calcium signaling.
Boris Zhivotovsky interconnects Cell cycle, DNA damage and Apoptosome in the investigation of issues within Cell biology.
His Caspase study integrates concerns from other disciplines, such as Proteases and DNA fragmentation.
His study in Mitochondrion is interdisciplinary in nature, drawing from both Cancer cell, Gene silencing and Mitochondrial permeability transition pore.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
What are the main themes of his work throughout his whole career to date?
Boris Zhivotovsky spends much of his time researching Apoptosis, Cell biology, Programmed cell death, Cancer research and Caspase.
His work deals with themes such as Cell, Signal transduction and Molecular biology, which intersect with Apoptosis.
His studies in Cell biology integrate themes in fields like Caspase 2, Biochemistry, DNA damage and Intrinsic apoptosis.
His research in Programmed cell death intersects with topics in Autophagy, Cancer cell, Cancer, Cell cycle and Necrosis.
His Cancer research study incorporates themes from Cell culture, Lung cancer, Gene silencing and Immunology.
The concepts of his Caspase study are interwoven with issues in Proteases and Intracellular.
He most often published in these fields:
- Apoptosis (74.94%)
- Cell biology (68.05%)
- Programmed cell death (60.92%)
What were the highlights of his more recent work (between 2017-2021)?
- Programmed cell death (60.92%)
- Cell biology (68.05%)
- Apoptosis (74.94%)
In recent papers he was focusing on the following fields of study:
Boris Zhivotovsky mainly focuses on Programmed cell death, Cell biology, Apoptosis, Cancer cell and Autophagy.
His study of Necroptosis is a part of Programmed cell death.
His biological study spans a wide range of topics, including Caspase 2 and Cell.
His Apoptosis research is multidisciplinary, incorporating elements of Suppressor, Signal transduction and Multicellular organism.
His Cancer cell research integrates issues from Cancer treatment, Biophysics, Downregulation and upregulation and Drug delivery.
Boris Zhivotovsky has researched Neuroscience in several fields, including Pyroptosis, Immunogenic cell death, Transcription and Intrinsic apoptosis.
Between 2017 and 2021, his most popular works were:
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
- Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
- Cell death-based treatment of lung adenocarcinoma. (91 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Programmed cell death, Apoptosis, Cell biology, Autophagy and Intracellular.
Boris Zhivotovsky combines subjects such as Cancer research, Targeted therapy, Metastasis, Disease and Immunotherapy with his study of Programmed cell death.
The various areas that Boris Zhivotovsky examines in his Apoptosis study include Cell, Neuroscience, Bioinformatics and Crosstalk.
His Cell biology research is multidisciplinary, incorporating perspectives in Caspase, Cleavage, Multicellular organism and Proteolysis.
Boris Zhivotovsky has included themes like Chaperone-mediated autophagy, Computational biology and Autolysosome in his Multicellular organism study.
His biological study deals with issues like Necroptosis, which deal with fields such as Intrinsic apoptosis and Pyroptosis.
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