2023 - Research.com Biology and Biochemistry in United Kingdom Leader Award
2010 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften For Physiology and Pharmacology/Toxicology
2000 - Fellow of the Royal Society, United Kingdom
1998 - Fellow of The Academy of Medical Sciences, United Kingdom
1988 - Member of Academia Europaea
His primary scientific interests are in Biophysics, Endocrinology, Internal medicine, Cell biology and Biochemistry. His Biophysics research incorporates themes from Patch clamp, Calcium and Intracellular. His Endocrinology research includes themes of Cholecystokinin and Ion channel.
The study of Internal medicine is intertwined with the study of Gating in a number of ways. His biological study spans a wide range of topics, including SERCA and Mitochondrial permeability transition pore. His work in Biochemistry addresses issues such as Neuron, which are connected to fields such as Neurite, Synaptic plasticity, Chaperone, Unfolded protein response and Lumen.
Ole H. Petersen mainly investigates Internal medicine, Endocrinology, Biophysics, Cell biology and Intracellular. His Internal medicine study typically links adjacent topics like Extracellular. Ole H. Petersen combines subjects such as Calcium and Membrane potential with his study of Endocrinology.
He usually deals with Biophysics and limits it to topics linked to Inositol trisphosphate and Inositol phosphate and Inositol trisphosphate receptor. He interconnects Pancreas and Cytosol in the investigation of issues within Cell biology. The Intracellular study combines topics in areas such as Acute pancreatitis and Inositol.
Ole H. Petersen mostly deals with Cell biology, Internal medicine, Endocrinology, Acute pancreatitis and Intracellular. His Cell biology research integrates issues from Receptor, Secretion and Hepatic stellate cell. Much of his study explores Internal medicine relationship to Guideline.
His work carried out in the field of Endocrinology brings together such families of science as Voltage-dependent calcium channel, Calcium, Channel blocker and Pharmacology. His studies in Intracellular integrate themes in fields like Necrosis, Liberation and Programmed cell death. His study on Biochemistry is mostly dedicated to connecting different topics, such as Biophysics.
Endocrinology, Internal medicine, Biochemistry, Inositol trisphosphate and Voltage-dependent calcium channel are his primary areas of study. His Endocrinology research includes elements of Calcium metabolism and Mitochondrion. His Internal medicine research is multidisciplinary, incorporating elements of Phosphodiesterase and Xanthine.
Biochemistry is closely attributed to Biophysics in his study. His Inositol trisphosphate study combines topics in areas such as Ryanodine receptor, Calcium, Intracellular, Trypsinogen activation and Pancreatitis. His Voltage-dependent calcium channel study combines topics from a wide range of disciplines, such as Cholecystokinin, Channel blocker, Trypsin and Cell biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Calcium-activated potassium channels and their role in secretion
Ole H. Petersen;Yoshio Maruyama.
Nature (1984)
Synergism of inositol trisphosphate and tetrakisphosphate in activating Ca2+-dependent K+ channels.
A. P. Morris;D. V. Gallacher;R. F. Irvine;O. H. Petersen.
Nature (1987)
Local and global cytosolic Ca2+ oscillations in exocrine cells evoked by agonists and inositol trisphosphate
Peter Thorn;Alison M. Lawrie;Peter M. Smith;David V. Gallacher.
Cell (1993)
Stimulus-secretion coupling: cytoplasmic calcium signals and the control of ion channels in exocrine acinar cells.
O H Petersen.
The Journal of Physiology (1992)
ATP-DEPENDENT ACCUMULATION AND INOSITOL TRISPHOSPHATE- OR CYCLIC ADP-RIBOSE-MEDIATED RELEASE OF CA2+ FROM THE NUCLEAR ENVELOPE
Oleg V Gerasimenko;Julia V Gerasimenko;Alexei V Tepikin;Ole H Petersen.
Cell (1995)
Active mitochondria surrounding the pancreatic acinar granule region prevent spreading of inositol trisphosphate-evoked local cytosolic Ca2+ signals
Hanna Tinel;Jose M. Cancela;Hideo Mogami;Julia V. Gerasimenko.
The EMBO Journal (1999)
Calcium-dependent enzyme activation and vacuole formation in the apical granular region of pancreatic acinar cells
Michael Raraty;Jeremy Ward;Gul Erdemli;Camille Vaillant.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Pulsatile intracellular calcium release does not depend on fluctuations in inositol trisphosphate concentration
Makoto Wakui;Barry V. L. Potter;Ole H. Petersen.
Nature (1989)
Cytoplasmic Ca2+ oscillations evoked by receptor stimulation, G-protein activation, internal application of inositol trisphosphate or Ca2+: simultaneous microfluorimetry and Ca2+ dependent Cl- current recording in single pancreatic acinar cells.
Y.V. Osipchuk;M. Wakui;D.I. Yule;D.V. Gallacher.
The EMBO Journal (1990)
The ARRIVE guidelines 2.0: Updated guidelines for reporting animal research.
Nathalie Percie du Sert;Viki Hurst;Amrita Ahluwalia;Sabina Alam.
Journal of Cerebral Blood Flow and Metabolism (2020)
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