His main research concerns Cell biology, Thapsigargin, STIM1, ORAI1 and Intracellular. His specific area of interest is Cell biology, where Ginés M. Salido studies Endoplasmic reticulum. His Thapsigargin study is associated with Biochemistry.
His ORAI1 study combines topics from a wide range of disciplines, such as TRPC1, TRPC Cation Channels, Transient receptor potential channel and TRPC. His Intracellular study integrates concerns from other disciplines, such as Extracellular and Cytosol. His Biophysics research includes elements of SERCA and Calcium.
Ginés M. Salido mainly focuses on Endocrinology, Internal medicine, Cell biology, Intracellular and Biochemistry. His Endocrinology research integrates issues from Cholecystokinin and Amylase. His study explores the link between Cell biology and topics such as Actin cytoskeleton that cross with problems in Cytochalasin D.
Ginés M. Salido studies Intracellular, focusing on Thapsigargin in particular. His work carried out in the field of Thapsigargin brings together such families of science as Molecular biology, SERCA, Ionomycin and Phospholipase C. His research combines Biophysics and Biochemistry.
Ginés M. Salido mainly investigates Cell biology, STIM1, ORAI1, Intracellular and Store-operated calcium entry. His research in Cell biology is mostly focused on Endoplasmic reticulum. As a member of one scientific family, Ginés M. Salido mostly works in the field of STIM1, focusing on Cytosol and, on occasion, Homeostasis.
In his study, which falls under the umbrella issue of ORAI1, Stimulation is strongly linked to TRPC1. His Intracellular research incorporates elements of TRPC3, Platelet, Biophysics and Extracellular. In the subject of general Biochemistry, his work in Thapsigargin, Antioxidant and p38 mitogen-activated protein kinases is often linked to Ebselen, thereby combining diverse domains of study.
His primary scientific interests are in Cell biology, STIM1, ORAI1, Store-operated calcium entry and Endoplasmic reticulum. His Cell biology research is multidisciplinary, incorporating perspectives in Biochemistry, Cell growth, Transient receptor potential channel and G alpha subunit. He has researched STIM1 in several fields, including Biophysics and Function.
The ORAI1 study combines topics in areas such as TRPC1, TRPC and Cancer research. Store-operated calcium entry is the subject of his research, which falls under Intracellular. His studies deal with areas such as Internal medicine and Endocrinology as well as NAD+ kinase.
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Interaction of STIM1 with endogenously expressed human canonical TRP1 upon depletion of intracellular Ca2+ stores.
José J. Lopez;Ginés M. Salido;José A. Pariente;Juan A. Rosado.
Journal of Biological Chemistry (2006)
Orai1 mediates the interaction between STIM1 and hTRPC1 and regulates the mode of activation of hTRPC1-forming Ca2+ channels.
Isaac Jardin;José J. Lopez;Gines M. Salido;Juan A. Rosado.
Journal of Biological Chemistry (2008)
TRPC channels and store-operated Ca2+ entry
Ginés M. Salido;Stewart O. Sage;Juan A. Rosado.
Biochimica et Biophysica Acta (2009)
Hydrogen Peroxide Generation Induces pp60src Activation in Human Platelets EVIDENCE FOR THE INVOLVEMENT OF THIS PATHWAY IN STORE-MEDIATED CALCIUM ENTRY
Juan A. Rosado;Pedro C. Redondo;Ginés M. Salido;Emilio Gómez-Arteta.
Journal of Biological Chemistry (2004)
Thrombin induces apoptotic events through the generation of reactive oxygen species in human platelets.
J. J. Lopez;G. M. Salido;E. Gómez-Arteta;J. A. Rosado.
Journal of Thrombosis and Haemostasis (2007)
Ca2+ accumulation into acidic organelles mediated by Ca2+- and vacuolar H+-ATPases in human platelets
José J. López;Cristina Camello-Almaraz;José A. Pariente;Ginés M. Salido.
Biochemical Journal (2005)
Hepatitis C virus NS5A and core proteins induce oxidative stress-mediated calcium signalling alterations in hepatocytes
Natalia Dionisio;Maria V. Garcia-Mediavilla;Sonia Sanchez-Campos;Pedro L. Majano.
Journal of Hepatology (2009)
Effect of hydrogen peroxide on Ca2+ mobilisation in human platelets through sulphydryl oxidation dependent and independent mechanisms.
Pedro C Redondo;Ginés M Salido;Juan A Rosado;José A Pariente.
Biochemical Pharmacology (2004)
Release of calcium from mitochondrial and nonmitochondrial intracellular stores in mouse pancreatic acinar cells by hydrogen peroxide.
J.A. Pariente;C. Camello;P.J. Camello;G.M. Salido.
The Journal of Membrane Biology (2001)
Hydrogen peroxide and peroxynitrite enhance Ca2+ mobilization and aggregation in platelets from type 2 diabetic patients.
Pedro C. Redondo;Isaac Jardin;Juan M. Hernández-Cruz;José A. Pariente.
Biochemical and Biophysical Research Communications (2005)
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