The scientist’s investigation covers issues in Genome-wide association study, Genetics, Meta-analysis, Single-nucleotide polymorphism and Odds ratio. He has researched Genome-wide association study in several fields, including Genetic heterogeneity, Genetic association, Allele frequency and Risk factor. His Folding research extends to the thematically linked field of Genetics.
His Odds ratio research includes elements of Case-control study and Parkinson's disease. His work carried out in the field of Case-control study brings together such families of science as Ashkenazi jews, Genotyping and Gene mutation. He interconnects Glucocerebrosidase, Computational biology and Genotype in the investigation of issues within Parkinson's disease.
His primary areas of study are Genetics, Genome-wide association study, Parkinson's disease, Genetic association and Single-nucleotide polymorphism. His research investigates the connection between Genetics and topics such as Disease that intersect with problems in Genetic architecture. The Genome-wide association study study combines topics in areas such as Minor allele frequency, Allele frequency, Meta-analysis, Age of onset and Genetic variation.
His Parkinson's disease study integrates concerns from other disciplines, such as Glucocerebrosidase, Family history and Bioinformatics. His studies in Single-nucleotide polymorphism integrate themes in fields like Odds ratio, Molecular genetics and Quantitative trait locus. His Odds ratio research incorporates elements of Case-control study and Genetic epidemiology.
Manu Sharma spends much of his time researching Genetics, Disease, Genetic association, Parkinson's disease and Genome-wide association study. His work on Allele, Gene and Transmission disequilibrium test as part of general Genetics study is frequently linked to Context, bridging the gap between disciplines. In Disease, Manu Sharma works on issues like Genetic architecture, which are connected to Comorbidity.
His Genetic association research focuses on Locus and how it connects with Risk factor, Cohort, Movement disorders and False discovery rate. His Parkinson's disease research integrates issues from Glucocerebrosidase, Dementia and Mitochondrion. His Genome-wide association study study incorporates themes from Age of onset, Genetic variation and Heritability.
Manu Sharma mostly deals with Genetics, Disease, Parkinson's disease, Genome-wide association study and Context. His research in Disease intersects with topics in Pathological and Single-nucleotide polymorphism. Manu Sharma interconnects Meta-analysis, Mendelian inheritance and Genetic architecture in the investigation of issues within Single-nucleotide polymorphism.
His biological study spans a wide range of topics, including Calcium metabolism, Calcium, Transition, Phenotype and Mitochondrion. His Genome-wide association study research is multidisciplinary, incorporating perspectives in Genetic association and Age of onset. The Genetic association study combines topics in areas such as Genetic variability, Genetic variation, Allele and Heritability.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Genome-wide association study reveals genetic risk underlying Parkinson's disease
Javier Simón-Sánchez;Claudia Schulte;Jose M Bras;Jose M Bras;Manu Sharma.
Nature Genetics (2009)
Multicenter Analysis of Glucocerebrosidase Mutations in Parkinson's Disease
Ellen Sidransky;Michael A. Nalls;Jan O. Aasly;Judith Aharon-Peretz.
The New England Journal of Medicine (2009)
α-Synuclein promotes SNARE-complex assembly in vivo and in vitro
Jacqueline Burre;Manu Sharma;Theodoros Tsetsenis;Vladimir L. Buchman.
Analysis of shared heritability in common disorders of the brain
Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Imputation of sequence variants for identification of genetic risks for Parkinson's disease: a meta-analysis of genome-wide association studies.
Michael A Nalls;Vincent Plagnol;Dena G Hernandez.
The Lancet (2011)
Identification of novel risk loci, causal insights, and heritable risk for Parkinson's disease: a meta-analysis of genome-wide association studies
Mike A Nalls;Cornelis Blauwendraat;Costanza L Vallerga;Karl Heilbron.
Lancet Neurology (2019)
Large-scale meta-analysis of genome-wide association data identifies six new risk loci for Parkinson’s disease
Mike A. Nalls;Nathan Pankratz;Christina M. Lill;Chuong B. Do.
Comprehensive research synopsis and systematic meta-analyses in Parkinson's disease genetics: the PDGene database.
Christina M. Lill;Johannes T. Roehr;Johannes T. Roehr;Matthew B. McQueen;Fotini K. Kavvoura;Fotini K. Kavvoura;Fotini K. Kavvoura.
PLOS Genetics (2012)
The ΔF508 cystic fibrosis mutation impairs domain-domain interactions and arrests post-translational folding of CFTR
Kai Du;Manu Sharma;Gergely L Lukacs.
Nature Structural & Molecular Biology (2005)
Misfolding diverts CFTR from recycling to degradation quality control at early endosomes
Manu Sharma;Francesca Pampinella;Csilla Nemes;Mohamed Benharouga.
Journal of Cell Biology (2004)
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