Michael H. Nathanson mostly deals with Cell biology, Biochemistry, Internal medicine, Endocrinology and Receptor. His research in the fields of Signal transduction and Nucleus overlaps with other disciplines such as Hepatocyte and Nucleoplasmic reticulum. As a part of the same scientific family, he mostly works in the field of Internal medicine, focusing on Cystic fibrosis transmembrane conductance regulator and, on occasion, Cytokine, Cyclic adenosine monophosphate and Bile Duct Epithelium.
His work carried out in the field of Endocrinology brings together such families of science as Confocal microscopy, Adenosine triphosphate and S cell. His biological study deals with issues like Biophysics, which deal with fields such as Acetylcholine. His study in Cytosol is interdisciplinary in nature, drawing from both Molecular biology and Calcium signaling.
His main research concerns Cell biology, Internal medicine, Endocrinology, Receptor and Calcium signaling. His Cell biology research is multidisciplinary, relying on both Biochemistry, Calcium and Inositol. His studies examine the connections between Biochemistry and genetics, as well as such issues in Biophysics, with regards to Acetylcholine.
He has researched Inositol in several fields, including Endoplasmic reticulum and Gene isoform. The concepts of his Receptor study are interwoven with issues in Molecular biology, Signal transduction, Adenosine and Stimulation. His Calcium signaling study incorporates themes from Pancreatitis and Voltage-dependent calcium channel.
Internal medicine, Calcium signaling, Cell biology, Endocrinology and Inositol are his primary areas of study. Internal medicine and General surgery are commonly linked in his work. His research integrates issues of Secretion, Flux and Second messenger system in his study of Calcium signaling.
The various areas that he examines in his Cell biology study include Apoptosis, Cell growth and Cytosol. His Endocrinology research includes elements of Phenotype and Exome sequencing. Inositol is the subject of his research, which falls under Receptor.
Michael H. Nathanson focuses on Internal medicine, Calcium signaling, Cell biology, Endocrinology and Cholangiocyte. When carried out as part of a general Internal medicine research project, his work on TBIL, Hepatitis, Retrospective cohort study and Mechanical ventilation is frequently linked to work in ARDS, therefore connecting diverse disciplines of study. His Calcium signaling research incorporates themes from Apoptosis, Endoplasmic reticulum, Cell growth and Flux.
Within one scientific family, Michael H. Nathanson focuses on topics pertaining to Cell migration under Cell growth, and may sometimes address concerns connected to Receptor. His studies deal with areas such as Nucleus and Inositol as well as Endocrinology. His Cholangiocyte study integrates concerns from other disciplines, such as Secretion, Bile duct and Cholestasis.
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Mechanisms and regulation of bile secretion.
Michael H. Nathanson;James L. Boyer.
Hepatology (1991)
Regulation of calcium signals in the nucleus by a nucleoplasmic reticulum
Wihelma Echevarría;M. Fatima Leite;Mateus T. Guerra;Warren R. Zipfel.
Nature Cell Biology (2003)
Isolated rat hepatocytes can signal to other hepatocytes and bile duct cells by release of nucleotides
Stephan F. Schlosser;Angela D. Burgstahler;Michael H. Nathanson.
Proceedings of the National Academy of Sciences of the United States of America (1996)
Type III InsP3 receptor channel stays open in the presence of increased calcium
Robert E. Hagar;Angela D. Burgstahler;Michael H. Nathanson;Barbara E. Ehrlich.
Nature (1998)
Localization of the type 3 inositol 1,4,5-trisphosphate receptor in the Ca2+ wave trigger zone of pancreatic acinar cells.
Michael H. Nathanson;Michael B. Fallon;Philip J. Padfield;Anthony R. Maranto.
Journal of Biological Chemistry (1994)
Role in host cell invasion of Trypanosoma cruzi-induced cytosolic-free Ca2+ transients.
Isabelle Tardieux;Michael H. Nathanson;Norma W. Andrews.
Journal of Experimental Medicine (1994)
Nuclear and cytosolic calcium are regulated independently
M. F. Leite;E. C. Thrower;W. Echevarria;P. Koulen.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Mechanism of Ca2+ wave propagation in pancreatic acinar cells.
M H Nathanson;P J Padfield;A J O'Sullivan;A D Burgstahler.
Journal of Biological Chemistry (1992)
Effects of tauroursodeoxycholic acid on cytosolic Ca2+ signals in isolated rat hepatocytes
Ulrich Beuers;Michael H. Nathanson;James L. Boyer.
Gastroenterology (1993)
The fate of Borrelia burgdorferi, the agent for Lyme disease, in mouse macrophages. Destruction, survival, recovery.
R R Montgomery;M H Nathanson;S E Malawista.
Journal of Immunology (1993)
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