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Michael H. Nathanson

Michael H. Nathanson

D-Index & Metrics

Biology and Biochemistry

D-Index
63
Citations
11358
World Ranking
10430
National Ranking
4528

Overview

Michael H. Nathanson is affiliated with Yale University in the United States. Their research primarily spans the fields of Medicine and Biochemistry, Genetics and Molecular Biology, with a focus on subfields such as Surgery, Molecular Biology, Epidemiology, Hepatology, and Physiology.

The main topics addressed in their work include:

  • Liver Disease Diagnosis and Treatment
  • Liver Disease and Transplantation
  • Pancreatitis Pathology and Treatment
  • Metabolism and Genetic Disorders
  • COVID-19 Clinical Research Studies
  • Long-Term Effects of COVID-19
  • Pancreatic function and diabetes

Michael H. Nathanson's frequent coauthors include:

  • Mateus T. Guerra
  • Paula Vieira Teixeira Vidigal
  • Melanie Hundt
  • Gerald I. Shulman
  • M. Fátima Leite

They have published multiple papers across well-known journals, with a recurring presence in venues such as Anaesthesia, Hepatology Communications, Hepatology, Journal of Hepatology, and Journal of Clinical Investigation.

Recent papers by Michael H. Nathanson include:

  • "Abnormal Liver Tests in COVID-19: A Retrospective Observational Cohort Study of 1,827 Patients in a Major U.S. Hospital Network" (2020, Hepatology)
  • "Glucagon stimulates gluconeogenesis by INSP3R1-mediated hepatic lipolysis" (2020, Nature)
  • "Neutrophils interact with cholangiocytes to cause cholestatic changes in alcoholic hepatitis" (2020, Gut)
  • "Regional analgesia for lower leg trauma and the risk of acute compartment syndrome" (2021, Anaesthesia)
  • "Accurate Force Fields for Atomistic Simulations of Oxides, Hydroxides, and Organic Hybrid Materials up to the Micrometer Scale" (2023, Journal of Chemical Theory and Computation)

Best Publications

  • Regulation of calcium signals in the nucleus by a nucleoplasmic reticulum

    Wihelma Echevarría;M. Fatima Leite;Mateus T. Guerra;Warren R. Zipfel

  • Isolated rat hepatocytes can signal to other hepatocytes and bile duct cells by release of nucleotides

    Stephan F. Schlosser;Angela D. Burgstahler;Michael H. Nathanson

  • Type III InsP3 receptor channel stays open in the presence of increased calcium

    Robert E. Hagar;Angela D. Burgstahler;Michael H. Nathanson;Barbara E. Ehrlich

  • Localization of the type 3 inositol 1,4,5-trisphosphate receptor in the Ca2+ wave trigger zone of pancreatic acinar cells.

    Michael H. Nathanson;Michael B. Fallon;Philip J. Padfield;Anthony R. Maranto

  • Role in host cell invasion of Trypanosoma cruzi-induced cytosolic-free Ca2+ transients.

    Isabelle Tardieux;Michael H. Nathanson;Norma W. Andrews

  • Nuclear and cytosolic calcium are regulated independently

    M. F. Leite;E. C. Thrower;W. Echevarria;P. Koulen

  • Succinate is a paracrine signal for liver damage.

    Paulo Renato A.V. Correa;Emma A. Kruglov;Mayerson Thompson;Mayerson Thompson;M. Fatima Leite

  • Mechanism of Ca2+ wave propagation in pancreatic acinar cells.

    M H Nathanson;P J Padfield;A J O'Sullivan;A D Burgstahler

  • Tauroursodeoxycholic acid stimulates hepatocellular exocytosis and mobilizes extracellular Ca++ mechanisms defective in cholestasis.

    U Beuers;M H Nathanson;C M Isales;J L Boyer

  • Connexin 43 hemichannels mediate the Ca2+ influx induced by extracellular alkalinization

    Kurt A. Schalper;Helmuth A. Sánchez;Sung C. Lee;Guillermo A. Altenberg

  • c-Met must translocate to the nucleus to initiate calcium signals.

    Dawidson A Gomes;Dawidson A Gomes;Michele Angela Rodrigues;Michele Angela Rodrigues;M Fatima Leite;Marcus V Gomez

  • The fate of Borrelia burgdorferi, the agent for Lyme disease, in mouse macrophages. Destruction, survival, recovery.

    R R Montgomery;M H Nathanson;S E Malawista

  • Nucleoplasmic calcium is required for cell proliferation.

    Michele A. Rodrigues;Michele A. Rodrigues;Dawidson A. Gomes;M. Fatima Leite;Wayne Grant

  • Ca2+ waves are organized among hepatocytes in the intact organ

    M. H. Nathanson;A. D. Burgstahler;A. Mennone;M. B. Fallon

  • Loss of Inositol 1,4,5-trisphosphate receptors from bile duct epithelia is a common event in cholestasis

    Kazunori Shibao;Keiji Hirata;Marie E Robert;Michael H Nathanson

  • Multiphoton imaging can be used for microscopic examination of intact human gastrointestinal mucosa ex vivo.

    Jason N. Rogart;Jun Nagata;Caroline S. Loeser;Robert D. Roorda

  • Proinflammatory cytokines inhibit secretion in rat bile duct epithelium.

    Carlo Spirlı̀;Michael H. Nathanson;Romina Fiorotto;Elena Duner

  • Cyclic AMP regulates bicarbonate secretion in cholangiocytes through release of ATP into bile.

    Noritaka Minagawa;Jun Nagata;Jun Nagata;Kazunori Shibao;Kazunori Shibao;Anatoliy I. Masyuk

  • Glucagon stimulates gluconeogenesis by INSP3R1-mediated hepatic lipolysis

    Rachel J. Perry;Dongyan Zhang;Mateus T. Guerra;Allison L. Brill

  • The type II inositol 1,4,5-trisphosphate receptor can trigger Ca2+ waves in rat hepatocytes

    Keiji Hirata;Thomas Pusl;Allison F. O'Neill;Jonathan A. Dranoff

Frequent Co-Authors

Barbara E. Ehrlich
Barbara E. Ehrlich Yale University
James L. Boyer
James L. Boyer Yale University
Emad M. El-Omar
Emad M. El-Omar University of New South Wales
Rajiv Jalan
Rajiv Jalan University College London
M. Bishr Omary
M. Bishr Omary University of Michigan–Ann Arbor
Jerrold R. Turner
Jerrold R. Turner Brigham and Women's Hospital
David E. Cohen
David E. Cohen Weill Cornell Medical College in Qatar
Richard M. Peek
Richard M. Peek Vanderbilt University Medical Center

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