What is he best known for?
The fields of study he is best known for:
His primary areas of study are Helicobacter pylori, CagA, Immunology, Cancer and Gastritis.
His Helicobacter pylori research is multidisciplinary, relying on both Stomach, Gastric mucosa and Microbiology.
The concepts of his CagA study are interwoven with issues in Apoptosis, Secretion, Molecular biology, Proinflammatory cytokine and Serology.
Richard M. Peek interconnects Gastrointestinal tract and Risk factor in the investigation of issues within Immunology.
His Cancer research integrates issues from Pathogen, Immune system and Virulence.
His Gastritis study integrates concerns from other disciplines, such as Pathogenesis and Adenocarcinoma.
His most cited work include:
- Helicobacter pylori and gastrointestinal tract adenocarcinomas (1464 citations)
- Infection with Helicobacter pylori Strains Possessing cagA Is Associated with an Increased Risk of Developing Adenocarcinoma of the Stomach (1464 citations)
- Mosaicism in Vacuolating Cytotoxin Alleles of Helicobacter pylori ASSOCIATION OF SPECIFIC vacA TYPES WITH CYTOTOXIN PRODUCTION AND PEPTIC ULCERATION (1375 citations)
What are the main themes of his work throughout his whole career to date?
Richard M. Peek focuses on Helicobacter pylori, CagA, Immunology, Cancer and Cancer research.
Richard M. Peek has included themes like Molecular biology, Gastric mucosa and Microbiology in his Helicobacter pylori study.
He combines subjects such as Secretion, Antigen, Interleukin 8 and Genotype with his study of CagA.
His biological study spans a wide range of topics, including Stomach and Chronic gastritis.
His Cancer research is multidisciplinary, incorporating perspectives in Helicobacter, Disease and Risk factor.
His research integrates issues of Carcinogenesis, Signal transduction and Epidermal growth factor receptor in his study of Cancer research.
He most often published in these fields:
- Helicobacter pylori (78.78%)
- CagA (32.42%)
- Immunology (26.92%)
What were the highlights of his more recent work (between 2015-2021)?
- Helicobacter pylori (78.78%)
- Cancer research (21.41%)
- Cancer (24.56%)
In recent papers he was focusing on the following fields of study:
Richard M. Peek mostly deals with Helicobacter pylori, Cancer research, Cancer, Internal medicine and CagA.
Richard M. Peek has researched Helicobacter pylori in several fields, including Carcinogenesis, Secretion, Immunology, Microbiology and Virulence.
His Cancer research study combines topics from a wide range of disciplines, such as Inflammation, PI3K/AKT/mTOR pathway and p14arf.
His work deals with themes such as Helicobacter Infections, Helicobacter, Disease and Oncology, which intersect with Cancer.
His studies deal with areas such as Gastroenterology and Serology as well as Internal medicine.
The study incorporates disciplines such as Virulence factor and Gastric mucosa in addition to CagA.
Between 2015 and 2021, his most popular works were:
- Pathobiology of Helicobacter pylori-induced Gastric Cancer (296 citations)
- Different gastric microbiota compositions in two human populations with high and low gastric cancer risk in Colombia (83 citations)
- miR-101-2, miR-125b-2 and miR-451a act as potential tumor suppressors in gastric cancer through regulation of the PI3K/AKT/mTOR pathway (67 citations)
In his most recent research, the most cited papers focused on:
Helicobacter pylori, Carcinogenesis, Cancer, Cancer research and CagA are his primary areas of study.
His Helicobacter pylori research is multidisciplinary, incorporating elements of Stomach, Gastric mucosa and Microbiology.
His Cancer research incorporates themes from Disease, Immunology and Cell growth.
The various areas that he examines in his Immunology study include Microbiome, Zoology, Intestinal metaplasia and Histopathology.
His Cancer research study combines topics in areas such as Epidermal growth factor receptor, Cancer cell, microRNA, Regulation of gene expression and PI3K/AKT/mTOR pathway.
His work carried out in the field of CagA brings together such families of science as Ubiquitin ligase, p14arf and Carcinogen.
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