2020 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of study are Helicobacter pylori, CagA, Immunology, Cancer and Gastritis. His Helicobacter pylori research is multidisciplinary, relying on both Stomach, Gastric mucosa and Microbiology. The concepts of his CagA study are interwoven with issues in Apoptosis, Secretion, Molecular biology, Proinflammatory cytokine and Serology.
Richard M. Peek interconnects Gastrointestinal tract and Risk factor in the investigation of issues within Immunology. His Cancer research integrates issues from Pathogen, Immune system and Virulence. His Gastritis study integrates concerns from other disciplines, such as Pathogenesis and Adenocarcinoma.
Richard M. Peek focuses on Helicobacter pylori, CagA, Immunology, Cancer and Cancer research. Richard M. Peek has included themes like Molecular biology, Gastric mucosa and Microbiology in his Helicobacter pylori study. He combines subjects such as Secretion, Antigen, Interleukin 8 and Genotype with his study of CagA.
His biological study spans a wide range of topics, including Stomach and Chronic gastritis. His Cancer research is multidisciplinary, incorporating perspectives in Helicobacter, Disease and Risk factor. His research integrates issues of Carcinogenesis, Signal transduction and Epidermal growth factor receptor in his study of Cancer research.
Richard M. Peek mostly deals with Helicobacter pylori, Cancer research, Cancer, Internal medicine and CagA. Richard M. Peek has researched Helicobacter pylori in several fields, including Carcinogenesis, Secretion, Immunology, Microbiology and Virulence. His Cancer research study combines topics from a wide range of disciplines, such as Inflammation, PI3K/AKT/mTOR pathway and p14arf.
His work deals with themes such as Helicobacter Infections, Helicobacter, Disease and Oncology, which intersect with Cancer. His studies deal with areas such as Gastroenterology and Serology as well as Internal medicine. The study incorporates disciplines such as Virulence factor and Gastric mucosa in addition to CagA.
Helicobacter pylori, Carcinogenesis, Cancer, Cancer research and CagA are his primary areas of study. His Helicobacter pylori research is multidisciplinary, incorporating elements of Stomach, Gastric mucosa and Microbiology. His Cancer research incorporates themes from Disease, Immunology and Cell growth.
The various areas that he examines in his Immunology study include Microbiome, Zoology, Intestinal metaplasia and Histopathology. His Cancer research study combines topics in areas such as Epidermal growth factor receptor, Cancer cell, microRNA, Regulation of gene expression and PI3K/AKT/mTOR pathway. His work carried out in the field of CagA brings together such families of science as Ubiquitin ligase, p14arf and Carcinogen.
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Helicobacter pylori and gastrointestinal tract adenocarcinomas
Richard M. Peek;Martin J. Blaser.
Nature Reviews Cancer (2002)
Mosaicism in Vacuolating Cytotoxin Alleles of Helicobacter pylori ASSOCIATION OF SPECIFIC vacA TYPES WITH CYTOTOXIN PRODUCTION AND PEPTIC ULCERATION
John C. Atherton;Ping Cao;Richard M. Peek;Murali K.R. Tummuru.
Journal of Biological Chemistry (1995)
Infection with Helicobacter pylori Strains Possessing cagA Is Associated with an Increased Risk of Developing Adenocarcinoma of the Stomach
Martin J. Blaser;Guillermo I. Perez-Perez;Harry Kleanthous;Timothy L. Cover.
Cancer Research (1995)
Helicobacter pylori and Gastric Cancer: Factors That Modulate Disease Risk
Lydia E. Wroblewski;Richard M. Peek;Richard M. Peek;Keith T. Wilson;Keith T. Wilson.
Clinical Microbiology Reviews (2010)
Helicobacter pylori: gastric cancer and beyond
D. Brent Polk;Richard M. Peek.
Nature Reviews Cancer (2010)
Clinical and pathological importance of heterogeneity in vacA, the vacuolating cytotoxin gene of Helicobacter pylori.
JC Atherton;RM Peek;KT Tham;TL Cover.
Heightened inflammatory response and cytokine expression in vivo to cagA+ Helicobacter pylori strains.
R. M. Peek;G. G. Miller;K. T. Tham;G. I. Perez-Perez.
Laboratory Investigation (1995)
Helicobacter infection and gastric neoplasia
Richard M Peek;Jean E Crabtree.
The Journal of Pathology (2006)
Helicobacter pylori CagA interacts with E-cadherin and deregulates the β-catenin signal that promotes intestinal transdifferentiation in gastric epithelial cells
N Murata-Kamiya;Y Kurashima;Y Teishikata;Y Yamahashi.
Activation of β-catenin by carcinogenic Helicobacter pylori
Aime T. Franco;Dawn A. Israel;Mary K. Washington;Uma Krishna.
Proceedings of the National Academy of Sciences of the United States of America (2005)
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