Rupesh Chaturvedi spends much of his time researching Molecular biology, Apoptosis, Helicobacter pylori, Inflammation and Immunology. His work deals with themes such as Spermine oxidase and Ornithine decarboxylase, which intersect with Molecular biology. His work carried out in the field of Apoptosis brings together such families of science as Oxidative stress and Signal transduction.
His Helicobacter pylori study combines topics in areas such as Immunohistochemistry and CagA. His research in Inflammation intersects with topics in Colitis, Inflammatory bowel disease and Pharmacology. His research investigates the connection between Programmed cell death and topics such as Small interfering RNA that intersect with problems in Carcinogenesis and Cancer research.
His main research concerns Helicobacter pylori, Immunology, Molecular biology, Cancer research and Inflammation. His studies deal with areas such as CagA, Arginase, Gastric mucosa and Microbiology as well as Helicobacter pylori. His study looks at the relationship between Immunology and fields such as Ulcerative colitis, as well as how they intersect with chemical problems.
His studies examine the connections between Molecular biology and genetics, as well as such issues in Apoptosis, with regards to Small interfering RNA. His Cancer research study integrates concerns from other disciplines, such as Oxidative stress, Cell and Carcinogenesis, Cancer, Epidermal growth factor receptor. In his study, Signal transduction is inextricably linked to Intestinal mucosa, which falls within the broad field of Inflammation.
The scientist’s investigation covers issues in Molecular biology, Helicobacter pylori, Cancer, Immunology and Spermine oxidase. His Molecular biology research integrates issues from Polyamine metabolism, Proteome, Apoptosis and Arginase. His biological study deals with issues like Gastric mucosa, which deal with fields such as Macrophage polarization, MMP7, Genetic model and Western blot.
His Cancer study incorporates themes from Inflammation, Cancer research, Pharmacology and Bioinformatics. A large part of his Immunology studies is devoted to Interleukin 8. His Spermine oxidase study also includes fields such as
Rupesh Chaturvedi mostly deals with Cancer, Helicobacter pylori, Spermine oxidase, Molecular biology and Spermine. His biological study spans a wide range of topics, including Precision medicine, Therapeutic approach, Cancer research, Bioinformatics and Pharmacology. In most of his Helicobacter pylori studies, his work intersects topics such as Immunology.
His Spermine oxidase research incorporates elements of ARG2, Arginase and Adenosylmethionine decarboxylase, Ornithine decarboxylase. His Molecular biology research is multidisciplinary, relying on both DNA methylation, Carcinogenesis, microRNA, Regulation of gene expression and Gene silencing. His Spermine research includes themes of Oxidative stress, Atrophic gastritis and Gastric Dysplasia.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Colon-specific delivery of a probiotic-derived soluble protein ameliorates intestinal inflammation in mice through an EGFR-dependent mechanism
Fang Yan;Hanwei Cao;Timothy L. Cover;M. Kay Washington.
Journal of Clinical Investigation (2011)
Designing a broad-spectrum integrative approach for cancer prevention and treatment
Keith I. Block;Charlotte Gyllenhaal;Leroy Lowe;Amedeo Amedei.
Seminars in Cancer Biology (2015)
Spermine Oxidation Induced by Helicobacter pylori Results in Apoptosis and DNA Damage Implications for Gastric Carcinogenesis
Hangxiu Xu;Rupesh Chaturvedi;Rupesh Chaturvedi;Yulan Cheng;Yulan Cheng;Francoise I. Bussiere;Francoise I. Bussiere.
Cancer Research (2004)
Induction of polyamine oxidase 1 by Helicobacter pylori causes macrophage apoptosis by hydrogen peroxide release and mitochondrial membrane depolarization.
Rupesh Chaturvedi;Yulan Cheng;Mohammad Asim;Françoise I. Bussière.
Journal of Biological Chemistry (2004)
Phylogeographic origin of Helicobacter pylori is a determinant of gastric cancer risk
Thibaut de Sablet;M Blanca Piazuelo;Carrie L Shaffer;Barbara G Schneider.
Gut (2011)
Spermine causes loss of innate immune response to Helicobacter pylori by inhibition of inducible nitric-oxide synthase translation.
Françoise I. Bussière;Rupesh Chaturvedi;Yulan Cheng;Alain P. Gobert.
Journal of Biological Chemistry (2005)
Protective Role of Arginase in a Mouse Model of Colitis
Alain P. Gobert;Yulan Cheng;Mahmood Akhtar;Benjamin D. Mersey.
Journal of Immunology (2004)
Spermine Oxidase Mediates the Gastric Cancer Risk Associated With Helicobacter pylori CagA
Rupesh Chaturvedi;Mohammad Asim;Mohammad Asim;Judith Romero-Gallo;Daniel P. Barry;Daniel P. Barry.
Gastroenterology (2011)
Tumor Suppressor Function of the Plasma Glutathione Peroxidase Gpx3 in Colitis-Associated Carcinoma
Caitlyn W. Barrett;Wei Ning;Xi Chen;Jesse Joshua Smith.
Cancer Research (2013)
Berberine promotes recovery of colitis and inhibits inflammatory responses in colonic macrophages and epithelial cells in DSS-treated mice
Fang Yan;Lihong Wang;Yan Shi;Hanwei Cao.
American Journal of Physiology-gastrointestinal and Liver Physiology (2012)
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