Douglas E. Berg spends much of his time researching Microbiology, Helicobacter pylori, Genetics, Gene and Virulence. His Microbiology research is multidisciplinary, relying on both Bacterial genetics, Bacterial adhesin and Bacteria. His study in Helicobacter pylori is interdisciplinary in nature, drawing from both Mutation, Immunology and Gastric mucosa.
His work in Gene tackles topics such as Molecular biology which are related to areas like DNA. His biological study spans a wide range of topics, including Pathogen and Vibrio cholerae. His research investigates the link between Genomic organization and topics such as Sequence analysis that cross with problems in Nucleic acid sequence.
Genetics, Helicobacter pylori, Gene, Microbiology and Molecular biology are his primary areas of study. His study in Transposable element, Plasmid, DNA sequencing, Genome and DNA falls under the purview of Genetics. The concepts of his Helicobacter pylori study are interwoven with issues in CagA, Immunology, Genotype and Metronidazole.
His Microbiology research is multidisciplinary, incorporating perspectives in Bacteria, Bacterial adhesin, Virulence and Polymerase chain reaction. Douglas E. Berg focuses mostly in the field of Polymerase chain reaction, narrowing it down to topics relating to RAPD and, in certain cases, DNA profiling and Primer. His study in Molecular biology is interdisciplinary in nature, drawing from both Nucleic acid sequence, Ribosomal RNA, Operon, Escherichia coli and Lambda phage.
His primary areas of study are Helicobacter pylori, Genetics, Microbiology, Gene and CagA. Douglas E. Berg combines subjects such as Virology, Immunology, Disease, Sequence analysis and Haplotype with his study of Helicobacter pylori. His Copy-number variation, DNA, Strain and Helicobacter study in the realm of Genetics interacts with subjects such as Transmission.
His Microbiology research incorporates themes from Bacterial adhesin, Virulence and Gastric mucosa. The various areas that Douglas E. Berg examines in his CagA study include Polymerase chain reaction, Genotype and Phosphorylation. His research integrates issues of Reversion and Escherichia coli in his study of Genome.
Douglas E. Berg mainly investigates Microbiology, Helicobacter pylori, Genetics, Gene and Bacterial adhesin. The concepts of his Microbiology study are interwoven with issues in Bacterial genetics, Gastric mucosa and Virulence. Douglas E. Berg has included themes like CagA, Whole genome sequencing, Immunology, Genetic variation and Polymerase chain reaction in his Helicobacter pylori study.
His study in Amino acid extends to Gene with its themes. His Bacterial adhesin research incorporates elements of Complementation, Genome and ABO blood group system. Molecular biology is closely connected to Secretion in his research, which is encompassed under the umbrella topic of Virology.
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The complete genome sequence of the gastric pathogen Helicobacter pylori
Jean-F. Tomb;Owen White;Anthony R. Kerlavage;Rebecca A. Clayton.
Helicobacter pylori Adhesin Binding Fucosylated Histo-Blood Group Antigens Revealed by Retagging
Dag Ilver;Anna Arnqvist;Johan Ögren;Inga-Maria Frick.
DNA diversity among clinical isolates of Helicobacter pylori detected by PCR-based RAPD fingerprinting
Natalia Akopyanz;Nickolai O. Bukanov;T.Ulf Westblom;Stephen Kresovich.
Nucleic Acids Research (1992)
Helicobacter pylori SabA adhesin in persistent infection and chronic inflammation.
Jafar Mahdavi;Berit Sondén;Martina Hurtig;Farzad O. Olfat;Farzad O. Olfat.
Culture Independent Analysis of Ileal Mucosa Reveals a Selective Increase in Invasive Escherichia Coli of Novel Phylogeny Relative to Depletion of Clostridiales in Crohn's Disease Involving the Ileum
Martin Baumgart;Belgin Dogan;Mark Rishniw;Gil Weitzman.
The ISME Journal (2007)
Analyses of the cag pathogenicity island of Helicobacter pylori
Akopyants Ns;Clifton Sw;Kersulyte D;Crabtree Je.
Molecular Microbiology (2002)
Helicobacter pylori genetic diversity and risk of human disease
Martin J. Blaser;Douglas E. Berg.
Journal of Clinical Investigation (2001)
Metronidazole resistance in Helicobacter pylori is due to null mutations in a gene (rdxA) that encodes an oxygen‐insensitive NADPH nitroreductase
Goodwin A;Kersulyte D;Sisson G;Veldhuyzen van Zanten Sj.
Molecular Microbiology (1998)
Recombination and clonal groupings within Helicobacter pylori from different geographical regions.
M Achtman;T Azuma;D E Berg;Y Ito.
Molecular Microbiology (1999)
Functional adaptation of BabA the H. pylori ABO blood group antigen binding adhesin
Marina Aspholm-Hurtig;Giedrius Dailide;Martina Lahmann;Awdhesh Kalia.
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