Cell biology, Voltage-dependent calcium channel, Receptor, Biophysics and Endoplasmic reticulum are his primary areas of study. His Cell biology study integrates concerns from other disciplines, such as Calcium metabolism and Programmed cell death. His biological study spans a wide range of topics, including Extracellular, Inositol trisphosphate, Inositol and Intracellular.
His Extracellular research incorporates themes from Ryanodine receptor and Calcium signaling. His studies in Receptor integrate themes in fields like Motor neuron, Amyotrophic lateral sclerosis and Neuroscience. His Biophysics study combines topics in areas such as Biochemistry and Ion transporter.
Geert Callewaert focuses on Biophysics, Cell biology, Biochemistry, Endoplasmic reticulum and Voltage-dependent calcium channel. His studies in Biophysics integrate themes in fields like Patch clamp and Calcium. His research on Cell biology frequently connects to adjacent areas such as Calcium metabolism.
The Endoplasmic reticulum study which covers Intracellular that intersects with Endocrinology, Internal medicine and Adenosine triphosphate. His Voltage-dependent calcium channel research is multidisciplinary, incorporating perspectives in Receptor, Ryanodine receptor, Inositol and Calmodulin. The concepts of his Receptor study are interwoven with issues in Motor neuron, Amyotrophic lateral sclerosis and Binding site.
Geert Callewaert mainly focuses on Cell biology, Biochemistry, Biophysics, Extracellular and Patch clamp. His work in Homeostasis and Endoplasmic reticulum is related to Cell biology. The various areas that Geert Callewaert examines in his Endoplasmic reticulum study include Molecular biology and Mitochondrion, Mitochondrial respiratory chain.
His work on Yeast, Programmed cell death and Oxidative phosphorylation as part of general Biochemistry study is frequently connected to Neurotoxicity, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His research integrates issues of Spike frequency, Quantum dot, Membrane, Odor and Nanofiber in his study of Biophysics. His research investigates the link between Patch clamp and topics such as Depolarization that cross with problems in Dorsal root ganglion, Membrane potential, Cotransporter, Verapamil and Repolarization.
His primary areas of investigation include Cell biology, Mitochondrion, Alzheimer's disease, Biochemistry and Chip. His work in the fields of Homeostasis overlaps with other areas such as Superoxide. As part of the same scientific family, Geert Callewaert usually focuses on Mitochondrion, concentrating on SOD1 and intersecting with Mitochondrial respiratory chain, Endoplasmic reticulum, Molecular biology and Oxidative phosphorylation.
His Alzheimer's disease research is multidisciplinary, incorporating elements of Hippocampus, Neuroscience, Tauopathy and Amyloid. Geert Callewaert interconnects Cotransporter and Biophysics in the investigation of issues within Biochemistry. His research in Biophysics intersects with topics in Patch clamp, Dorsal root ganglion and Premovement neuronal activity.
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Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio.
Inna Kuperstein;Kerensa Broersen;Kerensa Broersen;Iryna Benilova;Iryna Benilova;Iryna Benilova;Jef Rozenski.
The EMBO Journal (2010)
Abnormal intracellular ca(2+)homeostasis and disease.
L. Missiaen;W. Robberecht;L.Van Den Bosch;G. Callewaert.
Cell Calcium (2000)
Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity
Philip Van Damme;Elke Bogaert;Maarten Dewil;Nicole Hersmus.
Proceedings of the National Academy of Sciences of the United States of America (2007)
2-Aminoethoxydiphenyl borate affects the inositol 1,4,5-trisphosphate receptor, the intracellular Ca2+pump and the non-specific Ca2+leak from the non-mitochondrial Ca2+stores in permeabilized A7r5 cells
L. Missiaen;G. Callewaert;H. De Smedt;J.B. Parys.
Cell Calcium (2001)
Shear stress-induced calcium transients in endothelial cells from human umbilical cord veins.
G Schwarz;Geert Callewaert;Guillaume Droogmans;Bernd Nilius.
The Journal of Physiology (1992)
Caspase-3-induced Truncation of Type 1 Inositol Trisphosphate Receptor Accelerates Apoptotic Cell Death and Induces Inositol Trisphosphate-independent Calcium Release during Apoptosis
Zerihun Assefa;Geert Bultynck;Karolina Szlufcik;Nael Nadif Kasri.
Journal of Biological Chemistry (2004)
Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance
Bavo Heeman;Chris Van den Haute;Sarah-Ann Aelvoet;Federica Valsecchi.
Journal of Cell Science (2011)
Inhibition and Rapid Recovery of Ca2+ Current During Ca2+ Release From Sarcoplasmic Reticulum in Guinea Pig Ventricular Myocytes
Karin R. Sipido;Geert Callewaert;Edward Carmeliet.
Circulation Research (1995)
The Ca2+/Mn2+ pumps in the Golgi apparatus.
Kurt Van Baelen;Leonard Dode;Jo Vanoevelen;Geert Callewaert.
Biochimica et Biophysica Acta (2004)
Thimerosal stimulates Ca2+ flux through inositol 1,4,5-trisphosphate receptor type 1, but not type 3, via modulation of an isoform-specific Ca2+-dependent intramolecular interaction.
Geert Bultynck;Karolina Szlufcik;Nael Nadif Kasri;Zerihun Assefa.
Biochemical Journal (2004)
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