D-Index & Metrics Best Publications
Geert Callewaert

Geert Callewaert

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 49 Citations 6,296 86 World Ranking 10754 National Ranking 192

Overview

What is he best known for?

The fields of study he is best known for:

  • Biochemistry
  • Amino acid
  • Neuron

Cell biology, Voltage-dependent calcium channel, Receptor, Biophysics and Endoplasmic reticulum are his primary areas of study. His Cell biology study integrates concerns from other disciplines, such as Calcium metabolism and Programmed cell death. His biological study spans a wide range of topics, including Extracellular, Inositol trisphosphate, Inositol and Intracellular.

His Extracellular research incorporates themes from Ryanodine receptor and Calcium signaling. His studies in Receptor integrate themes in fields like Motor neuron, Amyotrophic lateral sclerosis and Neuroscience. His Biophysics study combines topics in areas such as Biochemistry and Ion transporter.

His most cited work include:

  • Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio. (353 citations)
  • Abnormal intracellular ca(2+)homeostasis and disease. (207 citations)
  • Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity (166 citations)

What are the main themes of his work throughout his whole career to date?

Geert Callewaert focuses on Biophysics, Cell biology, Biochemistry, Endoplasmic reticulum and Voltage-dependent calcium channel. His studies in Biophysics integrate themes in fields like Patch clamp and Calcium. His research on Cell biology frequently connects to adjacent areas such as Calcium metabolism.

The Endoplasmic reticulum study which covers Intracellular that intersects with Endocrinology, Internal medicine and Adenosine triphosphate. His Voltage-dependent calcium channel research is multidisciplinary, incorporating perspectives in Receptor, Ryanodine receptor, Inositol and Calmodulin. The concepts of his Receptor study are interwoven with issues in Motor neuron, Amyotrophic lateral sclerosis and Binding site.

He most often published in these fields:

  • Biophysics (36.96%)
  • Cell biology (32.61%)
  • Biochemistry (21.74%)

What were the highlights of his more recent work (between 2008-2020)?

  • Cell biology (32.61%)
  • Biochemistry (21.74%)
  • Biophysics (36.96%)

In recent papers he was focusing on the following fields of study:

Geert Callewaert mainly focuses on Cell biology, Biochemistry, Biophysics, Extracellular and Patch clamp. His work in Homeostasis and Endoplasmic reticulum is related to Cell biology. The various areas that Geert Callewaert examines in his Endoplasmic reticulum study include Molecular biology and Mitochondrion, Mitochondrial respiratory chain.

His work on Yeast, Programmed cell death and Oxidative phosphorylation as part of general Biochemistry study is frequently connected to Neurotoxicity, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His research integrates issues of Spike frequency, Quantum dot, Membrane, Odor and Nanofiber in his study of Biophysics. His research investigates the link between Patch clamp and topics such as Depolarization that cross with problems in Dorsal root ganglion, Membrane potential, Cotransporter, Verapamil and Repolarization.

Between 2008 and 2020, his most popular works were:

  • Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio. (353 citations)
  • Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance (137 citations)
  • Single-cell recording and stimulation with a 16k micro-nail electrode array integrated on a 0.18 μm CMOS chip (55 citations)

In his most recent research, the most cited papers focused on:

  • Biochemistry
  • Amino acid
  • Genetics

His primary areas of investigation include Cell biology, Mitochondrion, Alzheimer's disease, Biochemistry and Chip. His work in the fields of Homeostasis overlaps with other areas such as Superoxide. As part of the same scientific family, Geert Callewaert usually focuses on Mitochondrion, concentrating on SOD1 and intersecting with Mitochondrial respiratory chain, Endoplasmic reticulum, Molecular biology and Oxidative phosphorylation.

His Alzheimer's disease research is multidisciplinary, incorporating elements of Hippocampus, Neuroscience, Tauopathy and Amyloid. Geert Callewaert interconnects Cotransporter and Biophysics in the investigation of issues within Biochemistry. His research in Biophysics intersects with topics in Patch clamp, Dorsal root ganglion and Premovement neuronal activity.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio.

Inna Kuperstein;Kerensa Broersen;Kerensa Broersen;Iryna Benilova;Iryna Benilova;Iryna Benilova;Jef Rozenski.
The EMBO Journal (2010)

507 Citations

Abnormal intracellular ca(2+)homeostasis and disease.

L. Missiaen;W. Robberecht;L.Van Den Bosch;G. Callewaert.
Cell Calcium (2000)

314 Citations

Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity

Philip Van Damme;Elke Bogaert;Maarten Dewil;Nicole Hersmus.
Proceedings of the National Academy of Sciences of the United States of America (2007)

228 Citations

2-Aminoethoxydiphenyl borate affects the inositol 1,4,5-trisphosphate receptor, the intracellular Ca2+pump and the non-specific Ca2+leak from the non-mitochondrial Ca2+stores in permeabilized A7r5 cells

L. Missiaen;G. Callewaert;H. De Smedt;J.B. Parys.
Cell Calcium (2001)

227 Citations

Shear stress-induced calcium transients in endothelial cells from human umbilical cord veins.

G Schwarz;Geert Callewaert;Guillaume Droogmans;Bernd Nilius.
The Journal of Physiology (1992)

189 Citations

Caspase-3-induced Truncation of Type 1 Inositol Trisphosphate Receptor Accelerates Apoptotic Cell Death and Induces Inositol Trisphosphate-independent Calcium Release during Apoptosis

Zerihun Assefa;Geert Bultynck;Karolina Szlufcik;Nael Nadif Kasri.
Journal of Biological Chemistry (2004)

181 Citations

Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance

Bavo Heeman;Chris Van den Haute;Sarah-Ann Aelvoet;Federica Valsecchi.
Journal of Cell Science (2011)

178 Citations

Inhibition and Rapid Recovery of Ca2+ Current During Ca2+ Release From Sarcoplasmic Reticulum in Guinea Pig Ventricular Myocytes

Karin R. Sipido;Geert Callewaert;Edward Carmeliet.
Circulation Research (1995)

174 Citations

The Ca2+/Mn2+ pumps in the Golgi apparatus.

Kurt Van Baelen;Leonard Dode;Jo Vanoevelen;Geert Callewaert.
Biochimica et Biophysica Acta (2004)

167 Citations

Thimerosal stimulates Ca2+ flux through inositol 1,4,5-trisphosphate receptor type 1, but not type 3, via modulation of an isoform-specific Ca2+-dependent intramolecular interaction.

Geert Bultynck;Karolina Szlufcik;Nael Nadif Kasri;Zerihun Assefa.
Biochemical Journal (2004)

153 Citations

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