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Medicine

D-Index
144
Citations
80034
World Ranking
1442
National Ranking
843

Research.com Recognitions

  • 2013 - Fellow of the American Association for the Advancement of Science (AAAS)
  • 2012 - Distinguished Scientist Award, American Heart Association

Overview

Donald M. Bers is affiliated with the University of California, Davis in the United States. Their research spans multiple areas within medicine, biochemistry, genetics, and molecular biology, with a strong focus on cardiology and cardiovascular medicine. The scientist's work has contributed significantly to understanding cardiac electrophysiology, ion channel regulation, receptor mechanisms, and mitochondrial function among other related topics.

Their main fields of study include:

  • Medicine
  • Biochemistry, Genetics and Molecular Biology

Subfields of study prominently feature:

  • Cardiology and Cardiovascular Medicine
  • Molecular Biology
  • Cellular and Molecular Neuroscience
  • Physiology
  • Pathology and Forensic Medicine

The primary research topics covered by Donald M. Bers encompass:

  • Cardiac electrophysiology and arrhythmias
  • Ion channel regulation and function
  • Receptor Mechanisms and Signaling
  • Neuroscience and Neural Engineering
  • Cardiomyopathy and Myosin Studies
  • Mitochondrial Function and Pathology
  • Cardiovascular Function and Risk Factors

Publications appear frequently in several specialized venues, including the following journals where they have multiple contributions:

  • Biophysical Journal
  • Circulation Research
  • The Journal of Physiology
  • bioRxiv (Cold Spring Harbor Laboratory)
  • Journal of Molecular and Cellular Cardiology

Several recent papers authored by Donald M. Bers include:

  • Metabolic Maturation Media Improve Physiological Function of Human iPSC-Derived Cardiomyocytes, 2020, Cell Reports
  • Hyperglycemia Acutely Increases Cytosolic Reactive Oxygen Species via O-linked GlcNAcylation and CaMKII Activation in Mouse Ventricular Myocytes, 2020, Circulation Research
  • A 20/20 view of ANT function in mitochondrial biology and necrotic cell death, 2020, Journal of Molecular and Cellular Cardiology
  • Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors, 2021, Basic Research in Cardiology
  • Intracellular β 1-Adrenergic Receptors and Organic Cation Transporter 3 Mediate Phospholamban Phosphorylation to Enhance Cardiac Contractility, 2020, Circulation Research

Frequent coauthors in Bers's work include:

  • Bence Hegyi
  • Julie Bossuyt
  • Christopher Y. Ko
  • Rǎzvan L. Cornea
  • Robyn T. Rebbeck

Donald M. Bers has received notable recognitions including being named a Fellow of the American Association for the Advancement of Science (AAAS) in 2013 and receiving the Distinguished Scientist Award from the American Heart Association in 2012.

Best Publications

  • Cardiac excitation–contraction coupling

    Donald M Bers

  • Excitation-Contraction Coupling and Cardiac Contractile Force

    Donald M. Bers

  • Excitation-Contraction Coupling and Cardiac Contractile Force.

    Donald M. Bers;Yujie Zhu

  • Calcium cycling and signaling in cardiac myocytes.

    Donald M Bers

  • Arrhythmogenesis and contractile dysfunction in heart failure: Roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness.

    Steven M. Pogwizd;Klaus Schlotthauer;Li Li;Weilong Yuan

  • Relaxation in rabbit and rat cardiac cells: species‐dependent differences in cellular mechanisms.

    J. W M Bassani;R. A. Bassani;Donald M Bers

  • Ca2+/Calmodulin–Dependent Protein Kinase Modulates Cardiac Ryanodine Receptor Phosphorylation and Sarcoplasmic Reticulum Ca2+ Leak in Heart Failure

    Xun Ai;Jerry W. Curran;Thomas R. Shannon;Donald M. Bers

  • Calcium fluxes involved in control of cardiac myocyte contraction.

    Donald M. Bers

  • Abnormal Calcium Handling Properties Underlie Familial Hypertrophic Cardiomyopathy Pathology in Patient-Specific Induced Pluripotent Stem Cells

    Feng Lan;Andrew S. Lee;Ping Liang;Veronica Sanchez-Freire

  • The δC Isoform of CaMKII Is Activated in Cardiac Hypertrophy and Induces Dilated Cardiomyopathy and Heart Failure

    Tong Zhang;Lars S. Maier;Lars S. Maier;Nancy D. Dalton;Shigeki Miyamoto

  • A Mathematical Treatment of Integrated Ca Dynamics within the Ventricular Myocyte

    Thomas R. Shannon;Thomas R. Shannon;Fei Wang;José Puglisi;Christopher Weber

  • Fractional SR Ca release is regulated by trigger Ca and SR Ca content in cardiac myocytes.

    J. W M Bassani;W. Yuan;Donald M Bers

  • Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation

    Jeffrey R. Erickson;Laetitia Pereira;Lianguo Wang;Guanghui Han

  • Drug Screening Using a Library of Human Induced Pluripotent Stem Cell–Derived Cardiomyocytes Reveals Disease-Specific Patterns of Cardiotoxicity

    Ping Liang;Feng Lan;Andrew S. Lee;Tingyu Gong

  • Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels

    Stefan Wagner;Nataliya Dybkova;Eva C.L. Rasenack;Claudius Jacobshagen

  • Cellular Basis of Abnormal Calcium Transients of Failing Human Ventricular Myocytes

    Valentino Piacentino;Christopher R. Weber;Xiongwen Chen;Jutta Weisser-Thomas

  • Local InsP3-dependent perinuclear Ca2+ signaling in cardiac myocyte excitation-transcription coupling.

    Xu Wu;Tong Zhang;Julie B C Bossuyt;Xiaodong Li

  • Transgenic CaMKIIδC Overexpression Uniquely Alters Cardiac Myocyte Ca2+ Handling. Reduced SR Ca2+ Load and Activated SR Ca2+ Release

    Lars S. Maier;Tong Zhang;Lu Chen;Jaime DeSantiago

  • A novel computational model of the human ventricular action potential and Ca transient

    Eleonora Grandi;Francesco S. Pasqualini;Donald M. Bers

  • CaMKII in myocardial hypertrophy and heart failure.

    Mark E. Anderson;Joan Heller Brown;Donald M. Bers

Frequent Co-Authors

Joan Heller Brown
Joan Heller Brown University of California, San Diego
Lothar A. Blatter
Lothar A. Blatter Rush University Medical Center
Evangelia G. Kranias
Evangelia G. Kranias University of Cincinnati Medical Center
David D. Thomas
David D. Thomas University of Minnesota
Jeffery D. Molkentin
Jeffery D. Molkentin Cincinnati Children's Hospital Medical Center
Steven R. Houser
Steven R. Houser Temple University
Kenneth B. Margulies
Kenneth B. Margulies University of Pennsylvania
Luiz Belardinelli
Luiz Belardinelli University of Florida
Kenneth D. Philipson
Kenneth D. Philipson University of California, Los Angeles
Allen M. Samarel
Allen M. Samarel Loyola University Chicago

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