D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 80 Citations 20,647 283 World Ranking 12000 National Ranking 6274

Research.com Recognitions

Awards & Achievements

2019 - Distinguished Scientist Award, American Heart Association

2017 - Member of the National Academy of Medicine (NAM)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Cancer

Mark E. Anderson focuses on Internal medicine, Ca2+/calmodulin-dependent protein kinase, Endocrinology, Cell biology and Heart failure. His Internal medicine research is multidisciplinary, incorporating elements of Reactive oxygen species and Cardiology. His study in Ca2+/calmodulin-dependent protein kinase is interdisciplinary in nature, drawing from both Endoplasmic reticulum, Signal transduction and Mitochondrion.

Mark E. Anderson has researched Endocrinology in several fields, including Ryanodine receptor 2, Ryanodine receptor, Angiotensin II and Atrial fibrillation. The concepts of his Cell biology study are interwoven with issues in Biochemistry, Cardiac action potential and Nav channel. He combines subjects such as Sudden cardiac death and Ion channel with his study of Heart failure.

His most cited work include:

  • Correction: Corrigendum: The mitochondrial uniporter controls fight or flight heart rate increases (843 citations)
  • A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation (795 citations)
  • Calmodulin kinase II inhibition protects against structural heart disease (490 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Internal medicine, Ca2+/calmodulin-dependent protein kinase, Endocrinology, Cell biology and Cardiology. As part of his studies on Internal medicine, Mark E. Anderson often connects relevant subjects like Endoplasmic reticulum. His research integrates issues of Muscle hypertrophy, Calmodulin, Signal transduction and Intracellular in his study of Ca2+/calmodulin-dependent protein kinase.

His Endocrinology research is multidisciplinary, relying on both Angiotensin II, Calcium, Phospholamban and Afterdepolarization. Mark E. Anderson focuses mostly in the field of Cell biology, narrowing it down to matters related to Biochemistry and, in some cases, Cardiac action potential. His studies in Heart failure integrate themes in fields like Heart disease, Sudden death and Sudden cardiac death.

He most often published in these fields:

  • Internal medicine (61.02%)
  • Ca2+/calmodulin-dependent protein kinase (55.12%)
  • Endocrinology (42.91%)

What were the highlights of his more recent work (between 2013-2021)?

  • Internal medicine (61.02%)
  • Ca2+/calmodulin-dependent protein kinase (55.12%)
  • Cell biology (28.74%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Internal medicine, Ca2+/calmodulin-dependent protein kinase, Cell biology, Cardiology and Endocrinology. Many of his research projects under Internal medicine are closely connected to Uniporter with Uniporter, tying the diverse disciplines of science together. His work deals with themes such as Ryanodine receptor 2, Ryanodine receptor, Vertebrate, Disease and Neuroscience, which intersect with Ca2+/calmodulin-dependent protein kinase.

He interconnects Downregulation and upregulation and Skeletal muscle in the investigation of issues within Cell biology. His Atrial fibrillation, Impella and Ischemia study in the realm of Cardiology connects with subjects such as In stent restenosis. As a part of the same scientific study, he usually deals with the Endocrinology, concentrating on Phospholamban and frequently concerns with Cardiac muscle.

Between 2013 and 2021, his most popular works were:

  • Correction: Corrigendum: The mitochondrial uniporter controls fight or flight heart rate increases (843 citations)
  • The mitochondrial uniporter controls fight or flight heart rate increases (99 citations)
  • Microtubule-Mediated Defects in Junctophilin-2 Trafficking Contribute to Myocyte Transverse-Tubule Remodeling and Ca2+ Handling Dysfunction in Heart Failure (85 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

The scientist’s investigation covers issues in Internal medicine, Mitochondrion, Endocrinology, Ca2+/calmodulin-dependent protein kinase and Myocyte. The study incorporates disciplines such as Intensive care medicine and Cardiology in addition to Internal medicine. The Mitochondrion study combines topics in areas such as Inflammation, Immunology and Calcium.

His study in the fields of Sarcolemma under the domain of Endocrinology overlaps with other disciplines such as Sinoatrial node. His Ca2+/calmodulin-dependent protein kinase study is concerned with the larger field of Cell biology. He studied Cell biology and Biochemistry that intersect with Cardiac action potential.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Correction: Corrigendum: The mitochondrial uniporter controls fight or flight heart rate increases

Yuejin Wu;Tyler P. Rasmussen;Olha M. Koval;Mei-ling A. Joiner.
Nature Communications (2015)

1556 Citations

A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation

Jeffrey R. Erickson;Mei ling A. Joiner;Xiaoqun Guan;William Kutschke.
Cell (2008)

1073 Citations

Sudden Cardiac Death Prediction and Prevention: Report From a National Heart, Lung, and Blood Institute and Heart Rhythm Society Workshop

Glenn I. Fishman;Sumeet S. Chugh;John P. Dimarco;Christine M. Albert.
Circulation (2010)

680 Citations

Calmodulin kinase II inhibition protects against structural heart disease

Rong Zhang;Michelle S.C. Khoo;Yuejin Wu;Yingbo Yang.
Nature Medicine (2005)

640 Citations

CaMKII in myocardial hypertrophy and heart failure.

Mark E. Anderson;Joan Heller Brown;Donald M. Bers.
Journal of Molecular and Cellular Cardiology (2011)

460 Citations

Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways

Jenelle M. Timmins;Lale Ozcan;Tracie A. Seimon;Gang Li.
Journal of Clinical Investigation (2009)

431 Citations

Calmodulin kinase II–mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice

Mihail G. Chelu;Mihail G. Chelu;Satyam Sarma;Subeena Sood;Sufen Wang;Sufen Wang.
Journal of Clinical Investigation (2009)

406 Citations

Cardiac Ion Channels

Dan M. Roden;Jeffrey R. Balser;Alfred L. George;Mark E. Anderson.
Annual Review of Physiology (2002)

405 Citations

Calmodulin kinase determines calcium-dependent facilitation of L-type calcium channels.

Igor Dzhura;Yuejin Wu;Roger J. Colbran;Jeffrey R. Balser.
Nature Cell Biology (2000)

403 Citations

T-tubule remodeling during transition from hypertrophy to heart failure.

Sheng Wei;Ang Guo;Biyi Chen;William J. Kutschke.
Circulation Research (2010)

389 Citations

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