2019 - Distinguished Scientist Award, American Heart Association
2017 - Member of the National Academy of Medicine (NAM)
Mark E. Anderson focuses on Internal medicine, Ca2+/calmodulin-dependent protein kinase, Endocrinology, Cell biology and Heart failure. His Internal medicine research is multidisciplinary, incorporating elements of Reactive oxygen species and Cardiology. His study in Ca2+/calmodulin-dependent protein kinase is interdisciplinary in nature, drawing from both Endoplasmic reticulum, Signal transduction and Mitochondrion.
Mark E. Anderson has researched Endocrinology in several fields, including Ryanodine receptor 2, Ryanodine receptor, Angiotensin II and Atrial fibrillation. The concepts of his Cell biology study are interwoven with issues in Biochemistry, Cardiac action potential and Nav channel. He combines subjects such as Sudden cardiac death and Ion channel with his study of Heart failure.
His primary areas of investigation include Internal medicine, Ca2+/calmodulin-dependent protein kinase, Endocrinology, Cell biology and Cardiology. As part of his studies on Internal medicine, Mark E. Anderson often connects relevant subjects like Endoplasmic reticulum. His research integrates issues of Muscle hypertrophy, Calmodulin, Signal transduction and Intracellular in his study of Ca2+/calmodulin-dependent protein kinase.
His Endocrinology research is multidisciplinary, relying on both Angiotensin II, Calcium, Phospholamban and Afterdepolarization. Mark E. Anderson focuses mostly in the field of Cell biology, narrowing it down to matters related to Biochemistry and, in some cases, Cardiac action potential. His studies in Heart failure integrate themes in fields like Heart disease, Sudden death and Sudden cardiac death.
His primary scientific interests are in Internal medicine, Ca2+/calmodulin-dependent protein kinase, Cell biology, Cardiology and Endocrinology. Many of his research projects under Internal medicine are closely connected to Uniporter with Uniporter, tying the diverse disciplines of science together. His work deals with themes such as Ryanodine receptor 2, Ryanodine receptor, Vertebrate, Disease and Neuroscience, which intersect with Ca2+/calmodulin-dependent protein kinase.
He interconnects Downregulation and upregulation and Skeletal muscle in the investigation of issues within Cell biology. His Atrial fibrillation, Impella and Ischemia study in the realm of Cardiology connects with subjects such as In stent restenosis. As a part of the same scientific study, he usually deals with the Endocrinology, concentrating on Phospholamban and frequently concerns with Cardiac muscle.
The scientist’s investigation covers issues in Internal medicine, Mitochondrion, Endocrinology, Ca2+/calmodulin-dependent protein kinase and Myocyte. The study incorporates disciplines such as Intensive care medicine and Cardiology in addition to Internal medicine. The Mitochondrion study combines topics in areas such as Inflammation, Immunology and Calcium.
His study in the fields of Sarcolemma under the domain of Endocrinology overlaps with other disciplines such as Sinoatrial node. His Ca2+/calmodulin-dependent protein kinase study is concerned with the larger field of Cell biology. He studied Cell biology and Biochemistry that intersect with Cardiac action potential.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Correction: Corrigendum: The mitochondrial uniporter controls fight or flight heart rate increases
Yuejin Wu;Tyler P. Rasmussen;Olha M. Koval;Mei-ling A. Joiner.
Nature Communications (2015)
A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation
Jeffrey R. Erickson;Mei ling A. Joiner;Xiaoqun Guan;William Kutschke.
Sudden Cardiac Death Prediction and Prevention: Report From a National Heart, Lung, and Blood Institute and Heart Rhythm Society Workshop
Glenn I. Fishman;Sumeet S. Chugh;John P. Dimarco;Christine M. Albert.
Calmodulin kinase II inhibition protects against structural heart disease
Rong Zhang;Michelle S.C. Khoo;Yuejin Wu;Yingbo Yang.
Nature Medicine (2005)
CaMKII in myocardial hypertrophy and heart failure.
Mark E. Anderson;Joan Heller Brown;Donald M. Bers.
Journal of Molecular and Cellular Cardiology (2011)
Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways
Jenelle M. Timmins;Lale Ozcan;Tracie A. Seimon;Gang Li.
Journal of Clinical Investigation (2009)
Calmodulin kinase II–mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice
Mihail G. Chelu;Mihail G. Chelu;Satyam Sarma;Subeena Sood;Sufen Wang;Sufen Wang.
Journal of Clinical Investigation (2009)
Cardiac Ion Channels
Dan M. Roden;Jeffrey R. Balser;Alfred L. George;Mark E. Anderson.
Annual Review of Physiology (2002)
Calmodulin kinase determines calcium-dependent facilitation of L-type calcium channels.
Igor Dzhura;Yuejin Wu;Roger J. Colbran;Jeffrey R. Balser.
Nature Cell Biology (2000)
T-tubule remodeling during transition from hypertrophy to heart failure.
Sheng Wei;Ang Guo;Biyi Chen;William J. Kutschke.
Circulation Research (2010)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: