D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 57 Citations 10,774 154 World Ranking 6447 National Ranking 2996

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

His main research concerns Internal medicine, Cell biology, Endocrinology, Ca2+/calmodulin-dependent protein kinase and Ankyrin. His Internal medicine research includes elements of Mitochondrion and Cardiology. His research integrates issues of Membrane protein and Spectrin in his study of Cell biology.

The various areas that Peter J. Mohler examines in his Endocrinology study include Catecholaminergic polymorphic ventricular tachycardia, Sudden cardiac death and Ion channel. His Ca2+/calmodulin-dependent protein kinase study deals with Angiotensin II intersecting with NADPH oxidase. His work in Ankyrin covers topics such as Sodium channel which are related to areas like Cardiac action potential, Biochemistry, Ventricular tachycardia and Ventricle.

His most cited work include:

  • Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death (858 citations)
  • A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation (795 citations)
  • FKBP12.6 Deficiency and Defective Calcium Release Channel (Ryanodine Receptor) Function Linked to Exercise-Induced Sudden Cardiac Death (621 citations)

What are the main themes of his work throughout his whole career to date?

Peter J. Mohler focuses on Internal medicine, Cell biology, Endocrinology, Cardiology and Ankyrin. Internal medicine is closely attributed to Ca2+/calmodulin-dependent protein kinase in his work. His Cell biology research integrates issues from Cytoskeleton, Spectrin, Biochemistry, Ion channel and Membrane protein.

Peter J. Mohler combines subjects such as Sodium-calcium exchanger, Ryanodine receptor, Sudden death and Afterdepolarization with his study of Endocrinology. His Cardiology research is multidisciplinary, incorporating elements of Adenosine and Heart rate. His work deals with themes such as Signal transducing adaptor protein, Neuroscience, Brugada syndrome and Cardiac arrhythmia, which intersect with Ankyrin.

He most often published in these fields:

  • Internal medicine (49.03%)
  • Cell biology (35.06%)
  • Endocrinology (26.30%)

What were the highlights of his more recent work (between 2017-2021)?

  • Internal medicine (49.03%)
  • Cardiology (26.95%)
  • Cell biology (35.06%)

In recent papers he was focusing on the following fields of study:

Peter J. Mohler mostly deals with Internal medicine, Cardiology, Cell biology, Heart failure and Atrial fibrillation. His biological study spans a wide range of topics, including Endocrinology, Sodium channel and In vivo. Peter J. Mohler has included themes like Wild type and Kinase in his Endocrinology study.

The study incorporates disciplines such as Titin and Sinoatrial node, Bradycardia, Sinus bradycardia, Heart rate in addition to Cardiology. His Cell biology research includes themes of Cytoskeleton, Spectrin, microRNA, Ion channel and Gene isoform. His study on Cardiac function curve is often connected to Kinetics as part of broader study in Heart failure.

Between 2017 and 2021, his most popular works were:

  • Cardiovascular risk of electronic cigarettes: a review of preclinical and clinical studies. (28 citations)
  • Human Atrial Fibrillation Drivers Resolved With Integrated Functional and Structural Imaging to Benefit Clinical Mapping (24 citations)
  • βII-spectrin promotes mouse brain connectivity through stabilizing axonal plasma membranes and enabling axonal organelle transport (23 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His scientific interests lie mostly in Cell biology, Internal medicine, Spectrin, Heart failure and Ca2+/calmodulin-dependent protein kinase. His Cell biology research is multidisciplinary, incorporating perspectives in Oxidative stress, Ankyrin and In vivo. His Internal medicine study combines topics in areas such as Endocrinology and Cardiology.

In Spectrin, Peter J. Mohler works on issues like Axon, which are connected to NODAL, Ion channel, Action potential and Organelle. His Heart failure study integrates concerns from other disciplines, such as Fibrosis, Blockade, Sodium channel and Electrical conduction system of the heart. His work carried out in the field of Ca2+/calmodulin-dependent protein kinase brings together such families of science as Myocyte, Atrial fibrillation and Action potential duration.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death

Peter J. Mohler;Jean-Jacques Schott;Anthony O. Gramolini;Keith W. Dilly.
Nature (2003)

1124 Citations

A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation

Jeffrey R. Erickson;Mei ling A. Joiner;Xiaoqun Guan;William Kutschke.
Cell (2008)

976 Citations

FKBP12.6 Deficiency and Defective Calcium Release Channel (Ryanodine Receptor) Function Linked to Exercise-Induced Sudden Cardiac Death

Xander H T Wehrens;Stephan E. Lehnart;Fannie Huang;John A. Vest.
Cell (2003)

799 Citations

Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes

Peter J. Mohler;Ilaria Rivolta;Carlo Napolitano;Guy LeMaillet.
Proceedings of the National Academy of Sciences of the United States of America (2004)

412 Citations

A cardiac arrhythmia syndrome caused by loss of ankyrin-B function

Peter J. Mohler;Igor Splawski;Carlo Napolitano;Georgia Bottelli.
Proceedings of the National Academy of Sciences of the United States of America (2004)

381 Citations

Inherited Arrhythmias A National Heart, Lung, and Blood Institute and Office of Rare Diseases Workshop Consensus Report About the Diagnosis, Phenotyping, Molecular Mechanisms, and Therapeutic Approaches for Primary Cardiomyopathies of Gene Mutations Affecting Ion Channel Function

Stephan E. Lehnart;Michael J. Ackerman;D. Woodrow Benson;Ramon Brugada.
Circulation (2007)

343 Citations

CaMKII determines mitochondrial stress responses in heart

Mei Ling A. Joiner;Olha M. Koval;Jingdong Li;Jingdong Li;B. Julie He.
Nature (2012)

326 Citations

Atrial fibrillation driven by micro-anatomic intramural re-entry revealed by simultaneous sub-epicardial and sub-endocardial optical mapping in explanted human hearts.

Brian J. Hansen;Jichao Zhao;Thomas A. Csepe;Brandon T. Moore.
European Heart Journal (2015)

293 Citations

A βIV-spectrin/CaMKII signaling complex is essential for membrane excitability in mice

Thomas J. Hund;Olha M. Koval;Jingdong Li;Patrick J. Wright.
Journal of Clinical Investigation (2010)

247 Citations

RETRACTED: L-Type Ca2+ Channel Facilitation Mediated by Phosphorylation of the β Subunit by CaMKII

Chad E. Grueter;Sunday A. Abiria;Igor Dzhura;Yuejin Wu.
Molecular Cell (2006)

234 Citations

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