Allen M. Samarel mostly deals with Cell biology, Internal medicine, Endocrinology, Focal adhesion and Myocyte. Internal medicine is closely attributed to Cardiology in his research. Allen M. Samarel combines subjects such as Genetically modified mouse, Protein kinase C and Cardiomyopathy with his study of Endocrinology.
His Focal adhesion research is multidisciplinary, relying on both Integrin and Cell adhesion. As a member of one scientific family, Allen M. Samarel mostly works in the field of Myocyte, focusing on Cardiac muscle and, on occasion, CapZ, Actin, Contractility and Diastole. Allen M. Samarel usually deals with Muscle hypertrophy and limits it to topics linked to Cardiac myocyte and Endothelin 1.
Allen M. Samarel focuses on Internal medicine, Cell biology, Endocrinology, Myocyte and Focal adhesion. His Internal medicine study combines topics from a wide range of disciplines, such as Intracellular and Cardiology. His study in Cell biology is interdisciplinary in nature, drawing from both Integrin and Biochemistry.
His Endocrinology research includes themes of Protein kinase C, Downregulation and upregulation and Myosin. His research on Myocyte also deals with topics like
His primary areas of study are Cell biology, Internal medicine, Focal adhesion, Endocrinology and Phosphorylation. In his research on the topic of Cell biology, Endothelial stem cell and Endothelial dysfunction is strongly related with Molecular biology. His Internal medicine study frequently links to related topics such as Cardiology.
His Focal adhesion research is multidisciplinary, relying on both PTK2, Protein kinase B, Integrin and Neuroscience. In general Endocrinology study, his work on Stimulation and Myocyte often relates to the realm of Locus coeruleus, thereby connecting several areas of interest. His Phosphorylation study incorporates themes from Troponin and Kinase.
Cell biology, Heart failure, Signal transduction, Internal medicine and Endocrinology are his primary areas of study. His Cell biology study combines topics in areas such as Epigenetics and DNA methylation. His Heart failure research incorporates themes from Depression and Risk factor.
His Signal transduction research is mostly focused on the topic Focal adhesion. Allen M. Samarel studied Internal medicine and Connective tissue that intersect with Transplantation, Extracellular matrix and Matrix metalloproteinase. His Sarcomere study which covers CapZ that intersects with Myocyte.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Costameres, focal adhesions, and cardiomyocyte mechanotransduction.
Allen M. Samarel.
American Journal of Physiology-heart and Circulatory Physiology (2005)
Hydrogen Peroxide Activates Mitogen-Activated Protein Kinases and Na+-H+ Exchange in Neonatal Rat Cardiac Myocytes
Abdelkarim Sabri;Kenneth L. Byron;Allen M. Samarel;Jeremy Bell.
Circulation Research (1998)
Suppression of RhoA activity by focal adhesion kinase-induced activation of p190RhoGAP: role in regulation of endothelial permeability.
Michael Holinstat;Nebojsa Knezevic;Michael Broman;Allen M. Samarel.
Journal of Biological Chemistry (2006)
Vascular Endothelial Growth Factor Regulates Focal Adhesion Assembly in Human Brain Microvascular Endothelial Cells through Activation of the Focal Adhesion Kinase and Related Adhesion Focal Tyrosine Kinase
Hava Karsenty Avraham;Tae-Hee Lee;Youngho Koh;Tae-Aug Kim.
Journal of Biological Chemistry (2003)
Differential Activation of Mitogen-Activated Protein Kinase Cascades and Apoptosis by Protein Kinase C ε and δ in Neonatal Rat Ventricular Myocytes
Maria C. Heidkamp;Allison L. Bayer;Jody L. Martin;Allen M. Samarel.
Circulation Research (2001)
Calcium- and Protein Kinase C–Dependent Activation of the Tyrosine Kinase PYK2 by Angiotensin II in Vascular Smooth Muscle
Abdelkarim Sabri;Geetha Govindarajan;Tina M. Griffin;Kenneth L. Byron.
Circulation Research (1998)
An anticancer C-Kit kinase inhibitor is reengineered to make it more active and less cardiotoxic
Ariel Fernández;Angela Sanguino;Zhenghong Peng;Eylem Ozturk;Eylem Ozturk.
Journal of Clinical Investigation (2007)
Fibronectin fragment activation of proline-rich tyrosine kinase PYK2 mediates integrin signals regulating collagenase-3 expression by human chondrocytes through a protein kinase C-dependent pathway.
Richard Frank Loeser;Christopher B. Forsyth;Allen M. Samarel;Hee Jeong Im.
Journal of Biological Chemistry (2003)
Mechanical forces regulate focal adhesion and costamere assembly in cardiac myocytes
Will W. Sharp;David G. Simpson;Thomas K. Borg;Allen M. Samarel.
American Journal of Physiology-heart and Circulatory Physiology (1997)
Cardiac Dysfunction Occurs in the HIV-1 Transgenic Mouse Treated with Zidovudine
William Lewis;Ingrid L. Grupp;Günter Grupp;Brian D Hoit.
Laboratory Investigation (2000)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of California, Davis
New York University
Medical University of South Carolina
Imperial College London
Imperial College London
Virginia Commonwealth University
University of Pennsylvania
University of Sheffield
Rush University Medical Center
National Institutes of Health
University of Wisconsin–Madison
Texas A&M University
Nanyang Technological University
University of Valencia
University of North Carolina at Chapel Hill
University of Tokyo
Georgetown University
Vrije Universiteit Brussel
Harvard University
University of Cologne
Paul Scherrer Institute
Max Planck Institute for Biogeochemistry
Natural Environment Research Council
University of North Carolina at Chapel Hill
University of Cambridge
University of Bologna