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Medicine

D-Index
82
Citations
28588
World Ranking
15975
National Ranking
8037

Overview

Thomas Force is affiliated with Vanderbilt University in the United States. Their recent publications are primarily featured in the journal ESC Heart Failure. The available records include several papers titled "Issue Information," published between 2020 and 2021 in ESC Heart Failure.

  • Issue Information, 2021, ESC Heart Failure
  • Issue Information, 2020, ESC Heart Failure
  • Issue Information, 2020, ESC Heart Failure
  • Issue Information, 2020, ESC Heart Failure
  • Issue Information, 2020, ESC Heart Failure

The frequent co-authors collaborating with Thomas Force include the following researchers:

  • Daniela Forcella
  • Abdelhamid Magdy
  • Philip Cairo
  • John Glasgow
  • Arno W. Hoes

All recent works are disseminated via the ESC Heart Failure venue, which appears as the prominent platform for this scientist's publications.

  • ESC Heart Failure

Best Publications

  • Expert Consensus for Multimodality Imaging Evaluation of Adult Patients during and after Cancer Therapy: A Report from the American Society of Echocardiography and the European Association of Cardiovascular Imaging.

    Juan Carlos Plana;Maurizio Galderisi;Ana Barac;Michael S. Ewer

  • Cardiotoxicity of the cancer therapeutic agent imatinib mesylate

    Risto Kerkelä;Luanda Grazette;Rinat Yacobi;Cezar Iliescu

  • Heart failure: preventing disease and death worldwide.

    Piotr P. Ponikowski;Stefan D. Anker;Khalid F. AlHabib;Martin R. Cowie

  • Cardiotoxicity associated with tyrosine kinase inhibitor sunitinib

    Tammy F Chu;Tammy F Chu;Maria A Rupnick;Maria A Rupnick;Risto Kerkela;Susan M Dallabrida

  • Expression cloning of a common receptor for parathyroid hormone and parathyroid hormone-related peptide from rat osteoblast-like cells: a single receptor stimulates intracellular accumulation of both cAMP and inositol trisphosphates and increases intracellular free calcium

    A B Abou-Samra;H Jüppner;T Force;M W Freeman

  • Molecular mechanisms of cardiotoxicity of tyrosine kinase inhibition.

    Thomas Force;Daniela S. Krause;Richard A. Van Etten

  • Akt Activation Preserves Cardiac Function and Prevents Injury After Transient Cardiac Ischemia In Vivo

    Takashi Matsui;Jingzang Tao;Federica del Monte;Kyung-Han Lee

  • Protein kinase cascades in the regulation of cardiac hypertrophy

    Gerald W. Dorn;Thomas Force

  • Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.

    Beth A. Rose;Thomas Force;Yibin Wang

  • Glycogen Synthase Kinase-3β Is a Negative Regulator of Cardiomyocyte Hypertrophy

    Syed Haq;Gabriel Choukroun;Zhao Bin Kang;Hardeep Ranu

  • Differential Activation of Signal Transduction Pathways in Human Hearts With Hypertrophy Versus Advanced Heart Failure

    Syed Haq;Gabriel Choukroun;Hae Lim;Kevin M. Tymitz

  • Cardiovascular side effects of cancer therapies: a position statement from the Heart Failure Association of the European Society of Cardiology.

    Thomas Eschenhagen;Thomas Force;Michael S. Ewer;Gilles W. De Keulenaer

  • Growth Factors and Mitogen-Activated Protein Kinases

    Thomas Force;Joseph V. Bonventre

  • 17β-Estradiol Reduces Cardiomyocyte Apoptosis In Vivo and In Vitro via Activation of Phospho-Inositide-3 Kinase/Akt Signaling

    Richard D. Patten;Isaac Pourati;Mark J. Aronovitz;Jason Baur

  • Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

    John W. Elrod;Renee Wong;Shikha Mishra;Ronald J. Vagnozzi

  • Mechanisms of Cardiac Dysfunction Associated With Tyrosine Kinase Inhibitor Cancer Therapeutics

    Ming Hui Chen;Risto Kerkelä;Thomas Force

  • Cardiotoxicity of kinase inhibitors: the prediction and translation of preclinical models to clinical outcomes.

    Thomas Force;Kyle L. Kolaja

  • The stress-activated protein kinases are major c-Jun amino-terminal kinases activated by ischemia and reperfusion.

    C M Pombo;J V Bonventre;J Avruch;J R Woodgett

  • Calcineurin Promotes Protein Kinase C and c-Jun NH2-terminal Kinase Activation in the Heart CROSS-TALK BETWEEN CARDIAC HYPERTROPHIC SIGNALING PATHWAYS

    Leon J. De Windt;Hae W. Lim;Syed Haq;Thomas Force

  • Inhibition of ErbB2 causes mitochondrial dysfunction in cardiomyocytes: Implications for herceptin-induced cardiomyopathy

    Luanda P. Grazette;Wolfgang Boecker;Takashi Matsui;Marc Semigran

Frequent Co-Authors

Joseph V. Bonventre
Joseph V. Bonventre Brigham and Women's Hospital
John M. Kyriakis
John M. Kyriakis Tufts University
James R. Woodgett
James R. Woodgett Lunenfeld-Tanenbaum Research Institute
Erhe Gao
Erhe Gao Temple University
Anthony Rosenzweig
Anthony Rosenzweig Harvard University
Jeffery D. Molkentin
Jeffery D. Molkentin Cincinnati Children's Hospital Medical Center
Roger J. Hajjar
Roger J. Hajjar Icahn School of Medicine at Mount Sinai
Ming-Hui Chen
Ming-Hui Chen University of Connecticut
Ronglih Liao
Ronglih Liao Stanford University
Joseph Avruch
Joseph Avruch Harvard University

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