D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 88 Citations 34,916 277 World Ranking 6470 National Ranking 3574

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Mutation

Harald Jüppner mostly deals with Internal medicine, Endocrinology, Parathyroid hormone, Receptor and Fibroblast growth factor 23. His studies deal with areas such as Cancer research and Peptide as well as Internal medicine. His study in Hypophosphatemia, Autosomal dominant hypophosphatemic rickets, Hyperparathyroidism, Kidney and PHEX falls within the category of Endocrinology.

His biological study spans a wide range of topics, including Calcitriol, Calcium metabolism and Mutation. His work on Peptide hormone as part of general Receptor study is frequently linked to Parathyroid hormone receptor, bridging the gap between disciplines. His work carried out in the field of Fibroblast growth factor 23 brings together such families of science as Osteomalacia, Osteoid and Phosphate.

His most cited work include:

  • LDL Receptor-Related Protein 5 (LRP5) Affects Bone Accrual and Eye Development (1926 citations)
  • Primer on the metabolic bone diseases and disorders of mineral metabolism (1854 citations)
  • Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis. (1331 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Internal medicine, Endocrinology, Parathyroid hormone, Receptor and Parathyroid hormone receptor. His study in Fibroblast growth factor 23, Pseudohypoparathyroidism, Kidney disease, Kidney and Calcium are all subfields of Internal medicine. The concepts of his Fibroblast growth factor 23 study are interwoven with issues in Vitamin D and neurology, Osteomalacia, Hypophosphatemia and Phosphate.

His Endocrinology research is multidisciplinary, incorporating elements of Calcium metabolism, Secondary hyperparathyroidism and GNAS complex locus. His Parathyroid hormone study incorporates themes from Calcitriol, Renal osteodystrophy, Bone remodeling and Peptide sequence. His research integrates issues of Molecular biology, Signal transduction and Peptide in his study of Receptor.

He most often published in these fields:

  • Internal medicine (75.32%)
  • Endocrinology (73.25%)
  • Parathyroid hormone (45.45%)

What were the highlights of his more recent work (between 2012-2021)?

  • Internal medicine (75.32%)
  • Endocrinology (73.25%)
  • Pseudohypoparathyroidism (14.81%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Internal medicine, Endocrinology, Pseudohypoparathyroidism, GNAS complex locus and Fibroblast growth factor 23. Parathyroid hormone, Kidney disease, Kidney, Vitamin D and neurology and Nephrology are subfields of Internal medicine in which his conducts study. His work is dedicated to discovering how Parathyroid hormone, Receptor are connected with In vivo and other disciplines.

His research in the fields of Nephrocalcinosis overlaps with other disciplines such as Context. Harald Jüppner interconnects Pseudopseudohypoparathyroidism, Epigenetics, Allele and Exon in the investigation of issues within GNAS complex locus. His Fibroblast growth factor 23 study also includes fields such as

  • Acute kidney injury that intertwine with fields like Prospective cohort study,
  • Hypophosphatemia which connect with Osteomalacia.

Between 2012 and 2021, his most popular works were:

  • Primer on the metabolic bone diseases and disorders of mineral metabolism (1854 citations)
  • Plasma FGF23 levels increase rapidly after acute kidney injury (112 citations)
  • Epidemiology and Diagnosis of Hypoparathyroidism. (100 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Mutation

Harald Jüppner mainly investigates Internal medicine, Endocrinology, Fibroblast growth factor 23, Kidney disease and Vitamin D and neurology. His study in Internal medicine is interdisciplinary in nature, drawing from both Osteocyte, MEDLINE and Cardiology. His work deals with themes such as Parathyroid hormone and Pseudohypoparathyroidism, which intersect with Endocrinology.

Harald Jüppner works mostly in the field of Parathyroid hormone, limiting it down to topics relating to Receptor and, in certain cases, Homeostasis. His Fibroblast growth factor 23 research is multidisciplinary, incorporating perspectives in Hypophosphatemia, Osteomalacia, Klotho and Effector, Cell biology. He interconnects Interquartile range, Renal function, Secondary hyperparathyroidism, Confidence interval and Risk factor in the investigation of issues within Kidney disease.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Primer on the metabolic bone diseases and disorders of mineral metabolism

John P. Bilezikian;Roger Bouillon;Thomas Clemens;Juliet Compston.
Rhode Island dental journal (2013)

3255 Citations

LDL Receptor-Related Protein 5 (LRP5) Affects Bone Accrual and Eye Development

Y. Q. Gong;R. B. Slee;N. Fukai;G. Rawadi.
Cell (2001)

2336 Citations

A G protein-linked receptor for parathyroid hormone and parathyroid hormone-related peptide

Harald Jüppner;Abdul Badi Abou-Samra;Mason Freeman;Xiang F. Kong.
Science (1991)

1797 Citations

A novel cyclin encoded by a bcl1 -linked candidate oncogene

Toru Motokura;Theodora Bloom;Hyung Goo Kim;Harald Jüppner..
Nature (1991)

1787 Citations

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis.

Orlando M. Gutiérrez;Michael Mannstadt;Tamara Isakova;Jose Alejandro Rauh-Hain.
The New England Journal of Medicine (2008)

1773 Citations

PTH/PTHrP Receptor in Early Development and Indian Hedgehog--Regulated Bone Growth

Beate Lanske;Andrew C. Karaplis;Kaechong Lee;Arne Luz.
Science (1996)

1377 Citations

Fibroblast Growth Factor-23 Mitigates Hyperphosphatemia but Accentuates Calcitriol Deficiency in Chronic Kidney Disease

Orlando Gutierrez;Tamara Isakova;Eugene Rhee;Anand Shah.
Journal of The American Society of Nephrology (2005)

993 Citations

Expression cloning of a common receptor for parathyroid hormone and parathyroid hormone-related peptide from rat osteoblast-like cells: a single receptor stimulates intracellular accumulation of both cAMP and inositol trisphosphates and increases intracellular free calcium

A B Abou-Samra;H Jüppner;T Force;M W Freeman.
Proceedings of the National Academy of Sciences of the United States of America (1992)

968 Citations

Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers.

Tobias Larsson;U.L.F. Nisbeth;Östen Ljunggren;Harald Jüppner.
Kidney International (2003)

905 Citations

Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemia.

Kenneth B Jonsson;Richard Zahradnik;Tobias Larsson;Kenneth E White.
The New England Journal of Medicine (2003)

846 Citations

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