Member of the Association of American Physicians
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Osteoclast, Bone resorption and Bone remodeling. He has researched Internal medicine in several fields, including Macrophage colony-stimulating factor and Cell culture. His studies in Endocrinology integrate themes in fields like Stromal cell, Tartrate-resistant acid phosphatase, Osteoblast, Parathyroid hormone and Bone marrow.
His Osteoclast research is multidisciplinary, incorporating elements of Arthritis, Osteoprotegerin, RANKL and Cell biology. His Bone resorption research is multidisciplinary, relying on both Tamoxifen, Estrogen receptor beta, Estrogen receptor, Estrogen receptor alpha and Resorption. His Bone remodeling research is multidisciplinary, incorporating perspectives in Bone cell and Rheumatoid arthritis.
T. John Martin mainly investigates Internal medicine, Endocrinology, Osteoclast, Cell biology and Osteoblast. His study looks at the intersection of Internal medicine and topics like Cell culture with Messenger RNA and Molecular biology. His Endocrinology study combines topics from a wide range of disciplines, such as Receptor, Paracrine signalling, Parathyroid hormone and Bone marrow.
His studies deal with areas such as Cartilage, Bone resorption and RANKL as well as Osteoclast. His work carried out in the field of Cell biology brings together such families of science as Cellular differentiation, Bone remodeling, Mineralization and Leukemia inhibitory factor. T. John Martin has included themes like Mesenchymal stem cell, Glycoprotein 130 and Osteoid in his Osteoblast study.
His primary scientific interests are in Cell biology, Osteoblast, Endocrinology, Internal medicine and Parathyroid hormone. His Cell biology study integrates concerns from other disciplines, such as Mineralization, Osteocyte, Osteoid, Leukemia inhibitory factor and Resorption. As part of one scientific family, he deals mainly with the area of Resorption, narrowing it down to issues related to the Process, and often Bone resorption.
His Osteoblast study incorporates themes from Osteoclast, Signal transduction and Bone remodeling. His Endocrinology research integrates issues from Tibia and Bone morphogenetic protein. His Parathyroid hormone research includes elements of Paracrine signalling and Parathyroid hormone receptor.
T. John Martin mainly focuses on Cell biology, Osteoblast, Osteocyte, Cortical bone and Parathyroid hormone. His research integrates issues of Bone remodeling, Resorption and Leukemia inhibitory factor in his study of Cell biology. As a member of one scientific family, T. John Martin mostly works in the field of Osteocyte, focusing on Osteoclast and, on occasion, Endocrinology.
His study focuses on the intersection of Cortical bone and fields such as Cancellous bone with connections in the field of Bone marrow, Endochondral ossification, Retinoic acid receptor and Lymphopoiesis. His Parathyroid hormone research incorporates themes from Cancer research and Intracrine, Autocrine signalling. His Autocrine signalling research incorporates elements of Paracrine signalling, Bone resorption, Breast cancer, Bone metastasis and Forskolin.
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Therapeutic approaches to bone diseases.
Gideon A. Rodan;T. John Martin.
IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis
Shigeru Kotake;Nobuyuki Udagawa;Naoyuki Takahashi;Kenichiro Matsuzaki.
Journal of Clinical Investigation (1999)
Tumor Necrosis Factor α Stimulates Osteoclast Differentiation by a Mechanism Independent of the Odf/Rankl–Rank Interaction
Kanichiro Kobayashi;Naoyuki Takahashi;Eijiro Jimi;Nobuyuki Udagawa.
Journal of Experimental Medicine (2000)
Estrogen Prevents Bone Loss via Estrogen Receptor α and Induction of Fas Ligand in Osteoclasts
Takashi Nakamura;Yuuki Imai;Yuuki Imai;Takahiro Matsumoto;Shingo Sato.
Osteoclast-derived activity in the coupling of bone formation to resorption
T. John Martin;Natalie A. Sims.
Trends in Molecular Medicine (2005)
Control of mammary stem cell function by steroid hormone signalling
Marie Liesse Asselin-Labat;François Vaillant;Julie M. Sheridan;Bhupinder Pal.
Coupling the activities of bone formation and resorption: a multitude of signals within the basic multicellular unit
Natalie A Sims;T John Martin.
bonekey Reports (2014)
The bone marrow-derived stromal cell lines MC3T3-G2/PA6 and ST2 support osteoclast-like cell differentiation in cocultures with mouse spleen cells.
Nobuyuki Udagawa;Naoyuki Takahashi;Takuhiko Akatsu;Takahisa Sasaki.
A combination of osteoclast differentiation factor and macrophage-colony stimulating factor is sufficient for both human and mouse osteoclast formation in vitro.
Julian M. W. Quinn;Jan Elliott;Matthew T. Gillespie;T. John Martin.
Osteoprotegerin produced by osteoblasts is an important regulator in osteoclast development and function.
Nobuyuki Udagawa;Naoyuki Takahashi;Hisataka Yasuda;Atsuko Mizuno.
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