Osteoclast, Bone remodeling, Internal medicine, Endocrinology and Bone resorption are her primary areas of study. The various areas that Natalie A. Sims examines in her Osteoclast study include Osteoprotegerin, Bone cell, Arthritis, RANKL and Osteoblast. Her Osteoblast research includes elements of Leukemia inhibitory factor receptor, Oncostatin M, Cell biology, Sclerostin and Bone marrow.
Her research in Bone remodeling intersects with topics in Cortical bone, Pathology, Bone growth and Cannabinoid receptor. The study incorporates disciplines such as Cannabinoid, O-1602, Protein tyrosine phosphatase and c-Fos in addition to Endocrinology. Her Bone resorption study integrates concerns from other disciplines, such as Osteopetrosis, Osteocyte, Proto-oncogene tyrosine-protein kinase Src, Estrogen and Resorption.
Her primary areas of study are Internal medicine, Endocrinology, Osteoblast, Cell biology and Osteoclast. The study of Internal medicine is intertwined with the study of Sclerostin in a number of ways. Her studies deal with areas such as Cortical bone, Receptor and Parathyroid hormone as well as Endocrinology.
Her Osteoblast study incorporates themes from Immunology, Bone marrow, Osteoid and Paracrine signalling. Her Cell biology research incorporates themes from Cellular differentiation and Leukemia inhibitory factor. Her Osteoclast research also works with subjects such as
Natalie A. Sims mainly focuses on Cell biology, Osteoblast, Osteocyte, Osteoclast and Cortical bone. Natalie A. Sims has included themes like Bone marrow, Osteoid, Mineralization and Leukemia inhibitory factor in her Cell biology study. Her research in Osteoblast intersects with topics in Paracrine signalling, Phenotype, Matrix, Bone growth and Bone remodeling.
Her Osteoclast research is multidisciplinary, incorporating elements of Bone mass, Cell type, Bone resorption and RANKL. Her studies in RANKL integrate themes in fields like Osteoprotegerin and Mesenchymal stem cell. Her Receptor research is multidisciplinary, relying on both Calcium metabolism, Endocrinology and Parathyroid hormone-related protein, Parathyroid hormone.
Her primary scientific interests are in Cell biology, Osteoblast, Bone growth, Osteocyte and SOCS3. Her Cell biology study frequently draws connections to other fields, such as Bone marrow. Her study in Osteoblast is interdisciplinary in nature, drawing from both Autophagy, Type I collagen, Endocrinology, Bone remodeling and Osteogenesis imperfecta.
Her research integrates issues of Cortical bone, Calcification and Mineralization in her study of Osteocyte. Her biological study spans a wide range of topics, including Osteoclast, Cytokine and Leukemia inhibitory factor. Her Osteoclast study combines topics from a wide range of disciplines, such as Physiology, Paracrine signalling and RANKL.
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Bone marrow macrophages maintain hematopoietic stem cell (HSC) niches and their depletion mobilizes HSCs
Ingrid G. Winkler;Natalie A. Sims;Allison R. Pettit;Valérie Barbier.
Osteoclast-derived activity in the coupling of bone formation to resorption
T. John Martin;Natalie A. Sims.
Trends in Molecular Medicine (2005)
Coupling the activities of bone formation and resorption: a multitude of signals within the basic multicellular unit
Natalie A Sims;T John Martin.
bonekey Reports (2014)
Bone remodeling: Multiple cellular interactions required for coupling of bone formation and resorption.
Natalie A. Sims;Jonathan H. Gooi.
Seminars in Cell & Developmental Biology (2008)
Rb Regulates Interactions between Hematopoietic Stem Cells and Their Bone Marrow Microenvironment
Carl R. Walkley;Jeremy M. Shea;Natalie A. Sims;Louise E. Purton.
Overexpression of DeltaFosB transcription factor(s) increases bone formation and inhibits adipogenesis.
G Sabatakos;N A Sims;J Chen;K Aoki.
Nature Medicine (2000)
Deletion of estrogen receptors reveals a regulatory role for estrogen receptors-β in bone remodeling in females but not in males
N.A Sims;S Dupont;A Krust;P Clement-Lacroix.
Activated parathyroid hormone/parathyroid hormone–related protein receptor in osteoblastic cells differentially affects cortical and trabecular bone
L.M. Calvi;N.A. Sims;J.L. Hunzelman;M.C. Knight.
Journal of Clinical Investigation (2001)
Wnt inhibitory factor 1 is epigenetically silenced in human osteosarcoma, and targeted disruption accelerates osteosarcomagenesis in mice
Maya Kansara;Michael Tsang;Laurent Kodjabachian;Natalie A. Sims.
Journal of Clinical Investigation (2009)
Model structure and control of bone remodeling: a theoretical study.
Peter Pivonka;Jan Zimak;David W. Smith;Bruce S. Gardiner.
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