D-Index & Metrics Best Publications
Masafumi Fukagawa

Masafumi Fukagawa

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 70 Citations 17,378 469 World Ranking 15711 National Ranking 480

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Endocrinology
  • Gene

Masafumi Fukagawa mostly deals with Internal medicine, Endocrinology, Parathyroid hormone, Secondary hyperparathyroidism and Hyperparathyroidism. Internal medicine is frequently linked to Urology in his study. The various areas that Masafumi Fukagawa examines in his Endocrinology study include Osteoblast and Fibroblast growth factor 23.

His research in Parathyroid hormone intersects with topics in Downregulation and upregulation, Protein kinase A, Uremia and Hazard ratio. Masafumi Fukagawa interconnects Calcitriol, Parathyroid hormone secretion, Parathyroid gland and Calcium-sensing receptor in the investigation of issues within Secondary hyperparathyroidism. His Hyperparathyroidism research integrates issues from Parathyroidectomy, Cinacalcet, Hyperplasia and Calcification.

His most cited work include:

  • Decreased 1,25-dihydroxyvitamin D3 receptor density is associated with a more severe form of parathyroid hyperplasia in chronic uremic patients. (517 citations)
  • Executive summary of the 2017 KDIGO Chronic Kidney Disease–Mineral and Bone Disorder (CKD-MBD) Guideline Update: what’s changed and why it matters (364 citations)
  • Possible involvement of circulating fibroblast growth factor 23 in the development of secondary hyperparathyroidism associated with renal insufficiency. (281 citations)

What are the main themes of his work throughout his whole career to date?

Masafumi Fukagawa mostly deals with Internal medicine, Endocrinology, Secondary hyperparathyroidism, Parathyroid hormone and Kidney disease. His research brings together the fields of Gastroenterology and Internal medicine. His Endocrinology research includes themes of Calcium and Fibroblast growth factor 23.

His studies in Secondary hyperparathyroidism integrate themes in fields like Parathyroidectomy, Parathyroid gland and Urology. His work carried out in the field of Parathyroid hormone brings together such families of science as Calcium metabolism, Primary hyperparathyroidism and Uremia. His research investigates the connection between Kidney disease and topics such as Renal function that intersect with problems in Creatinine.

He most often published in these fields:

  • Internal medicine (71.97%)
  • Endocrinology (50.24%)
  • Secondary hyperparathyroidism (29.29%)

What were the highlights of his more recent work (between 2016-2021)?

  • Internal medicine (71.97%)
  • Hemodialysis (21.73%)
  • Endocrinology (50.24%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Internal medicine, Hemodialysis, Endocrinology, Kidney disease and Secondary hyperparathyroidism. His research on Internal medicine often connects related topics like Gastroenterology. His Hemodialysis research incorporates themes from Calcium, Urology, Mortality rate, Prospective cohort study and Practice patterns.

His study on Endocrinology is mostly dedicated to connecting different topics, such as Fibroblast growth factor 23. His Kidney disease research is multidisciplinary, incorporating perspectives in Bone disease, Osteoporosis, Bone remodeling and Renal function. His Secondary hyperparathyroidism study is concerned with the larger field of Parathyroid hormone.

Between 2016 and 2021, his most popular works were:

  • Executive summary of the 2017 KDIGO Chronic Kidney Disease–Mineral and Bone Disorder (CKD-MBD) Guideline Update: what’s changed and why it matters (364 citations)
  • Diagnosis, Evaluation, Prevention, and Treatment of Chronic Kidney Disease–Mineral and Bone Disorder: Synopsis of the Kidney Disease: Improving Global Outcomes 2017 Clinical Practice Guideline Update (113 citations)
  • Potential therapeutic interventions for chronic kidney disease-associated sarcopenia via indoxyl sulfate-induced mitochondrial dysfunction. (53 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Cancer
  • Gene

Masafumi Fukagawa spends much of his time researching Internal medicine, Secondary hyperparathyroidism, Kidney disease, Hemodialysis and Parathyroid hormone. He has included themes like Gastroenterology and Endocrinology in his Internal medicine study. His work focuses on many connections between Endocrinology and other disciplines, such as Fibroblast growth factor 23, that overlap with his field of interest in Anemia and Erythropoietin.

The study incorporates disciplines such as Hyperparathyroidism, Pharmacodynamics and Urology in addition to Secondary hyperparathyroidism. The Kidney disease study combines topics in areas such as Randomized controlled trial, Osteoporosis, Pathology and Bone remodeling. The Hemodialysis study which covers Placebo that intersects with Calcium metabolism and Interquartile range.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Decreased 1,25-dihydroxyvitamin D3 receptor density is associated with a more severe form of parathyroid hyperplasia in chronic uremic patients.

N Fukuda;H Tanaka;Y Tominaga;M Fukagawa.
Journal of Clinical Investigation (1993)

804 Citations

Executive summary of the 2017 KDIGO Chronic Kidney Disease–Mineral and Bone Disorder (CKD-MBD) Guideline Update: what’s changed and why it matters

Markus Ketteler;Geoffrey A. Block;Pieter Evenepoel;Masafumi Fukagawa.
Kidney International (2017)

522 Citations

Possible involvement of circulating fibroblast growth factor 23 in the development of secondary hyperparathyroidism associated with renal insufficiency.

Takashi Shigematsu;Junichiro James Kazama;Takeyoshi Yamashita;Seiji Fukumoto.
American Journal of Kidney Diseases (2004)

432 Citations

Depressed expression of Klotho and FGF receptor 1 in hyperplastic parathyroid glands from uremic patients

Hirotaka Komaba;Shunsuke Goto;Hideki Fujii;Yasuhiro Hamada.
Kidney International (2010)

291 Citations

p-Cresyl sulfate causes renal tubular cell damage by inducing oxidative stress by activation of NADPH oxidase

Hiroshi Watanabe;Yohei Miyamoto;Daisuke Honda;Hisae Tanaka.
Kidney International (2013)

282 Citations

Serum fibroblast growth factor-23 levels predict the future refractory hyperparathyroidism in dialysis patients

Shohei Nakanishi;Junichiro James Kazama;Tomoko Nii-Kono;Kentaro Omori.
Kidney International (2005)

276 Citations

Clinical Practice Guideline for the Management of Chronic Kidney Disease‐Mineral and Bone Disorder

Masafumi Fukagawa;Keitaro Yokoyama;Fumihiko Koiwa;Masatomo Taniguchi.
Therapeutic Apheresis and Dialysis (2013)

271 Citations

Serial evaluation of parathyroid size by ultrasonography is another useful marker for the long-term prognosis of calcitriol pulse therapy in chronic dialysis patients.

Masafumi Fukagawa;Masafumi Kitaoka;Hung Yi;Naoko Fukuda.
Nephron (1994)

236 Citations

Cloning and characterization of developmental endothelial locus-1: An embryonic endothelial cell protein that binds the αvβ3 integrin receptor

Chiaki Hidai;Thomas Zupancic;Kalyani Penta;Kalyani Penta;Adel Mikhail.
Genes & Development (1998)

234 Citations

Diagnosis, assessment, and treatment of bone turnover abnormalities in renal osteodystrophy.

Kevin J Martin;Klaus Olgaard;Jack W Coburn;Jack W Coburn;Giorgio M Coen;Giorgio M Coen.
American Journal of Kidney Diseases (2004)

227 Citations

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