His main research concerns Internal medicine, Endocrinology, Heart failure, Cardiology and Cell biology. Endothelial dysfunction, Mineralocorticoid receptor, Muscle hypertrophy, Aldosterone and Oxidative stress are the subjects of his Endocrinology studies. His Endothelial dysfunction research includes elements of Glucose-6-phosphate dehydrogenase activity, CREB, CAMP-Responsive Element Modulator, Nitric oxide and Hyperaldosteronism.
His Heart failure study integrates concerns from other disciplines, such as Heart disease, Macrophage and Glucose uptake. His Cardiology research incorporates themes from Tissue homeostasis, Diastole and Monocyte. His work carried out in the field of Cell biology brings together such families of science as Molecular biology, Cellular differentiation and Cell growth.
His primary areas of investigation include Internal medicine, Endocrinology, Heart failure, Cardiology and Cell biology. In his papers, Ronglih Liao integrates diverse fields, such as Internal medicine and Pressure overload. Ronglih Liao combines subjects such as Biochemistry, Signal transduction and Calcium with his study of Endocrinology.
The concepts of his Heart failure study are interwoven with issues in Heart disease and Disease. His Cell biology research includes elements of Immunology, Cell, Programmed cell death and Cellular differentiation. His work deals with themes such as Cancer research and Amyloidosis, which intersect with Cardiomyopathy.
Ronglih Liao mainly focuses on Internal medicine, Cardiology, Heart failure, Amyloidosis and Cardiac amyloidosis. Ronglih Liao performs multidisciplinary study in the fields of Internal medicine and Context via his papers. His work on Cardiac fibrosis and Muscle hypertrophy as part of general Cardiology research is frequently linked to Pressure overload, bridging the gap between disciplines.
The various areas that Ronglih Liao examines in his Heart failure study include Epidemiology, Dementia, Disease, Induced pluripotent stem cell and Diastole. His Amyloidosis research is multidisciplinary, incorporating perspectives in Cardiomyopathy and AL amyloidosis, Amyloid. The Cardiac amyloidosis study combines topics in areas such as Gastroenterology, Nephrotic syndrome and Cholesterol.
Ronglih Liao spends much of his time researching Heart failure, Cardiology, Internal medicine, Amyloidosis and Cardiac amyloidosis. His Heart failure study incorporates themes from Diabetes mellitus, Combination therapy, Bioinformatics and Diastole. His research is interdisciplinary, bridging the disciplines of Macrophage and Internal medicine.
When carried out as part of a general Amyloidosis research project, his work on Amyloid cardiomyopathy is frequently linked to work in Population, therefore connecting diverse disciplines of study. His Amyloid research is multidisciplinary, incorporating elements of Cardiomyopathy, Transthyretin and Multiple myeloma. His AL amyloidosis study combines topics from a wide range of disciplines, such as Cardiac function curve, Positron emission tomography and Interquartile range.
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Telomere dysfunction induces metabolic and mitochondrial compromise
Ergiin Sahin;Simona Colla;Marc Liesa;Javid Moslehi.
Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure
Ichiro Shiojima;Kaori Sato;Yasuhiro Izumiya;Stephan Schiekofer.
Journal of Clinical Investigation (2005)
CD31− but Not CD31+ Cardiac Side Population Cells Exhibit Functional Cardiomyogenic Differentiation
Otmar Pfister;Frédéric Mouquet;Mohit Jain;Ross Summer.
Circulation Research (2005)
Phenotypic Spectrum Caused by Transgenic Overexpression of Activated Akt in the Heart
Takashi Matsui;Ling Li;Justina C. Wu;Stuart Cook.
Journal of Biological Chemistry (2002)
Cell Therapy Attenuates Deleterious Ventricular Remodeling and Improves Cardiac Performance After Myocardial Infarction
Mohit Jain;Harout DerSimonian;Daniel A. Brenner;Soeun Ngoy.
Evidence for Human Lung Stem Cells
Jan Kajstura;Marcello Rota;Sean R Hall;Toru Hosoda.
The New England Journal of Medicine (2011)
Human amyloidogenic light chains directly impair cardiomyocyte function through an increase in cellular oxidant stress.
Daniel A. Brenner;Mohit Jain;David R. Pimentel;Bo Wang.
Circulation Research (2004)
Loss of Cardiac microRNA-Mediated Regulation Leads to Dilated Cardiomyopathy and Heart Failure
Prakash K. Rao;Yumiko Toyama;H. Rosaria Chiang;Sumeet Gupta.
Circulation Research (2009)
Ly-6Chigh Monocytes Depend on Nr4a1 to Balance both Inflammatory and Reparative Phases in the Infarcted Myocardium
Ingo Hilgendorf;Louisa M.S. Gerhardt;Timothy C. Tan;Carla Winter.
Circulation Research (2014)
Cardiac-Specific Overexpression of GLUT1 Prevents the Development of Heart Failure Attributable to Pressure Overload in Mice
Ronglih Liao;Mohit Jain;Lei Cui;Jessica D’Agostino.
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