Mohit Jain mainly focuses on Cell biology, Internal medicine, Gene expression, Mitochondrion and Cardiac function curve. His Cell biology research includes themes of Cell membrane and Cell polarity. His studies deal with areas such as Endocrinology and Cardiology as well as Internal medicine.
His Endocrinology research is multidisciplinary, relying on both Cardiomyopathy, Amyloid, Reversion and Amyloidosis. His research integrates issues of Proteasome, Molecular biology and Sulforaphane in his study of Gene expression. His Mitochondrion study combines topics in areas such as mitochondrial fusion, Nuclear gene, Mitochondrial DNA and Mitochondrial disease.
His main research concerns Internal medicine, Cell biology, Biochemistry, Endocrinology and Cardiology. Internal medicine is a component of his Cardiac function curve, Ischemia, Muscle hypertrophy, Heart failure and Blood pressure studies. As part of the same scientific family, Mohit Jain usually focuses on Cell biology, concentrating on Cell growth and intersecting with Cancer cell.
His study on Amino acid, Enzyme, Metabolism and Proteomics is often connected to Soluble guanylyl cyclase as part of broader study in Biochemistry. Many of his studies involve connections with topics such as Inflammation and Endocrinology. His is doing research in Ventricular remodeling and Myocardial infarction, both of which are found in Cardiology.
His primary scientific interests are in Internal medicine, Cell biology, Inflammation, Disease and Endocrinology. Mohit Jain combines subjects such as Diabetes mellitus, Gastroenterology and Cardiology with his study of Internal medicine. He studies Cell biology, namely Effector.
His Inflammation study also includes fields such as
Mohit Jain spends much of his time researching Internal medicine, Cell biology, Endocrinology, Inflammation and Second messenger system. His study focuses on the intersection of Internal medicine and fields such as Diabetes mellitus with connections in the field of Weight loss. The study incorporates disciplines such as Ornithine aminotransferase, Ornithine, Arginase, Cancer cell and Cell cycle in addition to Cell biology.
In general Endocrinology study, his work on Insulin, Carbohydrate metabolism and Metabolite often relates to the realm of Trimethylamine N-oxide, thereby connecting several areas of interest. His Inflammation research integrates issues from Pathogenesis, Human plasma, Human physiology, Plasma samples and Oxylipin. His Second messenger system research is multidisciplinary, incorporating elements of CRISPR, Endonuclease, Effector, Oligonucleotide and Nucleotidyltransferase.
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Metabolite Profiling Identifies a Key Role for Glycine in Rapid Cancer Cell Proliferation
Mohit Jain;Roland Nilsson;Sonia Sharma;Nikhil Madhusudhan;Nikhil Madhusudhan.
CD31− but Not CD31+ Cardiac Side Population Cells Exhibit Functional Cardiomyogenic Differentiation
Otmar Pfister;Frédéric Mouquet;Mohit Jain;Ross Summer.
Circulation Research (2005)
Human amyloidogenic light chains directly impair cardiomyocyte function through an increase in cellular oxidant stress.
Daniel A. Brenner;Mohit Jain;David R. Pimentel;Bo Wang.
Circulation Research (2004)
Cell Therapy Attenuates Deleterious Ventricular Remodeling and Improves Cardiac Performance After Myocardial Infarction
Mohit Jain;Harout DerSimonian;Daniel A. Brenner;Soeun Ngoy.
Systematic identification of human mitochondrial disease genes through integrative genomics.
Sarah Calvo;Mohit Jain;Mohit Jain;Xiaohui Xie;Sunil A Sheth;Sunil A Sheth.
Nature Genetics (2006)
Metabolic enzyme expression highlights a key role for MTHFD2 and the mitochondrial folate pathway in cancer
Roland Nilsson;Mohit Jain;Nikhil Madhusudhan;Nina Gustafsson Sheppard.
Nature Communications (2014)
Cardiac-Specific Overexpression of GLUT1 Prevents the Development of Heart Failure Attributable to Pressure Overload in Mice
Ronglih Liao;Mohit Jain;Lei Cui;Jessica D’Agostino.
An epigenetic mechanism of resistance to targeted therapy in T cell acute lymphoblastic leukemia.
Birgit Knoechel;Justine E Roderick;Kaylyn E Williamson;Jiang Zhu.
Nature Genetics (2014)
Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.
Flora Sam;Douglas B. Sawyer;Donny L.-F. Chang;Franz R. Eberli.
American Journal of Physiology-heart and Circulatory Physiology (2000)
Infusion of Light Chains From Patients With Cardiac Amyloidosis Causes Diastolic Dysfunction in Isolated Mouse Hearts
Ronglih Liao;Mohit Jain;Paige Teller;Lawreen H. Connors.
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